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This article aims to contribute to the fields of emotional geographies and geographies of sexualities by exploring the relationship between emotions and gender, sexuality, and ‘race’ in sexualised night-time leisure spaces. By drawing on ethnographic research conducted in Manchester's Gay Village, the article highlights the importance of taking into account intersections of social identities when exploring how people feel in certain spaces. It explores how relations of ‘othering’ work through emotions, in particular how people are othered through feelings of comfort and safety. Whilst these feelings are triggered by a particular reading of bodies and spaces, they also produce bodies and spaces that are gendered, sexualised, and racialised (and classed). The article offers a rethinking of comfort and safety as not just feelings individuals have but as being constitutive of sexual, gender, and racial subjectivities and spaces.

This chapter explores intersections of sexuality and ‘race’ by drawing on different projects: ethnographic research conducted in Manchester’s Gay Village and observations gathered through voluntary work with the grassroots organisation Lesbian Immigration Support Group (LISG) in Manchester. The chapter demonstrates that in both night-time lesbian leisure spaces and the asylum system in the UK, belonging to the group ‘lesbian’ is inherently racialised. In the Gay Village, some bodies are perceived and constructed as the ‘somatic norm’ while others are (made) ‘out of place’. The chapter argues that this norm is produced through representations, door policies, and other spatial practices. In the asylum system, for asylum claims based on sexuality to be successful the claimant must ‘prove’ their sexuality. The majority of sexuality cases are refused on grounds of credibility, where the claimant is not believed to be lesbian. The chapter demonstrates that these claims have a higher chance of success if the claimant conforms to homonormative notions that are racialised and constructed around a Western model of sexuality that is ‘out and proud’.

The mechanism of insulin resistance in obesity was examined in ten obese (BMI 33 +/- 1 kg/m2) and nine lean (BMI 22 +/- 1 kg/m2) Caucasian women during a hyperglycemic-hyperinsulinemic clamp using 13C and 31P nuclear magnetic resonance (NMR) spectroscopy to measure rates of muscle glycogen synthesis and intramuscular glucose-6-phosphate (G-6-P) concentrations. Under similar steady-state plasma concentrations of glucose (approximately 11 mmol/l) and insulin (approximately 340 pmol/l), rates of muscle glycogen synthesis were reduced approximately 70% in the obese subjects (52 +/- 8 micromol/[l muscle-min]) as compared with the rates in the lean subjects (176 +/- 22 micromol/[l muscle-min]; P < 0.0001). Basal concentrations of intramuscular G-6-P were similar in the obese and lean subjects; but during the clamp, G-6-P failed to increase in the obese group (deltaG-6-P obese 0.044 +/- 0.011 vs. lean 0.117 +/- 0.011 mmol/l muscle; P < 0.001), reflecting decreased muscle glucose transport and/or phosphorylation activity. We conclude that insulin resistance in obesity can be mostly attributed to impairment of insulin-stimulated muscle glycogen synthesis due to a defect in glucose transport and/or phosphorylation activity.