151 results on '"Gadeau, Alain-Pierre"'
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2. Signalling by senescent melanocytes hyperactivates hair growth
3. Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction
4. Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction
5. Empagliflozin inhibits angiotensin II-induced hypertrophy in H9c2 cardiomyoblasts through inhibition of NHE1 expression
6. Evaluating the effects of sodium glucose co-transporter -2 inhibitors from a renin–angiotensin–aldosterone system perspective in patients infected with COVID-19: contextualizing findings from the dapagliflozin in respiratory failure in patients with COVID-19 study
7. Desert Hedgehog-Driven Endothelium Integrity Is Enhanced by Gas1 (Growth Arrest-Specific 1) but Negatively Regulated by Cdon (Cell Adhesion Molecule-Related/Downregulated by Oncogenes)
8. Abstract P3107: Sexual Dimorphism In Diastolic Dysfunction And Cardiac Small Vessel Disease In Heart Failure With Preserved Ejection Fraction Mouse Models
9. Abstract P2082: Thrombosis In The Coronary Microvasculature May Be Responsible For Impaired Cardiac Relaxation And Diastolic Dysfunction
10. Partial Mural Cell Ablation Disrupts Coronary Vasculature Integrity and Induces Systolic Dysfunction
11. A new reliable, transposable and cost-effective assay for absolute quantification of total plasmatic bevacizumab by LC–MS/MS in human plasma comparing two internal standard calibration approaches
12. Intra-articular Injection of Mesenchymal Stem Cells and Platelet-Rich Plasma to Treat Patellofemoral Osteoarthritis: Preliminary Results of a Long-Term Pilot Study
13. Tamoxifen Accelerates Endothelial Healing by Targeting ERα in Smooth Muscle Cells
14. Thrombosis in the coronary microvasculature may be responsible for impaired cardiac relaxation and diastolic dysfunction
15. Cardiac pericytes may contribute to heart disease
16. ICAM1 expression by the microvasculature impairs capillary perfusion which compromises hind limb ischemia recovery in diabetic mice
17. Diastolic dysfunction is associated with cardiac small vessel disease in ovariectomized females but not in males
18. Increased endothelium activation and leakage do not promote diastolic dysfunction in mice fed with a high fat diet and treated with L-NAME
19. Praliciguat Promotes Ischemic Leg Reperfusion in Leptin Receptor-Deficient Mice
20. Restoring Endothelial Function by Targeting Desert Hedgehog Downstream of Klf2 Improves Critical Limb Ischemia in Adults
21. Cardiac pericytes are necessary for coronary vasculature integrity and cardiomyocyte differentiation
22. Testosterone Prevents Cutaneous Ischemia and Necrosis in Males Through Complementary Estrogenic and Androgenic Actions
23. Na+/H+ exchanger isoform 1 induced osteopontin expression in cardiomyocytes involves NFAT3/Gata4
24. Endothelial protection does not prevent diastolic dysfunction in high fat diet + L-NAME treated mice
25. Soluble guanylate cyclase stimulator praliciguat promotes ischemic leg reperfusion in db/db mice
26. Desert Hedgehog related endothelial dysfunction is sufficient to induce diastolic dysfunction
27. Cardiac and brain pericytes are able to regenerate over time
28. Impaired Hedgehog signalling-induced endothelial dysfunction is sufficient to induce neuropathy: implication in diabetes
29. Crosstalk between Sodium–Glucose Cotransporter Inhibitors and Sodium–Hydrogen Exchanger 1 and 3 in Cardiometabolic Diseases
30. Mast Cells Are the Trigger of Small Vessel Disease and Diastolic Dysfunction in Diabetic Obese Mice
31. Blood-brain barrier genetic disruption leads to protective barrier formation at the Glia Limitans
32. Blood–brain barrier genetic disruption leads to protective barrier formation at the Glia Limitans
33. Full-length Dhh and N-terminal Shh act as competitive antagonists to regulate angiogenesis and vascular permeability
34. Mast cells participate in the development of diastolic dysfunction in diabetic obese mice
35. Wavelet Analysis of Microcirculatory Flowmotion Reveals Cardiovascular Regulatory Mechanisms–Data from a Beta-Blocker
36. Desert Hedgehog-driven endothelium integrity is enhanced by Gas1 but negatively regulated by Cdon
37. Genetic disruption of the Blood Brain Barrier leads to protective barrier formation at the Glia Limitans
38. Endothelial cell response to Hedgehog ligands depends on their processing
39. Characterizing Vascular Dysfunction in Genetically Modified Mice through the Hyperoxia Model
40. Osteopontin: A Promising Therapeutic Target in Cardiac Fibrosis
41. Full-length Dhh and N-terminal Shh act as competitive antagonists to regulate angiogenesis and vascular permeability.
42. Evidence on microcirculatory dynamics that preserves the equivalent perfusion steady state in both inferior limbs
43. This hyperoxia mouse model using the wavelet transform analysis of flowmotion signals helps to look further into microvascular dysfunction
44. A METHOD FOR QUANTIFYING BEVACIZUMAB
45. Observations on the perfusion recovery of regenerative angiogenesis in an ischemic limb model under hyperoxia
46. Na+/H+ exchanger isoform 1-induced osteopontin expression facilitates cardiac hypertrophy through p90 ribosomal S6 kinase
47. Endogenous Sonic Hedgehog limits inflammation and angiogenesis in the ischaemic skeletal muscle of mice
48. Na+/H+ exchanger isoform 1-induced osteopontin expression facilitates cardiomyocyte hypertrophy
49. Na+/H+ exchanger isoform 1-induced osteopontin expression facilitates cardiac hypertrophy through p90 ribosomal S6 kinase.
50. Impaired Hedgehog signalling-induced endothelial dysfunction is sufficient to induce neuropathy: implication in diabetes
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