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1. APOEε4 potentiates amyloid β effects on longitudinal tau pathology

2. 14-3-3 ζ/δ-reported early synaptic injury in Alzheimer’s disease is independently mediated by sTREM2

3. Astrocyte biomarker signatures of amyloid-β and tau pathologies in Alzheimer’s disease

4. The relation of synaptic biomarkers with Aβ, tau, glial activation, and neurodegeneration in Alzheimer’s disease

5. Biomarker modeling of Alzheimer’s disease using PET-based Braak staging

7. Author Correction: [11C]Martinostat PET analysis reveals reduced HDAC I availability in Alzheimer’s disease

8. [11C]Martinostat PET analysis reveals reduced HDAC I availability in Alzheimer’s disease

9. CSF tau368/total-tau ratio reflects cognitive performance and neocortical tau better compared to p-tau181 and p-tau217 in cognitively impaired individuals

11. Microglial activation and tau propagate jointly across Braak stages

12. Amyloid-beta modulates the association between neurofilament light chain and brain atrophy in Alzheimer’s disease

14. Association of reactive astrogliosis and microglial activation with tau pathology in Alzheimer’s disease

15. Neuroinflammation Exacerbates Irritability and Agitation in Alzheimer’s Disease

16. APOEε4 potentiates the effects of Aβ pathology on the deposition of neurofibrillary tangles via tau phosphorylation

17. Potential utility of using both APOEε4 and Aβ positivity to enrich clinical trials of tau‐targeting therapies

18. Sex impacts the association of plasma Glial Fibrillary Acidic Protein with neurodegeneration in Alzheimer’s disease

19. Plasma biomarkers of tau for the differentiation between slow and fast tau‐PET accumulators

20. 14-3-3 $$\upzeta /\updelta$$-reported early synaptic injury in Alzheimer’s disease is independently mediated by sTREM2

21. Plasma pTau‐217 and N‐terminal tau (NTA) enhance sensitivity to identify tau PET positivity in amyloid‐β positive individuals.

22. Blood phosphorylated tau 181 as a biomarker for Alzheimer's disease: a diagnostic performance and prediction modelling study using data from four prospective cohorts

24. The Forgetful Golfer

26. Concerns about the Future

28. A Young Man with Memory and Walking Difficulties

29. A Young Missionary with Problems Quoting the Bible

30. A Challenging Thesis

32. Publisher Correction: Microglial activation and tau propagate jointly across Braak stages

33. Plasma and CSF concentrations of N‐terminal tau fragments associate with in vivo neurofibrillary tangle burden.

34. Amyloid beta plaque accumulation with longitudinal [18F]AZD4694 PET

35. Verbal memory formation across PET-based Braak stages of tau accumulation in Alzheimer’s disease

38. APOEε4 associates with microglial activation independently of Aβ plaques and tau tangles

40. Medial Temporal Tau Predicts Memory Decline in Cognitively Unimpaired Elderly (P5-6.011)

41. APOE ε4 associates with microglial activation independently of Aβ plaques and tau tangles

42. Blood‐brain barrier integrity impacts the use of plasma amyloid‐β as a proxy of brain amyloid‐β pathology

43. APOEε4 potentiates Aβ effects on longitudinal tangle accumulation via tau phosphorylation

44. Discriminative accuracy of the A/T/N scheme to identify cognitive impairment due to Alzheimer's disease

45. Medial temporal tau predicts memory decline in cognitively unimpaired elderly

46. Tau phosphorylation is more closely associated with amyloid‐β plaques than with tau neurofibrillary tangles

47. Verbal recognition declines in later Braak Stages compared to verbal delayed recall

48. Comparison of plasma amyloid, tau, and astrocyte biomarkers to identify AD pathophysiology

49. pTau heterogeneity as a measure for disease severity in incipient Alzheimer's disease

50. TRIAD multi‐dimensional biobank for biomarker discovery

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