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The extent to which chronic dialysis alters the serum copper and manganese content was examined. The copper content was not significantly altered, whereas the level of manganese in the blood rose highly significantly. This manganese originates from the dialysis fluid. The behaviour of the manganese level during dialysis treatment suggests that the element is deposited in the body. Its transport is clearly connected with protein, since the manganese is not found in the ultrafiltrate. The is no fear of manganese intoxication.

Nitrate and nitrite compounds in feedstuffs were readily and immediately absorbed from the digestive tract right after intake. Nitrate, therefore, is reflected in the blood, depending on intake quantity. Nitrate concentrations recorded from the blood are superior to values established from feedstuffs in substantiating conclusions as to intake quantity. A diagnosis of nitrate intoxication, based on feedstuff testing, should be confirmed by blood testing. Two methods are proposed for quantity determination of nitrate levels in feedstuffs and blood. The presence of hypomagnesaemia should be ruled out in any differential diagnosis undertaken for suspicion of nitrate intoxication.