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109 results on '"Quek, Lynn"'

1. Chromatin accessibility governs the differential response of cancer and T cells to arginine starvation

Author

Crump, Nicholas T., Hadjinicolaou, Andreas V., Xia, Meng, Walsby-Tickle, John, Gileadi, Uzi, Chen, Ji-Li, Setshedi, Mashiko, Olsen, Lars R., Lau, I-Jun, Godfrey, Laura, Quek, Lynn, Yu, Zhanru, Ballabio, Erica, Barnkob, Mike B., Napolitani, Giorgio, Salio, Mariolina, Koohy, Hashem, Kessler, Benedikt M., Taylor, Stephen, Vyas, Paresh, McCullagh, James S.O., Milne, Thomas A., and Cerundolo, Vincenzo

Published
2021
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3. Integrating clinical features with genetic factors enhances survival prediction for adults with acute myeloid leukemia

Author

Silveira, Douglas R.A., Quek, Lynn, Santos, Itamar S., Corby, Anna, Coelho-Silva, Juan L., Pereira-Martins, Diego A., Vallance, Grant, Brown, Benjamin, Nardinelli, Luciana, Silva, Wellington F., Velloso, Elvira D.R.P., Lucena-Araujo, Antonio R., Traina, Fabiola, Peniket, Andy, Vyas, Paresh, Rego, Eduardo M., Bendit, Israel, and Rocha, Vanderson

Published
2020
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4. Molecular mechanisms mediating relapse following ivosidenib monotherapy in IDH1-mutant relapsed or refractory AML

Author

Choe, Sung, Wang, Hongfang, DiNardo, Courtney D., Stein, Eytan M., de Botton, Stéphane, Roboz, Gail J., Altman, Jessica K., Mims, Alice S., Watts, Justin M., Pollyea, Daniel A., Fathi, Amir T., Tallman, Martin S., Kantarjian, Hagop M., Stone, Richard M., Quek, Lynn, Konteatis, Zenon, Dang, Lenny, Nicolay, Brandon, Nejad, Parham, Liu, Guowen, Zhang, Vickie, Liu, Hua, Goldwasser, Meredith, Liu, Wei, Marks, Kevin, Bowden, Chris, Biller, Scott A., Attar, Eyal C., and Wu, Bin

Published
2020
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5. TP53 mutation status divides myelodysplastic syndromes with complex karyotypes into distinct prognostic subgroups

Author

Haase, Detlef, Stevenson, Kristen E., Neuberg, Donna, Maciejewski, Jaroslaw P., Nazha, Aziz, Sekeres, Mikkael A., Ebert, Benjamin L., Garcia-Manero, Guillermo, Haferlach, Claudia, Haferlach, Torsten, Kern, Wolfgang, Ogawa, Seishi, Nagata, Yasunobu, Yoshida, Kenichi, Graubert, Timothy A., Walter, Matthew J., List, Alan F., Komrokji, Rami S., Padron, Eric, Sallman, David, Papaemmanuil, Elli, Campbell, Peter J., Savona, Michael R., Seegmiller, Adam, Adès, Lionel, Fenaux, Pierre, Shih, Lee-Yung, Bowen, David, Groves, Michael J., Tauro, Sudhir, Fontenay, Michaela, Kosmider, Olivier, Bar-Natan, Michal, Steensma, David, Stone, Richard, Heuser, Michael, Thol, Felicitas, Cazzola, Mario, Malcovati, Luca, Karsan, Aly, Ganster, Christina, Hellström-Lindberg, Eva, Boultwood, Jacqueline, Pellagatti, Andrea, Santini, Valeria, Quek, Lynn, Vyas, Paresh, Tüchler, Heinz, Greenberg, Peter L., Bejar, Rafael, and for the International Working Group for MDS Molecular Prognostic Committee

Published
2019
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7. Clonal heterogeneity of acute myeloid leukemia treated with the IDH2 inhibitor enasidenib

Author

Quek, Lynn, David, Muriel D., Kennedy, Alison, Metzner, Marlen, Amatangelo, Michael, Shih, Alan, Stoilova, Bilyana, Quivoron, Cyril, Heiblig, Maël, Willekens, Christophe, Saada, Véronique, Alsafadi, Samar, Vijayabaskar, M. S., Peniket, Andy, Bernard, Oliver A., Agresta, Sam, Yen, Katharine, MacBeth, Kyle, Stein, Eytan, Vassiliou, George S., Levine, Ross, De Botton, Stephane, Thakurta, Anjan, Penard-Lacronique, Virginie, and Vyas, Paresh

