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387 results on '"Tsoi, Lam C"'

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3. Systems-based identification of the Hippo pathway for promoting fibrotic mesenchymal differentiation in systemic sclerosis

5. Secondary ACMG and non-ACMG genetic findings in a multiethnic cohort of 16,713 pediatric participants

8. Genome-wide association meta-analysis identifies risk loci for abdominal aortic aneurysm and highlights PCSK9 as a therapeutic target

11. Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis

18. Single-cell profiling of prurigo nodularis demonstrates immune-stromal crosstalk driving profibrotic responses and reversal with nemolizumab

19. Single-cell sequencing reveals Hippo signaling as a driver of fibrosis in hidradenitis suppurativa

22. Macrophage-specific inhibition of the histone demethylase JMJD3 decreases STING and pathologic inflammation in diabetic wound repair

26. Gene Expression Signatures in Inflammatory and Sclerotic Morphea Skin and Sera Distinguish Morphea from Systemic Sclerosis

28. Noninvasive Tape-Stripping with High-Resolution RNA Profiling Effectively Captures a Preinflammatory State in Nonlesional Psoriatic Skin

32. Diabetic Wound Keratinocytes Induce Macrophage JMJD3-Mediated Nlrp3 Expression via IL-1R Signaling.

34. Genome-Wide Association Study in Acute Tubulointerstitial Nephritis

35. GATA-3 is a proto-oncogene in T-cell lymphoproliferative neoplasms

36. IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses

38. Keratinocytes sense and eliminate CRISPR DNA through STING/IFN-[kappa] activation and APOBEC3G induction

40. The cellular architecture of the antimicrobial response network in human leprosy granulomas

41. Progression of acute-to-chronic atopic dermatitis is associated with quantitative rather than qualitative changes in cytokine responses

44. KLK6 expression in skin induces PAR1-mediated psoriasiform dermatitis and inflammatory joint disease

45. Atopic Dermatitis Is an IL-13–Dominant Disease with Greater Molecular Heterogeneity Compared to Psoriasis

48. Neutrophil Subsets, Platelets, and Vascular Disease in Psoriasis

49. Direct cellular reprogramming enables development of viral T antigen-driven Merkel cell carcinoma in mice

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