198 results on '"Jonas, Jean-Christophe"'
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2. The lack of functional nicotinamide nucleotide transhydrogenase only moderately contributes to the impairment of glucose tolerance and glucose-stimulated insulin secretion in C57BL/6J vs C57BL/6N mice
3. Metallothionein 1 negatively regulates glucose-stimulated insulin secretion and is differentially expressed in conditions of beta cell compensation and failure in mice and humans
4. NNT reverse mode of operation mediates glucose control of mitochondrial NADPH and glutathione redox state in mouse pancreatic β-cells
5. Phlda3 regulates beta cell survival during stress
6. Hypoxia reduces ER-to-Golgi protein trafficking and increases cell death by inhibiting the adaptive unfolded protein response in mouse beta cells
7. Identification of a human-specific alteration of beta cell function after prolonged culture of pancreatic islets under glucotoxic conditions
8. Unveiling a common mechanism of apoptosis in β-cells and neurons in Friedreichʼs ataxia
9. MicroRNAs contribute to compensatory β cell expansion during pregnancy and obesity
10. Endoplasmic reticulum accumulation of Kir6.2 without activation of ER stress response in islet cells from adult Sur1 knockout mice
11. Effects of c-MYC activation on glucose stimulus-secretion coupling events in mouse pancreatic islets
12. Glucose-induced mixed [[[Ca.sup.2+]].sub.c] oscillations in mouse [beta]-cells are controlled by the membrane potential and the SERCA3 [Ca.sup.2+]-ATPase of the endoplasmic reticulum
13. HDLs Protect Pancreatic β-Cells Against ER Stress by Restoring Protein Folding and Trafficking
14. Mechanisms of Control of the Free Ca2+ Concentration in the Endoplasmic Reticulum of Mouse Pancreatic β-Cells: Interplay With Cell Metabolism and [Ca2+]c and Role of SERCA2b and SERCA3
15. Probe-Independent and Direct Quantification of Insulin mRNA and Growth Hormone mRNA in Enriched Cell Preparations
16. Mechanisms of Pancreatic β-Cell Death in Type 1 and Type 2 Diabetes: Many Differences, Few Similarities
17. Induction of Adiponectin in Skeletal Muscle by Inflammatory Cytokines: in Vivo and in Vitro Studies
18. Atypical Ca2+-induced Ca2+ release from a sarco-endoplasmic reticulum Ca2+-ATPase 3-dependent Ca2+ pool in mouse pancreatic β-cells
19. Corticosteroids Induce Expression of Aquaporin-1 and Increase Transcellular Water Transport in Rat Peritoneum
20. SERCA3 Ablation Does Not Impair Insulin Secretion but Suggests Distinct Roles of Different Sarcoendoplasmic Reticulum Ca2+ Pumps for Ca2+ Homeostasis in Pancreatic β-cells
21. Increased Glucose Sensitivity of Stimulus-Secretion Coupling in Islets From Psammomys obesus After Diet Induction of Diabetes
22. Do Oscillations of Insulin Secretion Occur in the Absence of Cytoplasmic Ca2+ Oscillations in β-Cells?
23. Control Mechanisms of the Oscillations of Insulin Secretion In Vitro and In Vivo
24. Signals and Pools Underlying Biphasic Insulin Secretion
25. Increased Expression of Antioxidant and Antiapoptotic Genes in Islets That May Contribute to β-Cell Survival During Chronic Hyperglycemia
26. β-Cell Adaptation to Hyperglycemia
27. Potential Role of the Early Response Gene c-myc in β-Cell Adaptation to Changes in Glucose Concentration
28. Transcriptome analysis of islets from diabetes-resistant and diabetes-prone obese mice reveals novel gene regulatory networks involved in beta-cell compensation and failure.
29. Glucose Regulates Expression of Inositol 1,4,5-Trisphosphate Receptor Isoforms in Isolated Rat Pancreatic Islets*
30. Metallothionein 1 inhibits glucose-stimulated insulin secretion and is differentially regulated in conditions of beta cell compensation and failure
31. Temporal and Quantitative Correlations Between Insulin Secretion and Stably Elevated or Oscillatory Cytoplasmic Ca2+ in Mouse Pancreatic beta-Cells
32. Possible Links between Glucose-induced Changes in the Energy State of Pancreatic B Cells and Insulin Release: Unmasking by Decreasing a Stable Pool of Adenine Nucleotides in Mouse Islets
33. Inhibition of aquaporin-1 prevents myocardial remodeling by blocking the transmembrane transport of hydrogen peroxide.
34. Proof-of-concept for 2D/CT element analysis of entire cryofrozen islets of Langerhans using a cryoloop synchrotron X-ray fluorescence setup.
35. Aspalathin Protects Insulin‐Producing β Cells against Glucotoxicity and Oxidative Stress‐Induced Cell Death.
36. Nutrient Metabolism, Subcellular Redox State, and Oxidative Stress in Pancreatic Islets and β-Cells.
37. Glucose Acutely Reduces Cytosolic and Mitochondrial H2O2 in Rat Pancreatic Beta Cells.
38. Somatostatin Is Only Partly Required for the Glucagonostatic Effect of Glucose but Is Necessary for the Glucagonostatic Effect of KATP Channel Blockers.
39. Pancreatic β-cell tRNA hypomethylation and fragmentation link TRMT10A deficiency with diabetes.
40. Editorial Overview: "Islet Biology in Type 2 Diabetes".
41. Mechanisms of β-cell dedifferentiation in diabetes: recent findings and future research directions.
42. Biomarkers of tumour redox status in response to modulations of glutathione and thioredoxin antioxidant pathways.
43. Mechanisms of control of the free Ca2+ concentration in the endoplasmic reticulum of mouse pancreatic β-cells : interplay with cell metabolism and [Ca2+]c and role of SERCA2b and SERCA3
44. Role of activating transcription factor 3 in low glucose- and thapsigargin-induced apoptosis in cultured mouse islets
45. Effects of deglycating enzyme Fructosamine-3-kinase gene knockout on pancreatic beta cell glucotoxicity
46. High glucose activates hypoxia inducible factors 1 and 2 in cultured rat islets and INS1-E cell monolayers
47. The Ca2+ -ATPase SERCA3 influences membrane potential and the pattern of cytosolic [Ca2+]i oscillations in glucose-stimulated mouse beta-cells
48. Glucokinase activation is beneficial or toxic to cultured rat pancreatic islets depending on the prevailing glucose concentration.
49. Physiological ER Stress: The Model of Insulin-Secreting Pancreatic b-Cells.
50. Acute nutrient regulation of the mitochondrial glutathione redox state in pancreatic ß-cells.
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