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Introduction: In third cranial nerve palsy, the lack of mydriasis evokes a noncompressive mechanism. Case report. We report a case of a slightly painful, pure extrinsic third-nerve palsy, complete except for the partial ptosis secondary to the compression by an intracavernous carotid aneurysm. Percutaneous endovascular embolization was followed by complete regression of the palsy within 4 weeks. The pupil may have been spared by the mechanism of compression after the separation of the pupillomotor and extrinsic fibers or the ischemia of the third nerve by its arterial trunk lesion., Conclusion: This case report underlines that brain radiological explorations are necessary in some third-nerve pupil-sparing palsies.

Introduction: Infection of the central nervous system with human immunodeficiency virus (HIV) can be associated with movement disorders., Case Report: A case of chorea during HIV encephalitis which responded well to antiretroviral therapy is reported. Choreic movements disappeared with a decrease of MRI lesions observed in basal ganglia., Conclusion: The efficacy of anti-retroviral therapy in choreic movements, a rare syndrome with HIV encephalitis, can be underlined.

Verapamil poisonings are rare, but are associated with a high proportion of deaths. The authors present a case of acute poisoning by verapamil associated with collapsus and a high degree of heart block. They propose a strategy for therapeutic management based on the study of calcium-channel blocker mechanisms of toxicity.

Since the original report, acute paralysis and albuminocytologic dissociation have remained hallmarks of the Guillain-Barré syndrome but the initially described favorable outcome with complete motor recovery is not always observed. Guillain-Barré syndrome can be complicated by early respiratory distress, dysautonomia and late functional impairment. The conduction block induced by acute demyelination accounts for the spontaneous neurological improvement. On the other hand, early axonal damage or sustained demyelination can be responsible for residual deficit. The cause and mechanism of the disease still remain unclear. For these patients, general care is essential and should be provided in appropriate hospital units. Today, specific treatment consists of either plasma exchange or high-dose intravenous immunoglobulins; the choice depends on their respective contraindications.