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Your search keyword '"Beydon L"' showing total 244 results
Start Over Author "Beydon L" Publication Type Academic Journals
244 results on '"Beydon L"'

19. Acute/Chronic respiratory failure III

Author

Mondèjar, E. Fernàndez, Mata, G. Vazquez, Ferròn, F., Navarrete, P., Ruiz, J. M. Torres, Lestavel, P., Tronchon, L., Chambrin, M. C., Mangalaboyi, J., Rime, A., Chopin, C., Valta, P., Campodonico, R., Corbeil, C., Chassè, M., Châtillon, A., Braidy, J., Matar, N., Milic-Emili, J., Lòpez-Messa, J., Penas, L., Valverde, A., Dambrosio, M., Roupie, E., Carneiro, A., Anglade, M. C., Vasile, N., Brochard, L., Lemaire, F., Rubio, J., Carrasco, M. S., Mateo, I., Sierra, R., Escolar, A., Cozar, J., Bastin, K., Knapen, R., Moraine, J. J., Melot, C., Sergysels, R., Kahn, R. J., Pelosi, P., Cereda, M., Foti, G., D’Andrea, L., Manetti, B., Lissoni, A., Pesenti, A., Gallego, J. M. Allegue, Rubi, J. A. Gòmez, Sànchez, C. Palazòn, Moreno, A. Melgarejo, Lherm, T., Boiteau, R., Valente, E., Beaussier, M., Chamieh, F., Tenaillon, A., Righini, E. R., Alvisi, R., Ragazzi, R., Volta, C. A., Capuzzo, M., Gritti, G., Sydow, M., Burchardi, H., Zinserling, J., Crozier, T. A., Guttmann, J., Eberhard, L., Bertschmann, W., Fabry, B., Wolff, G., Rubini, A., DelMonte, D. D., Catena, V., Attar, I., Rattazzi, G., Alati, G. L., Diaz, M. Arias, Mata, G. Vàzquez, Navarro, P. Navarrete, Lòpez, F. Guerrero, Morales, A. Mèrida, Isenegger, J., Picazo, L., Sanchez, A., Hernandez, B., Pons, A., Conti, G., Di Chiara, L., De Blasi, R. A., Dell’Utri, D., Cogliati, A., Pelaia, P., Ferretti, A., Bernasconi, F., Banfi, G., Pesenti, A., Putensen, C., Putensen-Himmer, G., Leon, M., Huygen, P. E. M., Gültuna, I., Zwart, A., Ince, C., Bruining, H. A., Pompe, J. C., Kesecioĝlu, J., Rabbat, A., Laaban, J. P., Orvoen-Frija, E., Achkar, A., Rochemaure, J., Frigo, V., Solca, M., Melloni, G., Gerbsa, C., Ornaghi, A., Mancini, S., Cavagnoli, R., Fasano, W., Santos, C., Roca, J., Torres, A., Cardùs, J., Barberà, J. A., Felez, M. A., Rodriguez-Roisin, R., Oviedo-Moreira, R., Beydon, L., Nakos, G., Precates, A., Mathas, C., Bassilakis, N., Chagianagnostou, K., Massoura, L., Labropoulos, S., Devroey, M., Vansnick, P., Mèlot, C., Naeije, R., Nagy, V., Kiiski, R., Kaitainen, S., Karppi, R., Takala, J., Kesecioglu, J., Erdmann, W., Marin, J., Arnau, A., Tejeda, M., Olivares, D., Servera, E., Boix, J. H., Alvarez, F., Peydro, F., Mira, J. P., Belghith, M., Renaud, B., Deland, E., Brunet, F., Brusset, A., Lanore, J. J., Hamy, I., Termignon, J. L., Soubrane, O., Pochard, F., Dhainaut, J. F., Sidhu, P. S., Cockburn, J. F., Nicholson, D. A., Kennedy, A., Dawson, P., and Servera, F. E.

Published
1992
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29. Cerebral hemodynamics during arterial and [CO.sub.2] pressure changes: in vivo prediction by a mathematical model

Author

URSINO, M., MINASSIAN, A. TER, LODI, C. A., and BEYDON, L.

Subjects
Hemodynamics -- Research, Intracranial pressure -- Physiological aspects, Carbon dioxide in the body -- Physiological aspects, Biological sciences
Abstract

