188 results on '"Oh, S. Paul"'
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2. BMP10 functions independently from BMP9 for the development of a proper arteriovenous network
3. Bone Marrow-Derived Alk1 Mutant Endothelial Cells and Clonally Expanded Somatic Alk1 Mutant Endothelial Cells Contribute to the Development of Brain Arteriovenous Malformations in Mice
4. Correcting Smad1/5/8, mTOR, and VEGFR2 treats pathology in hereditary hemorrhagic telangiectasia models
5. TMEM100 is a key factor for specification of lymphatic endothelial progenitors
6. Pazopanib may reduce bleeding in hereditary hemorrhagic telangiectasia
7. Reduced activin receptor-like kinase 1 activity promotes cardiac fibrosis in heart failure
8. Chapter 41 - Molecular Biology and Novel Treatments of Intracranial AVMs
9. Overexpression of Activin Receptor-Like Kinase 1 in Endothelial Cells Suppresses Development of Arteriovenous Malformations in Mouse Models of Hereditary Hemorrhagic Telangiectasia
10. Increasing brain angiotensin converting enzyme 2 activity decreases anxiety-like behavior in male mice by activating central Mas receptors
11. Highlights of the 13th International Hereditary Hemorrhagic Telangiectasia Scientific conference
12. Conditional knockout of activin like kinase-1 (ALK-1) leads to heart failure without maladaptive remodeling
13. Persistent infiltration and pro-inflammatory differentiation of monocytes cause unresolved inflammation in brain arteriovenous malformation
14. CXCL12-CXCR4 signalling plays an essential role in proper patterning of aortic arch and pulmonary arteries
15. Bone morphogenetic protein-9 inhibits lymphatic vessel formation via activin receptor-like kinase 1 during development and cancer progression
16. Angiotensin-converting enzyme 2 inhibits high-mobility group box 1 and attenuates cardiac dysfunction post-myocardial ischemia
17. Adenine Nucleotide Translocase 4 Is Expressed Within Embryonic Ovaries and Dispensable During Oogenesis
18. VEGF neutralization can prevent and normalize arteriovenous malformations in an animal model for hereditary hemorrhagic telangiectasia 2
19. Restraint of angiogenesis by zinc finger transcription factor CTCF-dependent chromatin insulation
20. Contributors
21. Mitochondrial ATP transporter depletion protects mice against liver steatosis and insulin resistance
22. TGF-β signaling in endothelial cells, but not neuroepithelial cells, is essential for cerebral vascular development
23. Emerging pathogenic mechanisms in human brain arteriovenous malformations: a contemporary review in the multiomics era.
24. ALK5- and TGFBR2-independent role of ALK1 in the pathogenesis of hereditary hemorrhagic telangiectasia type 2
25. Growth differentiation factor 11 signaling controls retinoic acid activity for axial vertebral development
26. Real-time imaging of de novo arteriovenous malformation in a mouse model of hereditary hemorrhagic telangiectasia
27. Dysregulation of intestinal crypt cell proliferation and villus cell migration in mice lacking Kruppel-like factor 9
28. A murine model for human Sepiapterin-reductase deficiency
29. Change in gene expression subsequent to induction of Pnn/DRS/memA: increase in p21cip1/waf1
30. Suppression of BMP signaling by PHD2 deficiency in Pulmonary Arterial hypertension.
31. Nonoverlapping expression patterns of ALK1 and ALK5 reveal distinct roles of each receptor in vascular development
32. TMEM100, a Lung-Specific Endothelium Gene.
33. A mouse model for hereditary hemorrhagic telangiectasia (HHT) type 2
34. Interaction Between ALK1 Signaling and Connexin40 in the Development of Arteriovenous MalformationsSignificance
35. SMAD4 Deficiency Leads to Development of Arteriovenous Malformations in Neonatal and Adult Mice.
36. Activin type IIA and IIB receptors mediate Gdf11 signaling in axial vertebral patterning
37. CXCL12-CXCR4 signalling plays an essential role in proper patterning of aortic arch and pulmonary arteries.
38. 475 Local Conditional Induction of Brain Arteriovenous Malformations in Alk1-Inducible Knock-Out Mice.
39. Effect of Topical Intranasal Therapy on Epistaxis Frequency in Patients With Hereditary Hemorrhagic Telangiectasia: A Randomized Clinical Trial.
40. Neuropilin 1 balances ß8 integrin-activated TGFß signaling to control sprouting angiogenesis in the brain.
41. Mouse models of hereditary hemorrhagic telangiectasia: recent advances and future challenges.
42. Endothelial Depletion of Acvrl1 in Mice Leads to Arteriovenous Malformations Associated with Reduced Endoglin Expression.
43. SMAD1 Deficiency in Either Endothelial or Smooth Muscle Cells Can Predispose Mice to Pulmonary Hypertension.
44. Enhanced Responses to Angiogenic Cues Underlie the Pathogenesis of Hereditary Hemorrhagic Telangiectasia 2
45. Conditional Deletion of Jak2 Reveals an Essential Role in Hematopoiesis throughout Mouse Ontogeny: Implications for Jak2 Inhibition in Humans.
46. Reduced Mural Cell Coverage and Impaired Vessel Integrity After Angiogenic Stimulation in the Alk1-deficient Brain.
47. Minimal Homozygous Endothelial Deletion of Eng with VEGF Stimulation Is Sufficient to Cause Cerebrovascular Dysplasia in the Adult Mouse.
48. Vascular smooth muscle Jak2 deletion prevents angiotensin II-mediated neointima formation following injury in mice
49. Arteriovenous malformation in the adult mouse brain resembling the human disease.
50. Impaired Terminal Differentiation of Hippocampal Granule Neurons and Defective Contextual Memory in PC3/Tis21 Knockout Mice.
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