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5. Genetic drivers of heterogeneity in type 2 diabetes pathophysiology

6. The Type 2 Diabetes Knowledge Portal: An open access genetic resource dedicated to type 2 diabetes and related traits

7. Primate-specific ZNF808 is essential for pancreatic development in humans

8. Genome-wide association study of placental weight identifies distinct and shared genetic influences between placental and fetal growth

10. Second international consensus report on gaps and opportunities for the clinical translation of precision diabetes medicine

11. Genetic analysis of blood molecular phenotypes reveals common properties in the regulatory networks affecting complex traits

12. Genetic effects on the timing of parturition and links to fetal birth weight

18. Non-coding variants disrupting a tissue-specific regulatory element in HK1 cause congenital hyperinsulinism

19. A saturated map of common genetic variants associated with human height

21. The Type 2 Diabetes Knowledge Portal: An open access genetic resource dedicated to type 2 diabetes and related traits

25. Author Correction: Genetic effects on the timing of parturition and links to fetal birth weight

26. Multi-ancestry genetic study of type 2 diabetes highlights the power of diverse populations for discovery and translation

30. Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution

31. A multi-layer functional genomic analysis to understand noncoding genetic variation in lipids

32. Systematic genetic testing for recessively inherited monogenic diabetes: a cross-sectional study in paediatric diabetes clinics

35. Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

36. The power of genetic diversity in genome-wide association studies of lipids

37. Higher maternal adiposity reduces offspring birthweight if associated with a metabolically favourable profile

39. Monogenic disease analysis establishes that fetal insulin accounts for half of human fetal growth

44. YIPF5 mutations cause neonatal diabetes and microcephaly through endoplasmic reticulum stress

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