Ursino, M., A. Ter Minassian, C. A. Lodi, and L. Beydon. Cerebral hemodynamics during arterial and C[O.sub.2] pressure changes: in vivo prediction by a mathematical model. Am J Physiol Heart Circ Physiol 279: H2439-H2455, 2000.--The aim of this work was to analyze changes in cerebral hemodynamics and intracranial pressure (ICP) evoked by mean systemic arterial pressure (SAP) and arterial C[O.sub.2] pressure [MATHEMATICAL EXPRESSION NOT REPRODUCIBLE IN ASCII] challenges in patients with acute brain damage. The study was performed by means of a new simple mathematical model of intracranial hemodynamics, particularly aimed at routine clinical investigation. The model was validated by comparing its results with data from transcranial Doppler velocity in the middle cerebral artery ([V.sub.MCA]) and ICP measured in 44 tracings on 13 different patients during mean SAP and [MATHEMATICAL EXPRESSION NOT REPRODUCIBLE IN ASCII] challenges. The validation consisted of individual identification of 6 parameters in all 44 tracings by means of a best fitting algorithm. The parameters chosen for the identification summarize the main aspects of intracranial dynamics, i.e., cerebrospinal fluid circulation, intracranial elastance, and cerebrovascular control. The results suggest that the model is able to reproduce the measured time patterns of [V.sub.MCA] and ICP in all 44 tracings by using values for the parameters that lie within the ranges reported in the pathophysiological literature. The meaning of parameter estimates is discussed, and comments on the main virtues and limitations of the present approach are offered. carbon dioxide reactivity; intracranial pressure; cerebral autoregulation; severe brain damage; transcranial Doppler

Published
2000

33. BIS response to tamponade and dobutamine in swine varies with hypnotic/opiate ratio

Author

Beydon, L., Desfontis, J.C., Ganster, F., Petres, J., Gautier, F., Ferec, S., Cailleux, A., Dussaussoy, C., Liu, N., Chazot, T., Fischler, M., and Gogny, M.

Subjects
*DOBUTAMINE, *DOSE-response relationship in biochemistry, *CARDIAC tamponade, *LABORATORY swine, *HYPNOTICS, *VETERINARY electroencephalography, *CARDIAC output, *VETERINARY anesthesia, *COMBINATION drug therapy
Abstract

Abstract: Objectives: This study in swine assessed BIS stability in response to decreases and increases in cardiac output under two propofol/remifentanil dosage combinations, both producing the same depth of surgical anaesthesia. Methods: Eight anaesthetized-paralyzed ventilated adult swine were studied using a random-order cross-over design. Four received a P low/R high combination (P, 8.4±0.9mg/kg/h; and R, 0.54±0.02μg/kg/min) and then a P high/R low combination (P, 26.7±2.1mg/kg/h; and R, 0.34±0.01μg/kg/min). The other four had these two combinations in the reverse order. Under each P/R combination, and after a 60-minutes steady state, a 15-minute stable cardiac tamponade was induced by intrapericardial gelatine infusion. Then, after returning to pre tamponade condition, a 15minutes period with dobutamine was allowed. Results: Tamponade induced falls in average mean arterial pressure (MAP) (from 79±18 to 47±9mmHg; p <0.05) and cardiac output (Qc) (from 1.90±0.46l/min to 1.20±0.38l/min, p <0.05). Conversely, dobutamine increased both MAP and Qc (p <0.05). During each type of hemodynamic challenges, changes in anaesthesia depth as assessed by BIS differed dramatically between the two drug combinations, despite observing the same percent change in P and R effect-site concentration. With P high/R low and tamponade, BIS fell from 65±5 to 29±10 (p <0.05); dobutamine produced opposite effects. With P low/R high, in contrast, BIS was not influenced by either of the hemodynamic challenges. Conclusion: Conversely to a high propofol/low remifentanil combination, a low propofol/high remifentanil combination allows constant anaesthetic depth in the face of haemodynamic challenges. [Copyright &y& Elsevier]

Published
2009
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38. Cerebral hemodynamics during arterial and CO[sub 2] pressure changes: in vivo prediction by a mathematical model.

Author

Ursino, M., Minassian, A. Ter, Lodi, C.A., and Beydon, L.

Subjects
BRAIN injuries, CEREBRAL circulation, HEMODYNAMICS, INTRACRANIAL pressure, PHYSIOLOGY
Abstract

Presents information on a study which investigated the changes in cerebral hemodynamics and intracranial pressure evoked by mean system arterial pressure and arterial carbon dioxide pressure challenges in patients with acute brain damage. Methodology of the study; Results and discussion on the study.

Published
2000
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44. Champ 4. Sécurisation du matériel en réanimation

Author

Fougère, S., Beydon, L., and Saulnier, F.

Subjects
*INTENSIVE care units, *SAFETY, *HOSPITALS, *BIOMEDICAL engineering
Abstract

Abstract: Medical devices are known to carry risks from design to scrap. Accident reports in ICU show that medical device account for only 20 % of accidents. Formation of users and providing a postmarketing incident reporting are thus essential in health institutions. Clinical and engineering departments should cooperate to produce and secure procedures which should be applied during the lifetime of each clinical device. Several points should be especially fulfilled: close cooperation between clinical departments and biomedical engineering departments with available technicians, computer-based inventory of all devices, evaluation of specifications required before purchasing a new device, education of users on utilisation and maintenance, technical follow-up of devices and keeping maintenance and repair logs, ability to provide users with replacement devices, provision of check-lists before use, forging criteria to decide when device should be discarded. These principles are simple and should be considered as mandatory in order to improve medical device related security. [Copyright &y& Elsevier]

Published
2008
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