Published
2018
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10. Single-cell analysis reveals the continuum of human lympho-myeloid progenitor cells

Author

Karamitros, Dimitris, Stoilova, Bilyana, Aboukhalil, Zahra, Hamey, Fiona, Reinisch, Andreas, Samitsch, Marina, Quek, Lynn, Otto, Georg, Repapi, Emmanouela, Doondeea, Jessica, Usukhbayar, Batchimeg, Calvo, Julien, Taylor, Stephen, Goardon, Nicolas, Six, Emmanuelle, Pflumio, Francoise, Porcher, Catherine, Majeti, Ravindra, Göttgens, Berthold, and Vyas, Paresh

Published
2018
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11. Clinical and biological implications of driver mutations in myelodysplastic syndromes

Author

Papaemmanuil, Elli, Gerstung, Moritz, Malcovati, Luca, Tauro, Sudhir, Gundem, Gunes, Van Loo, Peter, Yoon, Chris J., Ellis, Peter, Wedge, David C., Pellagatti, Andrea, Shlien, Adam, Groves, Michael John, Forbes, Simon A., Raine, Keiran, Hinton, Jon, Mudie, Laura J., McLaren, Stuart, Hardy, Claire, Latimer, Calli, Della Porta, Matteo G., O'Meara, Sarah, Ambaglio, Ilaria, Galli, Anna, Butler, Adam P., Walldin, Gunilla, Teague, Jon W., Quek, Lynn, Sternberg, Alex, Gambacorti-Passerini, Carlo, Cross, Nicholas C.P., Green, Anthony R., Boultwood, Jacqueline, Vyas, Paresh, Hellstrom-Lindberg, Eva, Bowen, David, Cazzola, Mario, Stratton, Michael R., and Campbell, Peter J.

Published
2013
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15. Coexistence of LMPP-like and GMP-like Leukemia Stem Cells in Acute Myeloid Leukemia

Author

Goardon, Nicolas, Marchi, Emanuele, Atzberger, Ann, Quek, Lynn, Schuh, Anna, Soneji, Shamit, Woll, Petter, Mead, Adam, Alford, Kate A., Rout, Raj, Chaudhury, Salma, Gilkes, Amanda, Knapper, Steve, Beldjord, Kheira, Begum, Suriya, Rose, Susan, Geddes, Nicola, Griffiths, Mike, Standen, Graham, Sternberg, Alexander, Cavenagh, Jamie, Hunter, Hannah, Bowen, David, Killick, Sally, Robinson, Lisa, Price, Andrew, Macintyre, Elizabeth, Virgo, Paul, Burnett, Alan, Craddock, Charles, Enver, Tariq, Jacobsen, Sten Eirik W., Porcher, Catherine, and Vyas, Paresh

Published
2011
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16. Co-occurrence of DNMT3A, NPM1, FLT3 mutations identifies a subset of acute myeloid leukemia with adverse prognosis

Author

Bezerra, Matheus F., Lima, Aleide S., Piqué-Borràs, Maria-Riera, Silveira, Douglas R., Coelho-Silva, Juan L., Pereira-Martins, Diego A., Weinhäuser, Isabel, Franca-Neto, Pedro L., Quek, Lynn, Corby, Anna, Oliveira, Mayara M., Lima, Marinus M., de Assis, Reijane A., de Melo Campos, Paula, Duarte, Bruno K., Bendit, Israel, Rocha, Vanderson, Rego, Eduardo M., Traina, Fabiola, Saad, Sara T., Beltrão, Eduardo I., Bezerra, Marcos A., and Lucena-Araujo, Antonio R.

Published
2020
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18. CEBPA-Driven Expression of the Transcriptionally Inactive deltaTP73 Isoform Phenocopies TP53mutated Poor Risk and Drug-Resistant Acute Myeloid Leukemia

Author

Pereira-Martins, Diego, Ortiz Rojas, César Alexander, Weinhaeuser, Isabel, Wierenga, Bart-Jan, Van Den Boom, Vincent, Mojallali, Fatemeh, Sternadt, Dominique, Bianco, Thiago Mantello, van der Meer, Nisha, Silveira, Douglas RA, Quek, Lynn, Ammatuna, Emanuele, Lucena-Araujo, Antonio R, Huls, Gerwin A., Rego, Eduardo Magalhães, and Schuringa, Jan Jacob

