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Your search keyword '"Jégou, Jean-François"' showing total 29 results

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29 results on '"Jégou, Jean-François"'

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2. IL-34 Exerts Anti-Inflammatory Activities on Keratinocytes and Is Downregulated in Psoriatic-Inflamed Skin

6. IL-22 Promotes Neural Stem Cell Self-Renewal in the Adult Brain

8. IL-17 and IL-22 are pivotal cytokines to delay wound healing of S. aureus and P. aeruginosa infected skin

9. Dermal fibroblasts are the key sensors of aseptic skin inflammation through interleukin 1 release by lesioned keratinocytes

13. Oncostatin M is overexpressed in skin squamous-cell carcinoma and promotes tumor progression

14. Th-17 response and antimicrobial peptide expression are uniformly expressed in gastric mucosa of Helicobacter pylori -infected patients independently of their clinical outcomes

16. Interleukin-17A-induced production of acute serum amyloid A by keratinocytes contributes to psoriasis pathogenesis

17. Development of a new model of reconstituted mouse epidermis and characterization of its response to proinflammatory cytokines

18. Measles virus infection of human keratinocytes: Possible link between measles and atopic dermatitis

19. IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome

20. Développement d’un modèle d’épiderme reconstruit murin : caractérisation histologique et réponse aux cytokines proinflammatoires

21. Oncostatin M overexpression induces skin inflammation but is not required in the mouse model of imiquimod‐induced psoriasis‐like inflammation

26. Interleukin 34 exerts anti-inflammatory activities on keratinocytes and is down-regulated in psoriatic-inflamed skin

27. Photometric computer vision-aided system for psoriasis severity scoring: a preclinical study based on a mouse model of psoriasis.

28. E x vivo cytokine production in psoriatic disease: Towards specific signatures in cutaneous psoriasis and peripheral psoriatic arthritis.

29. IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome.

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