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7. AKT-mTORC1 reactivation is the dominant resistance driver for PI3Kβ/AKT inhibitors in PTEN-null breast cancer and can be overcome by combining with Mcl-1 inhibitors

9. The CADM1 tumor suppressor gene is a major candidate gene in MDS with deletion of the long arm of chromosome 11

12. ARID1A influences HDAC1/BRD4 activity, intrinsic proliferative capacity and breast cancer treatment response

13. Minimal genome-wide human CRISPR-Cas9 library

17. Prioritization of cancer therapeutic targets using CRISPR–Cas9 screens

18. Constitutively Active SMAD2/3 Are Broad-Scope Potentiators of Transcription-Factor-Mediated Cellular Reprogramming

21. Genome‐wide loss‐of‐function genetic screen identifies INSIG2 as the vulnerability of hepatitis B virus‐integrated hepatoma cells.

22. Agreement between two large pan-cancer CRISPR-Cas9 gene dependency data sets

23. Functional linkage of gene fusions to cancer cell fitness assessed by pharmacological and CRISPR-Cas9 screening

25. Author Correction: ARID1A influences HDAC1/BRD4 activity, intrinsic proliferative capacity and breast cancer treatment response

26. ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forks

27. SRPK1 maintains acute myeloid leukemia through effects on isoform usage of epigenetic regulators including BRD4

30. A Genetic Progression Model of BrafV600E-Induced Intestinal Tumorigenesis Reveals Targets for Therapeutic Intervention

35. PiggyBac Transposon Mutagenesis: A Tool for Cancer Gene Discovery in Mice

39. Targeted gene correction of α1-antitrypsin deficiency in induced pluripotent stem cells

40. Genome-wide phenotype analysis in ES cells by regulated disruption of Bloom's syndrome gene

43. Rad54 is dispensable for the ALT pathway

44. A CRISPR Dropout Screen Identifies Genetic Vulnerabilities and Therapeutic Targets in Acute Myeloid Leukemia

46. SRPK1 Is a Therapeutic Vulnerability in Acute Myeloid Leukemia through Its Effects on Alternative Isoforms of Epigenetic Regulators Including BRD4

48. Mutational History of a Human Cell Lineage from Somatic to Induced Pluripotent Stem Cells.

50. A Crispr/Cas9 Drop-out Screen Identifies Genome-Wide Genetic Valnerubilities in Acute Myeloid Leukaemia

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