Published
2023
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19. An Integrated Single Cell Immunogenomic Atlas Reveals Pathways of Clonal Expansion, Inflammation and Dyserythropoiesis in Low and High-Risk Clonal Haematopoiesis

Author

Zheng, Xuesen, Tan, Amandine, Casares Alaez, Maria del Pilar, Mallett, Garry, Kaya, Deniz Ece, Seymen, Nogayhan, Rzepkowska, Marta, Mackie, Sarah, Chatzikyriakou, Prodromos, Bogaanay, Laarni, Lewis, Jen, Hasan, Maroof, Suragani, Rajasekhar N.V.S., Carmichael, James, Mufti, Ghulam, Gandhi, Anita K., Napolitani, Giorgio, and Quek, Lynn

Published
2023
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20. Metabolism-Related Features Identify the Combination Metformin Plus NAMPT Inhibitors As a Selective Treatment Strategy in Acute Myeloid Leukemia

Author

Weinhaeuser, Isabel, Pereira-Martins, Diego, Sternadt, Dominique, Griessinger, Emmanuel F, Silveira, Douglas RA, Coelho-Silva, Juan L, Alves-Silva, Antonio Bruno, Erdem, Ayşegül, Duarte, Bruno Koza Lino, Quek, Lynn, Traina, Fabiola, Saad, Sara Teresinha Olalla, Hilberink, Jacobien R, Ammatuna, Emanuele, Rego, Eduardo Magalhães, Huls, Geert A., Lucena-Araujo, Antonio R, and Schuringa, Jan Jacob

Published
2023
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23. Heterogeneity of human lympho-myeloid progenitors at the single cell level

Author

Karamitros, Dimitris, Stoilova, Bilyana, Aboukhalil, Zahra, Hamey, Fiona, Reinisch, Andreas, Samitsch, Marina, Quek, Lynn, Otto, Georg, Repapi, Emmanouela, Doondeea, Jessica, Usukhbayar, Batchimeg, Calvo, Julien, Taylor, Stephen, Goardon, Nicolas, Six, Emmanuelle, Pflumio, Francoise, Porcher, Catherine, Majeti, Ravindra, Gottgens, Berthold, and Vyas, Paresh

Subjects
Gene Expression Profiling, Transplantation, Heterologous, Hematopoietic Stem Cell Transplantation, Cell Differentiation, Cell Separation, Lymphoid Progenitor Cells, Article, Hematopoiesis, Mice, Animals, Humans, Cell Lineage, Single-Cell Analysis, Cells, Cultured, Myeloid Progenitor Cells
Abstract

The hierarchy of human hemopoietic progenitor cells that produce lymphoid and granulocytic-monocytic (myeloid) lineages is unclear. Multiple progenitor populations produce lymphoid and myeloid cells, but they remain incompletely characterized. Here we demonstrated that lympho-myeloid progenitor populations in cord blood - lymphoid-primed multi-potential progenitors (LMPPs), granulocyte-macrophage progenitors (GMPs) and multi-lymphoid progenitors (MLPs) - were functionally and transcriptionally distinct and heterogeneous at the clonal level, with progenitors of many different functional potentials present. Although most progenitors had the potential to develop into only one mature cell type ('uni-lineage potential'), bi- and rarer multi-lineage progenitors were present among LMPPs, GMPs and MLPs. Those findings, coupled with single-cell expression analyses, suggest that a continuum of progenitors execute lymphoid and myeloid differentiation, rather than only uni-lineage progenitors' being present downstream of stem cells.

Published
2017

24. The immune landscape in BCR‐ABL negative myeloproliferative neoplasms: inflammation, infections and opportunities for immunotherapy.

Author

Strickland, Marie, Quek, Lynn, and Psaila, Bethan

Subjects
*MYELOPROLIFERATIVE neoplasms, *HEMATOPOIETIC stem cells, *IMMUNOREGULATION, *EMERGING infectious diseases, *INFLAMMATION, *INFECTION
Abstract

