118 results on '"de Plaen, Isabelle G."'
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2. Microbiota regulates neonatal disease tolerance to virus-evoked necrotizing enterocolitis by shaping the STAT1-NLRC5 axis in the intestinal epithelium
3. Scattered Crypt Intestinal Epithelial Cell Apoptosis Induces Necrotizing Enterocolitis Via Intricate Mechanisms
4. Nailfold Capillaroscopy: A Promising, Noninvasive Approach to Predict Retinopathy of Prematurity
5. Macrophage-derived IGF-1 protects the neonatal intestine against necrotizing enterocolitis by promoting microvascular development
6. Blocking NF-κB Activation in Ly6c+ Monocytes Attenuates Necrotizing Enterocolitis
7. Intestinal microcirculation and necrotizing enterocolitis: The vascular endothelial growth factor system
8. In Inflamed Intestinal Tissues and Epithelial Cells, Interleukin 22 Signaling Increases Expression of H19 Long Noncoding RNA, Which Promotes Mucosal Regeneration
9. Epithelial PIK3R1 (p85) and TP53 Regulate Survivin Expression during Adaptation to Ileocecal Resection
10. Sirtuin-6 Preserves R-spondin-1 Expression and increases Resistance of intestinal Epithelium to injury in Mice
11. Endothelial FOXC1 and FOXC2 promote intestinal regeneration after ischemia–reperfusion injury.
12. Bifidobacteria Stabilize Claudins at Tight Junctions and Prevent Intestinal Barrier Dysfunction in Mouse Necrotizing Enterocolitis
13. Feeding mode influences dynamic gut microbiota signatures and affects susceptibility to anti-CD3 mAb-induced intestinal injury in neonatal mice.
14. Platelet-activating factor induces the processing of nuclear factor-[kappa]B p105 into p50, which mediates acute bowel injury in mice
15. Tu1245 CD8 T CELLS MEDIATE THE EFFECT OF INTESTINAL CRYPT EPITHELIAL CELL APOPTOSIS ON THE INDUCTION OF NECROTIZING ENTEROCOLITIS-LIKE MUCOSAL INJURY IN NEONATAL MICE
16. Su1211 ENDOTHELIAL CELL FOXM1 IS DECREASED IN THE INTESTINE PRIOR TO NEC DEVELOPMENT AND MEDIATES VEGFR2-DEPENDENT ENDOTHELIAL CELL PROLIFERATION AND MICROVASCULAR DEVELOPMENT
17. Neonatal Necrotizing Enterocolitis: Clinical Considerations and Pathogenetic Concepts
18. Macrophage inflammatory protein-2 mediates the bowel injury induced by platelet-activating factor
19. Lipopolysaccharide induces CXCL2/macrophage inflammatory protein-2 gene expression in enterocytes via NF-κB activation: independence from endogenous TNF-α and platelet-activating factor
20. Endotoxin, but not platelet-activating factor, activates nuclear factor-κB and increases IκBα and IκBβ turnover in enterocytes
21. Neuronal nitric oxide synthase (NOS) regulates the expression of inducible NOS in rat small intestine via modulation of nuclear factor kappa B
22. SARS-CoV-2 ORF8 Forms Intracellular Aggregates and Inhibits IFNγ-Induced Antiviral Gene Expression in Human Lung Epithelial Cells.
23. Interferon-γ inhibits sirtuin 6 gene expression in intestinal epithelial cells through a microRNA-92b-dependent mechanism.
24. 270 NEONATAL INTESTINAL MACROPHAGES PROMOTE VASCULAR TUBELIKE STRUCTURE FORMATION IN VITRO VIA INSULIN GROWTH FACTOR-1
25. 267 IMPAIRMENT OF THE EMBRYONIC MACROPHAGE COMPARTMENT DURING NECROTIZING ENTEROCOLITIS AFFECTS POSTNATAL INTESTINAL MICROVASCULAR DEVELOPMENT
26. 766 SCATTERED INTESTINAL EPITHELIAL APOPTOSIS INDUCES MOUSE PUP INTESTINAL INJURY THAT MIMICS NECROTIZING ENTEROCOLITIS IN HUMANS
27. Prenatal inflammation impairs intestinal microvascular development through a TNF-dependent mechanism and predisposes newborn mice to necrotizing enterocolitis.
28. Tu1863 - L-Selectin Antibodies Prevent the Increase in Intestinal Monocyte-Derived Macrophages in Necrotizing Enterocolitis
29. Tu1266 - IGF1 Protects the Neonatal small Intestinal Microvasculature in a Model of Necrotizing Enterocolitis
30. Lack of VEGFR2 signaling causes maldevelopment of the intestinal microvasculature and facilitates necrotizing enterocolitis in neonatal mice.
31. Molecular Mechanisms Underlying the Regulation of the MFG-E8 Gene Promoter Activity in Physiological and Inflammatory Conditions.
32. Intestinal Vascular Endothelial Growth Factor Is Decreased in Necrotizing Enterocolitis.
33. FOXM1 Promotes Intestinal Vascular Endothelial Cell Proliferation and Protects Neonatal Mice against Necrotizing Enterocolitis.
34. Anti‐CD3 mAb Induced Necrotizing Enterocolitis in Formula‐Fed Neonatal Mice through a Gut Microbiome Colonization Dependent Mechanism.
35. Inflammatory signaling in NEC: Role of NF-κB, cytokines and other inflammatory mediators.
36. Platelet-activating factor induces the processing of nuclear factor-κ p105 into p50, which mediates acute bowel injury in mice.
37. Human Milk Growth Factors and Their Role in NEC Prevention: A Narrative Review.
38. Tetrahydrobiopterin prevents platelet-activating factor-induced intestinal hypoperfusion and necrosis: Role of neuronal nitric oxide synthase.
39. Macrophage inflammatory protein-2 mediates the bowel injury induced by platelet-activating factor.
40. Platelet-activating factor and endotoxin activate CCAAT/enhancer binding protein in rat small intestine.
41. Lipopolysaccharide activates nuclear factor κB in rat intestine: role of endogenous platelet-activating factor and tumour necrosis factor.
42. INDEX OF SUSPICION.
43. M1693 Bifidobacterium Infantis Protects the Intestinal Barrier in a Neonatal Mouse Model of Necrotizing Enterocolitis
44. 479 Myosin Light Chain Kinase Inhibition Decreases the Incidence of Severe Bowel Injury in a Neonatal Mouse Model of Necrotizing Enterocolitis
45. MFG-E8 Plays an Important Role in Attenuating Cerulein-Induced Acute Pancreatitis in Mice.
46. Spherical nucleic acid targeting microRNA-99b enhances intestinal MFG-E8 gene expression and restores enterocyte migration in lipopolysaccharide-induced septic mice.
47. T1284 Anti-TNF Antibodies Do Not Ameliorate Bowel Injury in a Model of Acute Bowel Damage Induced By PAF and in a Neonatal Rat Model of Necrotizing Enterocolitis.
48. S1290 NF-κB-Dependent Gene Transcription in Response to Lipopolysaccharide Is Delayed and Prolonged in the Neonatal Intestine.
49. Epithelial TNF Receptor Signaling Promotes Mucosal Repair in Inflammatory Bowel Disease.
50. Sirtuin-6 Preserves R-spondin-1 Expression and Increases Resistance of Intestinal Epithelium to Injury in Mice.
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