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1. Identification of TFPI as a receptor reveals recombination-driven receptor switching in Clostridioides difficile toxin B variants

2. Bypass Grafting vs Endovascular Therapy in Patients With Non-Dialysis-Dependent Chronic Kidney Disease and Chronic Limb-Threatening Ischemia (CRITISCH Registry)

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3. Clostridioides difficile Toxin CDT Induces Cytotoxic Responses in Human Mucosal-Associated Invariant T (MAIT) Cells

4. The Binary Toxin of Clostridioides difficile Alters the Proteome and Phosphoproteome of HEp-2 Cells

5. Clostridium difficile Toxin B activates the NLRP3 inflammasome in human macrophages, demonstrating a novel regulatory mechanism for the Pyrin inflammasome

6. Sulfated glycosaminoglycans and low-density lipoprotein receptor contribute to Clostridium difficile toxin A entry into cells

7. Transcriptional licensing is required for Pyrin inflammasome activation in human macrophages and bypassed by mutations causing familial Mediterranean fever

8. TcdB of Clostridioides difficile Mediates RAS-Dependent Necrosis in Epithelial Cells

9. Genome-Wide CRISPR Screen Identifies Semaphorin 6A and 6B as Receptors for Paeniclostridium sordellii Toxin TcsL

10. Endovascular Therapy Versus Bypass Surgery as First-Line Treatment Strategies for Critical Limb Ischemia

11. The Conserved Cys-2232 in Clostridioides difficile Toxin B Modulates Receptor Binding

12. Development of Neutralizing and Non-neutralizing Antibodies Targeting Known and Novel Epitopes of TcdB of

13. One-Year Results of First-Line Treatment Strategies in Patients With Critical Limb Ischemia (CRITISCH Registry)

14. Evidence for an Adaptation of a Phage-Derived Holin/Endolysin System to Toxin Transport in

15. Difference in Mono-O-Glucosylation of Ras Subtype GTPases Between Toxin A and Toxin B From

16. The Binary Toxin CDT of

17. Early cell death induced by Clostridium difficile TcdB: Uptake and Rac1-glucosylation kinetics are decisive for cell fate

18. Clostridium difficile toxin B inhibits the secretory response of human mast cell line-1 (HMC-1) cells stimulated with high free-Ca2+ and GTPγS

19. Outcomes of dialysis patients with critical limb ischemia after revascularization compared with patients with normal renal function

20. Association between statin therapy and amputation-free survival in patients with critical limb ischemia in the CRITISCH registry

21. Pyknotic cell death induced byClostridium difficile TcdB: chromatin condensation and nuclear blister are induced independently of the glucosyltransferase activity

22. Human mast cell line-1 (HMC-1) cells exhibit a membrane capacitance increase when dialysed with high free-Ca2+ and GTPγS containing intracellular solution

23. Substrate Specificity of Clostridial Glucosylating Toxins and Their Function on Colonocytes Analyzed by Proteomics Techniques

24. Glucosyltransferase-dependent and -independent effects of TcdB on the proteome of HEp-2 cells

25. Overexpression of the Endosomal Anion/Proton Exchanger ClC-5 Increases Cell Susceptibility toward

26. Receptors and Binding Structures for Clostridium difficile Toxins A and B

27. Frizzled proteins are colonic epithelial receptors for C. difficile toxin B

28. Toxin A of the nosocomial pathogen Clostridium difficile induces primary effects in the proteome of HEp-2 cells

29. The spinal muscular atrophy disease protein SMN is linked to the rho-kinase pathway via profilin

30. Down-regulation of interleukin-16 in human mast cells HMC-1 by Clostridium difficile toxins A and B

31. Serine-71 phosphorylation of Rac1/Cdc42 diminishes the pathogenic effect ofClostridium difficiletoxin A

32. Killing of Rat Basophilic Leukemia Cells by Lethal Toxin from Clostridium sordellii: Critical Role of Phosphatidylinositide 3′-OH Kinase/Akt Signaling

33. Glucosylation of Rho GTPases by Clostridium difficile toxin A triggers apoptosis in intestinal epithelial cells

34. Fatal haemorrhage due to extensive fragility of medium- and large-sized arteries and veins in a young patient with neurofibromatosis 1

35. Difference in the Cytotoxic Effects of Toxin B fromClostridium difficileStrain VPI 10463 and Toxin B from VariantClostridium difficileStrain 1470

36. In-hospital outcomes in patients with critical limb ischemia and end-stage renal disease after revascularization

37. Current practice of first-line treatment strategies in patients with critical limb ischemia

38. Autorenverzeichnis

39. In-situ-Rekonstruktion mit alloplastischen Prothesen beim Gefäßinfekt

40. Application of Mutated Clostridium difficile Toxin A for Determination of Glucosyltransferase-Dependent Effects

41. ?2-Adrenoceptor-mediated suppression of human intestinal mast cell functions is caused by disruption of filamentous actin dynamics

42. Comparison of wild type with recombinant Clostridium difficile toxin A

43. Screening und Surveillance des abdominellen Aortenaneurysmas

44. Regulation of mastoparan-induced increase of paracellular permeability in T84 cells by RhoA and basolateral potassium channels

45. Indikationen für Stents in der Arteria femoralis – Sicht des Chirurgen

46. The complete receptor-binding domain of Clostridium difficile toxin A is required for endocytosis

47. Quantification of small GTPase glucosylation by clostridial glucosylating toxins using multiplexed MRM analysis

48. Clostridium difficile toxin B inhibits the secretory response of human mast cell line-1 (HMC-1) cells stimulated with high free-Ca²⁺ and GTPγS

49. Human neutrophils are activated by a peptide fragment of Clostridium difficile toxin B presumably via formyl peptide receptor

50. Time-resolved cellular effects induced by TcdA from Clostridium difficile