Summary: Breakpoint cluster region‐Abelson (BCR‐ABL) negative myeloproliferative neoplasms (MPNs) are chronic myeloid neoplasms initiated by the acquisition of gene mutation(s) in a haematopoietic stem cell, leading to clonal expansion and over‐production of blood cells and their progenitors. MPNs encompass a spectrum of disorders with overlapping but distinct molecular, laboratory and clinical features. This includes polycythaemia vera, essential thrombocythaemia and myelofibrosis. Dysregulation of the immune system is key to the pathology of MPNs, supporting clonal evolution, mediating symptoms and resulting in varying degrees of immunocompromise. Targeting immune dysfunction is an important treatment strategy. In the present review, we focus on the immune landscape in patients with MPNs – the role of inflammation in disease pathogenesis, susceptibility to infection and emerging strategies for therapeutic immune modulation. Further detailed work is required to delineate immune perturbation more precisely in MPNs to determine how and why vulnerability to infection differs between clinical subtypes and to better understand how inflammation results in a competitive advantage for the MPN clone. These studies may help shed light on new designs for disease‐modifying therapies. [ABSTRACT FROM AUTHOR]

Published
2022
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28. Dysregulation of Stem-Progenitor and Differentiation Programmes through Modulation of Bivalent Chromatin Drives Leukemogenesis in Mutant IDH2 Acute Myeloid Leukaemia

Author

Silveira, Douglas RA RA, Chatzikyriakou, Prodromos, Casares Alaez, Maria del Pilar, Mackie, Sarah, Hulks, Roan, Yavorska, Olena, Siejka-Zielinska, Paulina, Metzner, Marlen, Smith, Alastair, Usukhbayar, Batchimeg, Penard-Lacronique, Virginie, Song, Chunxiao, Gandhi, Anita K., Hasan, Maroof, Vyas, Paresh, Milne, Thomas A., Kriaucionis, Skirmantas, and Quek, Lynn

Published
2022
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29. High MN1 Expression Is Associated with an Lspc-Enriched Phenotype and Glycolysis Representing a New Vulnerability in Acute Myeloid Leukemia

Author

Pereira-Martins, Diego, van Dijk, Noortje, Weinhaeuser, Isabel, Silveira, Douglas RA, Coelho Da Silva, Juan, Bianco, Thiago M, Ortiz, César Alexander, Traina, Fabiola, Figueiredo-Pontes, Lorena L, Ammatuna, Emanuele, Quek, Lynn, Huls, Gerwin A., Lucena-Araujo, Antonio R., Rego, Eduardo M, and Schuringa, Jan Jacob

Published
2022
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31. M2 Macrophages Drive Resistance to Phagocytosis and Improve Mitochondrial Metabolism in Acute Myeloid Leukemia Facilitating Leukemic Transformation and In Vivo Engraftment

Author

Weinhaeuser, Isabel, Pereira-Martins, Diego, Yamamoto de Almeida, Luciana, Hilberink, Jacobien R, Silveira, Douglas RA, Quek, Lynn, Ortiz, César Alexander, Lucena-Araujo, Antonio R., Visser, Nienke, Ammatuna, Emanuele, Huls, Gerwin A., Rego, Eduardo M, and Schuringa, Jan Jacob

Published
2022
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32. British Society for Haematology guidelines for the management of adult myelodysplastic syndromes.

Author

Killick, Sally B., Ingram, Wendy, Culligan, Dominic, Enright, Helen, Kell, Jonathan, Payne, Elspeth M., Krishnamurthy, Pramila, Kulasekararaj, Austin, Raghavan, Manoj, Stanworth, Simon J., Green, Simone, Mufti, Ghulam, Quek, Lynn, Cargo, Catherine, Jones, Gail L., Mills, Juliet, Sternberg, Alex, Wiseman, Daniel H., and Bowen, David

Subjects
MYELODYSPLASTIC syndromes, ADULTS, PURE red cell aplasia, HEMATOPOIETIC stem cell transplantation, HEMATOLOGY, MAGNETIC resonance imaging
Abstract

