Search

Trauma brain injury is a heterogeneous and dynamic entity characterized, whatever its etiology, by a decrease in cerebral perfusion the first hours after the impact. Brain injury due to hypoxia can occur after trauma, so monitoring brain hypoxia, metabolic dysfunction, intracranial hypertension and seizure activity must be detected early to prevent brain sequelae. Neuromonitoring will detect those anomalies that could compromise the adequate oxygen supply and substrates of cerebral metabolism. Despite cerebral oximetry monitoring has increased in recent years, unfortunately very limited in our country, neuromonitoring is often based on intracranial pressure and cerebral perfusion pressure, insufficient to measure cerebral oxygenation. The objective of this review is to integrate the pathophysiology of trauma brain injury with the different neuromonitoring techniques to provide an updated and more individualized management that improves the prognosis of neurocritical patients. [ABSTRACT FROM AUTHOR]

The Delayed Hypoxic Leukoencephalopathy is a rare and still unknown pathogenesis process that provokes the appearance of acute confusional symptomatology and cognitive deterioration after a period of 2 to 10 days of apparent complete recovery from hypoxia. In some cases, acute psychiatric symptoms, including apathy, anxiety and even the development of psychosis, have been observed. Mainly associated with carbon monoxide poisoning, these symptoms have also been found as secondary to self-poisoning with toxic and psychotropic drugs that cause prolonged hypotensive episodes. The number of published cases and the literature on the subject is really scanty. The rare case, within four years, of two patients with a prior mental disorder who entered Psychiatric Inpatients Unit after autointoxication, is collected in these clinical notes. They develop, from a psychiatric perspective, the concepts of this unknown clinic entity and the inevitable passage from the psychiatric approach to neurological. [ABSTRACT FROM AUTHOR]

Copyright of CES Medicina is the property of Universidad CES and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Objective: this study was designed to characterize the differences between normal children and those with neonatal encephalopathy, regarding the brain auditory evoked potentials (BAEPs), these differences may be which may be used as an exploratory guide of the brain stem condition. Methods. A sample of 34 infants suffering from neonatal encephalopathy (NE) and 34 healthy children were studied. Using the BAEP technique, the latencies and amplitudes of the waves I, II, III, and V, were measured, as well as the intervals I-III, III-V, and I-V. The differences between groups were evaluated through a analysis of variance and a conglomerate analysis. Results: the latencies of the waves I, II, and V, were more prolonged in the group of children with NE than in the group of healthy children, and the amplitudes of the NE group were larger than controls. The intervals I-III and I-V and waves III and V, were also different in both groups. The conglomerate analysis formed three different functional profiles in alI children: rapid, intermediate, and a slow time responders. Slow responders were exclusively found in children with NE. Conclusions. The BAEPs of infants with NE showed larger wave latencies and increased wave amplitudes, when compared to normal children. These statistical analyses classified them in different functional profiles, which may be useful in detecting children with greater risk of neurological sequels. [ABSTRACT FROM AUTHOR]

Copyright of Universitas Médica is the property of Pontificia Universidad Javeriana and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Cirugía y Cirujanos is the property of Publicidad Permanyer SLU and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)