Search

Your search keyword '"Verfaillie, Sander C.J."' showing total 6 results
Start Over Author "Verfaillie, Sander C.J." Publication Type Magazines
6 results on '"Verfaillie, Sander C.J."'

2. [18F]Flortaucipir PET Across Various MAPTMutations in Presymptomatic and Symptomatic Carriers

Author

Wolters, Emma E., Papma, Janne M., Verfaillie, Sander C.J., Visser, Denise, Weltings, Emma, Groot, Colin, van der Ende, Emma L., Giannini, Lucia A.A., Tuncel, Hayel, Timmers, Tessa, Boellaard, Ronald, Yaqub, Maqsood, van Assema, Danielle M.E., Kuijper, Dennis A., Segbers, Marcel, Rozemuller, Annemieke J.M., Barkhof, Frederik, Windhorst, Albert D., van der Flier, Wiesje M., Pijnenburg, Yolande A.L., Scheltens, Philip, van Berckel, Bart N.M., van Swieten, John C., Ossenkoppele, Rik, and Seelaar, Harro

Published
2021
Full Text
View/download PDF

3. Subjective Cognitive Decline Is Associated With Altered Default Mode Network Connectivity in Individuals With a Family History of Alzheimer’s Disease

Author

Verfaillie, Sander C.J., Pichet Binette, Alexa, Vachon-Presseau, Etienne, Tabrizi, Shirin, Savard, Mélissa, Bellec, Pierre, Ossenkoppele, Rik, Scheltens, Philip, van der Flier, Wiesje M., Breitner, John C.S., Villeneuve, Sylvia, Aisen, Paul, Anthal, Elena, Appleby, Melissa, Ayranci, Gülebru, Barkun, Alan, Beaudry, Thomas, Bellec, Pierre, Benbouhoud, Fatiha, Bohbot, Veronique, Brandt, Jason, Breitner, John, Carmo, Leopoldina, Carrier Charles, Edouard, Chakravarty, Mallar, Cheewakriengkrai, Laksanun, Collins, Louis, Courcot, Blandine, Couture, Doris, Craft, Suzanne, Cuello, Claudio, Dadar, Mahsa, Dansereau, Christian, DasSamir, Marina, Dauar-Tedeschi, Dea, Doris, Debacker, Clement, Desautels, Rene, Dubuc, Sylvie, Duclair, Guerda, Dufour, Marianne, Eisenberg, Mark, El-Khoury, Rana, Etienne, Pierre, Evans, Alan, Faubert, Anne-Marie, Ferdinand, Fabiola, Fonov, Vladimir, Fontaine, David, Frappier, Josée, Joanne, Frenette, Gagne, Guylaine, Gauthier, Serge, Gervais, Valérie, Giles, Renuka, Gonneaud, Julie, Gordon, Renee, Hoge, Rick, Hyman, Bradley, Ituria-Medina, Yasser, Jack, Clifford, Kat, Justin, Kazazian, Christina, Khachaturian, Zaven, Knopman, David, Kostopoulos, Penelope, Labonte, Anne, Lafaille-Magnan, Marie-Elyse, Lee, Tanya, Leoutsakos, Jeannie-Marie, Lepage, Claude, Leppert, Illana, Madjar, Cécile, Mahar, Laura, Maillet, David, Maltais, Jean-Robert, Mathieu, Axel, Mathotaarachchi, Sulantha, Maultaup, Gerhard, Mayrand, Ginette, Meyer, Pierre-François, Michaud, Diane, Miron, Justin, Montine, Thomas, Morris, John, Münter, Lisa-Marie, Nair, Vasavan, Near, Jamie, Newbold-Fox, Holly, Orban, Pierre, Page, Véronique, Pascoal, Tharick, Petkova, Mirela, Picard, Cynthia, Pichet Binette, Alexa, Pogossova, Galina, Poirier, Judes, Pruessner, Jens, Rajah, Natasha, Rioux, Pierre, Rosa-Neto, Pedro, Sager, Mark, Saint-Fort, Eunice Farah, Savard, Mélissa, Sperling, Reisa, Tabrizi, Shirin, Tam, Angela, Tardif, Christine, Tariot, Pierre, Teigner, Eduard, Theroux, Louise, Thomas, Ronald, Toussaint, Paule-Joanne, Tremblay-Mercier, Jennifer, Tuwaig, Miranda, Vallee, Isabelle, Venugopalan, Vinod, Verfaillie, Sander, Villeneuve, Sylvia, Vogel, Jacob, Wan, Karen, Wang, Seqian, and Yu, Elsa