Patient characteristics and donor selection Patient age I per se i is not a limiting factor for transplant.113 Careful selection of older patients (>70 years) with good performance status and low haematopoietic cell transplantation-specific comorbidity index (HCT-CI) improves outcomes.127 Patients with high-risk MDS and high comorbidity scores (HCT-CI >= 3) have the worst outcomes128 and alternative treatments should be considered. In a subgroup analysis of patients >=75 years, azacitidine also significantly improved two-year OS compared to conventional care (55% vs 15%), suggesting that this is the treatment of choice in older higher-risk MDS patients with good performance status.89 Even patients with poor-prognosis cytogenetic profiles may benefit from azacitidine treatment.90 Reliable molecular predictors of response have not been identified, although patients with poor-prognosis indicators, including I TP53 i mutations, may respond. Keywords: myelodysplastic syndromes; MDS; guideline; management EN myelodysplastic syndromes MDS guideline management 267 281 15 07/19/21 20210715 NES 210715 Scope This document represents an update of the British Society of Haematology Guideline published in 2014 due to advances in understanding the biology and therapy of the myelodysplastic syndromes (MDS).1 The objective of these guidelines is to provide healthcare professionals with clear guidance on the management of adult patients with MDS. Hypoplastic MDS Approximately 10-20% of MDS patients have decreased marrow cellularity.78 The WHO classification of myeloid neoplasm designates this hypoplastic MDS (h-MDS), although it does not assign it a distinct category.79 Hypocellularity in MDS can present diagnostic difficulties with other bone marrow failure (BMF) syndromes especially aplastic anaemia. Improved survival in MDS patients receiving iron chelation therapy - a matched pair analysis of 188 patients from the Dusseldorf MDS registry. [Extracted from the article]

Published
2021
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33. British Society for Haematology guidelines for the diagnosis and evaluation of prognosis of Adult Myelodysplastic Syndromes.

Author

Killick, Sally B., Wiseman, Daniel H., Quek, Lynn, Cargo, Catherine, Culligan, Dominic, Enright, Helen, Green, Simone, Ingram, Wendy, Jones, Gail L., Kell, Jonathan, Krishnamurthy, Pramila, Kulasekararaj, Austin, Mills, Juliet, Mufti, Ghulam, Payne, Elspeth M., Raghavan, Manoj, Stanworth, Simon J., Sternberg, Alex, and Bowen, David

Subjects
MYELODYSPLASTIC syndromes, DIAGNOSIS, PROGNOSIS, HEMATOLOGY, PAROXYSMAL hemoglobinuria, PANCYTOPENIA, MEDICAL personnel
Abstract

Keywords: myelodysplastic syndromes; MDS; guideline; diagnosis EN myelodysplastic syndromes MDS guideline diagnosis 282 293 12 07/19/21 20210715 NES 210715 Scope This document represents an update of the British Society of Haematology guideline published in 2014 due to advances in understanding the biology and therapy of the myelodysplastic syndromes (MDS).1 The objective of these guidelines is to provide healthcare professionals with clear guidance on the diagnosis and evaluation of prognosis of adult patients with MDS. Trephine section histology is especially helpful for the diagnosis of hypocellular MDS and MDS/myeloproliferative neoplasms (MPN) overlap syndromes.9 Patients with MDS/MPN overlap including CMML are now considered a distinct entity by the WHO when features of both MDS and MPN are present. Patients and family members should ideally be offered genetic counselling before genetic screening if there is a high clinical suspicion.42 Recommendations Myelodysplastic Syndromes (MDS) should be suspected in patients with otherwise unexplained cytopenias(s) or macrocytosis (1A). In the context of persistent and otherwise unexplained cytopenias, a WHO-defined diagnosis of MDS requires either (i) morphological dysplasia (involving >=10% of bone marrow cells in >=1 lineage); (ii) increased myeloblasts (>=5%, but <20%); or (iii) evidence of clonality with a typical MDS-associated cytogenetic abnormality.2,4 Dysplasia is not restricted to MDS patients and can occur following a toxic insult, in reactive conditions or secondary to haematinic deficiencies. [Extracted from the article]

Published
2021
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34. Repressed Chromatin Drives Leukaemogenesis in Mutant IDH2 Acute Myeloid Leukaemia Via Inhibition of Granulocyte Differentiation and Cell Cycle Progression

Author

Silveira, Douglas RA, Chatzikyriakou, Prodromos, Yavorska, Olena, Mackie, Sarah, Hulks, Roan, Siejka-Zielinska, Paulina, Corby, Anna, Smith, Alastair, Brown, Benjamin, Metzner, Marlen, Milne, Thomas A., Penard-Lacronique, Virginie, Song, Chunxiao, Hasan, Maroof, Thakurta, Anjan, Vyas, Paresh, Kriaucionis, Skirmantas, and Quek, Lynn

Published
2021
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39. A multicenter comparative acute myeloid leukemia study: can we explain the differences in the outcomes in resource-constrained settings?

