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Figure S1. Mutant KIT localizes preferentially to EL in HMC-1.1 and SKNO-1 cells. Figure S2. In HMC-1.1 and SKNO-1, the major site for KIT autophosphorylation is colocalized with the Golgi region. Figure S3. Distribution of signal molecules in Kasumi-1 and HMC-1.1 cells. Figure S4. Effect of knockdown of PTP1B, SHP-1, and SHP-2 on KIT signals. Figure S5. KITN822K does not activate AKT, ERK, and STAT5 in SKNO-1 cells. Figure S6. Effect of inhibition of normal lipid raft formation on KIT distribution. (PDF 14400 kb)