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Unlabelled: BACKGROUND, PATIENT RESPECTIVELY AND METHODS: Based on a case-report we demonstrate which symptoms are indicative of a cerebral sinus-venous thrombosis, how it can develop, and which diagnostic and therapeutic methods are available., Results: Cerebral sinus-venous thrombosis in puerperium can be hidden behind unspecific symptoms like strong headache, nausea, vomiting. Literature reorts varying mortality between 5 and 30%., Conclusions: The obstetrician has to be attentive whenever these symptoms appear for the course and prognosis decisively depend on a quick diagnosis by means of computertomography and angiography., Discussion: Blood coagulation disorders such as lack of Antithrombin III, protein C, or protein S, have been identified as a particularly important origin of the cerebral sinus-venous thrombosis. It is subject to discussion whether the patient should be advised against any further pregnancy. From a medical point of view, a subsequent pregnancy seems acceptable because the risk of another thrombosis, which is a rare event anyway, can be significantly reduced by providing anticoagulation substances.

In a balance trial with 10 pigs (mean body mass 50 kg) the influence of a bacterial protein supplement (Alcaligenes eutrophus) on N-metabolism was investigated. The bacteria were included into the diet at levels of 7 and 14% at the expense of extracted soyabean meal. Thus bacterial "pure protein" (bacterial non-nucleic acid N X X 6.25) amounted to 30 and 60% of the protein of the ration. Consuming 2 kg of feed dry matter per day the animals of the control group (I) and the experimental groups (II and III) ingested 48 g, 52 g and 55 g of total N respectively. The difference in N-intake is explained by the additional nucleic acid-N, amounting to 19,8% of total bacterial N. Daily weight gain (on average 1054 g) and feed conversion efficiency (feed ingested/weight gain; on average 1,9) were relatively improved at the highest dietary level of bacterial cell mass. Faecal N-excretion was increased significantly, whereas renal N-excretion remained unchanged. Mean apparent N-digestibility was 87,4% showing no significant difference between the experimental groups. N-balance values were noticibly increased following the intake of the bacterial protein supplement. The excretion of urinary urea-N was slightly reduced whereas 4-6 times as much allantoin-N was excreted when bacteria were fed. It is calculated that about 80% of the bacterial purines are renally excreted as allantoin and uric acid.