Author

Silveira, Douglas R. A., Coelho-Silva, Juan L., Silva, Wellington F., Vallance, Grant, Pereira-Martins, Diego A., Madeira, Maria I. A., Figueredo-Pontes, Lorena L., Velloso, Elvira D. R. P., Simões, Belinda P., Peniket, Andy, Danby, Robert, Rego, Eduardo M., Vyas, Paresh, Traina, Fabiola, Bendit, Israel, Quek, Lynn, and Rocha, Vanderson

Subjects
ACUTE myeloid leukemia, GRAM-negative bacterial diseases, STEM cell transplantation, HEMATOPOIETIC stem cells, INFECTION prevention
Abstract

Outcomes in acute myeloid leukemia (AML) are dependent on patient- and disease-characteristics, treatment, and socioeconomic factors. AML outcomes between resource-constrained and developed countries have not been compared directly. We analyzed two cohorts: from São Paulo state, Brazil (USP, n = 312) and Oxford, United Kingdom (OUH, n = 158). USP cohort had inferior 5-year overall survival compared with OUH (29% vs. 49%, adjusted-p=.027). USP patients have higher early-mortality (23% vs. 6% p<.001) primarily due to multi-resistant Gram-negative bacterial and fungal infections. USP had higher 5-year cumulative incidence of relapse (60% vs. 50%, p=.0022), were less likely to undergo hematopoietic stem cell transplant (HSCT) (28% vs. 75%, p<.001) and waited longer for HSCT (median, 23.8 vs. 7.2 months, p<.001). Three-year survival in relapsed patients was worse in USP than OUH (10% vs. 39%, p<.001). Our study indicates that efforts to improve AML outcomes in Brazil should focus on infection prevention and control, and access to HSCT. [ABSTRACT FROM AUTHOR]

Published
2021
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43. Enasidenib Plus Azacitidine Significantly Improves Complete Remission and Overall Response Compared with Azacitidine Alone in Patients with Newly Diagnosed Acute Myeloid Leukemia (AML) with Isocitrate Dehydrogenase 2 (IDH2) Mutations: Interim Phase II Results from an Ongoing, Randomized Study

Author

DiNardo, Courtney D., Schuh, Andre C., Stein, Eytan M., Fernandez, Pau Montesinos, Wei, Andrew, De Botton, Stephane, Zeidan, Amer M., Fathi, Amir T., Quek, Lynn, Kantarjian, Hagop M., Frattini, Mark G., Lersch, Frederik, Gong, Jing, Franovic, Aleksandra, MacBeth, Kyle, Vyas, Paresh, and Döhner, Hartmut

Published
2019
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47. Combining Clinical Features with Genetic Factors Improves Survival Prediction for Adults with Acute Myeloid Leukemia: Validation of a New Score System in 3 Cohorts

Author

Silveira, Douglas R.A., Quek, Lynn, Corby, Anna, Coelho-Silva, Juan L, Pereira-Martins, Diego A, Vallance, Grant, Brown, Benjamin, Nardinelli, Luciana, Silva, Wellington F, Velloso, Elvira DRP, Traina, Fabiola, Peniket, Andy, Vyas, Paresh, Rego, Eduardo M, Bendit, Israel, and Rocha, Vanderson

Published
2019
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49. A Novel Model of Human Hemopoiesis Based on Single Cell Functional and Transcriptional Analysis of Lympho-Myeloid Progenitors

Author

Stoilova, Bilyana, Karamitros, Dimitris, Aboukhalil, Zahra, Hamey, Fiona, Reinisch, Andreas, Samitsch, Marina, Quek, Lynn, Otto, Georg, Repapi, Emmanouela, Doondeea, Jessica, Usukhbayar, Batchimeg, Calvo, Julien, Taylor, Stephen, Goardon, Nicolas, Six, Emmanuelle, Pflumio, Francoise, Porcher, Catherine, Majeti, Ravi, Gottgens, Berthold, and Vyas, Paresh

Published
2017
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50. Clonal Heterogeneity in Differentiation Response and Resistance to the IDH2 Inhibitor Enasidenib in Acute Myeloid Leukemia

Author

Quek, Lynn, David, Muriel, Kennedy, Alison, Metzner, Marlen, Amatangelo, Michael, Shih, Alan H., Stoilova, Bilyana, Karamitros, Dimitris, Quivoron, Cyril, Heiblig, Mael, Willekens, Christophe, Saada, Veronique, Peniket, Andrew, Bernard, Olivier, Agresta, Sam, Yen, Katharine, Stein, Eytan M., De Botton, Stephane, Thakurta, Anjan, Levine, Ross L., Penard-Lacronique, Virginie, and Vyas, Paresh

Published
2017
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