Abstract

Both subjective cognitive decline (SCD) and a family history of Alzheimer’s disease (AD) portend risk of brain abnormalities and progression to dementia. Posterior default mode network (pDMN) connectivity is altered early in the course of AD. It is unclear whether SCD predicts similar outcomes in cognitively normal individuals with a family history of AD.

Published
2018
Full Text
View/download PDF

4. The course of the neural correlates of reversal learning in obsessive–compulsive disorder and major depression: A naturalistic follow-up fMRI study

Author

Verfaillie, Sander C.J., de Wit, Stella J., Vriend, Chris, Remijnse, Peter L., Veltman, Dick J., and van den Heuvel, Odile A.

Abstract

Reversal learning (RL) is impaired in obsessive–compulsive disorder (OCD) as well as in major depressive disorder (MDD). It is yet unknown to what extent pathophysiological mechanisms are state-dependent.

Published
2016
Full Text
View/download PDF

5. The Association of Glucose Metabolism and Eigenvector Centrality in Alzheimer's Disease

Author

Adriaanse, Sofie M., Wink, Alle Meije, Tijms, Betty M., Ossenkoppele, Rik, Verfaillie, Sander C.J., Lammertsma, Adriaan A., Boellaard, Ronald, Scheltens, Philip, van Berckel, Bart N.M., and Barkhof, Frederik

Abstract

AbstractBoth fluorine-18-labeled fluorodeoxyglucose ([18F]FDG) positron emission tomography, examining glucose metabolism, and resting-state functional magnetic resonance imaging (rs-fMRI), using covarying blood oxygen levels, can be used to explore neuronal dysfunction in Alzheimer's disease (AD). Both measures are reported to identify similar brain regions affected in AD patients. The spatial overlap and association of [18F]FDG with rs-fMRI in AD patients and controls were examined to investigate whether these two measures are associated, and if so, to what extent. For 24 AD patients and 18 controls, [18F]FDG and rs-fMRI data were available. [18F]FDG standardized uptake value ratios (SUVr), with cerebellar gray matter (GM) as reference tissue, were calculated. Eigenvector centrality (EC) mapping was used to spatially analyze the functional brain network. Group differences were calculated for [18F]FDG and eigenvector centrality mapping (ECM) values in four cortical regions (occipital, parietal, frontal, and temporal) and across voxels, with age, gender, and GM as covariates. Correlation of [18F]FDG with ECM was calculated within groups. Both lowered [18F]FDG SUVr and EC values were seen in the parietal and occipital cortex of AD patients. However, [18F]FDG yielded more robust and widespread brain areas affected in AD patients; hypometabolism was also observed in the temporal cortex and regions within frontal brain areas. Poor spatial overlap of both measures was observed. No associations were found between local [18F]FDG SUVr and ECM. In conclusion, agreement of [18F]FDG and ECM in AD patients seems moderate at best. [18F]FDG was most accurate in distinguishing AD patients from controls.

Published
2016
Full Text
View/download PDF

6. Long-term effects of amyloid, hypometabolism, and atrophy on neuropsychological functions

Author

Ossenkoppele, Rik, Flier, Wiesje M. van der, Verfaillie, Sander C.J., Vrenken, Hugo, Versteeg, Adriaan, Schijndel, Ronald A. van, Sikkes, Sietske A., Twisk, Jos, Adriaanse, Sofie M., Zwan, Marissa D., Boellaard, Ronald, Windhorst, Albert D., Barkhof, Frederik, Scheltens, Philip, Lammertsma, Adriaan A., and Berckel, Bart N.M. van

Abstract

To assess how amyloid deposition, glucose hypometabolism, and cerebral atrophy affect neuropsychological performance in patients with Alzheimer disease (AD) dementia, patients with mild cognitive impairment (MCI), and controls over time.

Published
2014
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results