45 results on '"Xu D"'
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2. ROLE OF AIF IN TRAUMA HEMORRHAGIC SHOCK-INDUCED ENDOTHELIAL APOPTOSIS
3. THE RELATIVE ROLE OF CASPASE-DEPENDENT AND INDEPENDENT APOPTOSIS OF ENDOTHELIAL AND PULMONARY EPITHELIAL CELLS, INDUCED BY T/HS LYMPH, VARIES BY CELLTYPE
4. MESENTERIC LYMPH FROM THE RATS SUBJECTED TO TRAUMA/HEMORRHAGE IS CYTOTOXIC TO MULTIPLE CELL LINES FROM DIFFERENT TISSUES.
5. MESENTERIC LYMPH DUCT LIGATION IMPROVES SURVIVAL IN SPLANCHNIC ARTERY OCCLUSION SHOCK.
6. T/HS MESENTERIC LYMPH DIRECTLY LEADS TO LUNG INJURY AND IMPAIRED RED BLOOD CELL DEFORMABILITY IN A RAT MODEL.
7. SEVERITY OF HEMORRHAGIC SHOCK-INDUCED RBC DAMAGE IN FEMALES DEPENDS ON THE STAGE OF ESTRUS CYCLE
8. SEX HORMONES MODULATE DISTANT ORGAN INJURY IN A BURN MODEL.
9. Title: PANCREATIC DUCT LIGATION REDUCES LUNG INJURY FOLLOWING TRAUMA/HEMORRHAGIC SHOCK.
10. ALBUMIN PROTECTS AGAINST GUT-INDUCED LUNG INJURY IN VITRO AND IN VIVO.
11. MESENTERIC LYMPH FROM MALE RATS INDUCES ENDOTHELIAL CELL APOPTOSIS IN A NEW IN VIVO MODEL.
12. MESENTERIC LYMPH FROM BURNED RATS WITH EVIDENCE OF GUT INJURY IS CYTOTOXIC FOR ENDOTHELIAL CELLS.
13. INTESTINAL BACTERIAL OVERGROWTH-INDUCED BACTERIAL TRANSCATION RESULTS IN THE GENERATION OF TOXIC MESENTERIC LYMPH THAT INJURES ENDOTHELIAL CELLS
14. DIFFERENT METHODS TO EVALUATE SHOCKINDUCED CHANGES IN RBC DEFORMABILITY: ADVANTAGES AND LIMITATIONS.
15. GENE EXPRESSION IN LUNG TISSUE AFTER TRAUMAHEMORRHAGIC SHOCK IS ALTERED BY MESENTERIC LYMPH DUCT LIGATION.
16. Title: POST-HEMORRHAGIC SHOCK MESENTERIC LYMPH KILLS HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS VIA A CASPASE-3 MEDIATED MECHANISM.
17. MESENTERIC LYMPH TOXICITY FOR ENDOTHELIAL CELLS IS MEDIATED BY MULTIPLE PATHWAYS.
18. INDUCIBLE NITRIC OXIDE SYNTHASE IS ACTIVATED BY EXOGENOUS NITRIC OXIDE GENERATION AND INHIBITED BY PREINDUCTION OF HEAT SHOCK PROTEIN 72 IN RAT ENTEROCYTES
19. ROUTES OF BACTERIAL TRANSLOCATION AND ITS ROLE AFTER HEMORRHAGIC SHOCK.
20. MESENTERIC LYMPH DUCT DIVERSION REDUCES CD11b/CD18 EXPRESSION AND ABROGATES LUNG INJURY AFTER TRAUMA HEMORRHAGIC SHOCK.
21. POST SHOCK MESENTERIC LYPMH CONTAINS AN INCREASED PROPORTION OF CD4/CD8 DOUBLE POSITIVE LYMPHOCYTES.
22. HYPERTONIC SALINE IMPROVES INTESTINAL MUCOSA BARRIER FUNCTION AND LUNG INJURY AFTER HEMORRHAGIC SHOCK.
23. EFFECT OF HEMORRHAGIC SHOCK ON RB DEFORMABILITY
24. DENDRITIC CELLS: ANOTHER POSSIBLE GERM CARRIER IN BACTERIAL TRANSLOCATION AFTER HEMORRHAGIC SHOCK.
25. POST HEMORRHAGIC SHOCK MESENTERIC LYMPH UPREGULATES ADHESION MOLECULE EXPRESION IN HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS (HUVECs).
26. Mesenteric Lymph Duct Ligation Provides Long Term Protection Against Hemorrhagic Shock-induced Lung Injury
27. Na+,K+-ATPASE ACTIVITY IS INHIBITED IN CULTURED INTESTINAL EPITHELIAL CELLS BY ENDOTOXIN AND NITRIC OXIDE
28. A RELATIONSHIP EXISTS BETWEEN DECREASED ILEAL Na+,K+-ATPASE ACTIVITY AND BACTERIAL TRANSLOCATION AFTER ENDOTOXIN CHALLENGE
29. MESENTERIC LYMPH FROM RATS SUBJECTED TO HEMORRHAGIC SHOCK IS CYTOTOXIC TO RAT PULMONARY MICROVASCULAR ENDOTHELIAL CELLS
30. GUT-DERIVED MESENTERIC LYMPH: A LINK BETWEEN BURN AND LUNG INJURY.
31. INHIBITION OF NO BY L-NMMA REDUCES ENDOTOXIN-INDUCED INCREASES IN INTESTINAL PERMEABILITY TO BACTERIAL, iNOS ACTIVITY AND IL-6 PRODUCTION IN USSING CHAMBER MODEL.
32. INDUCIBLE NITRIC OXIDE SYNTHASE GENE KNOCK-OUT MICE WERE RESISTANT TO THE BACTERIAL TRANSLOCATION AFTER INTESTINAL ISCHEMIA-REPERFUSION.
33. THE GUT LIBERATES PROINFLAMMATORY CYTOKINES AFTER INTESTINAL ISCHEMIA
34. Effect of Gut Microflora on Cytokine Levels in Mesenteric Lymph of Rats
35. EFFECT OF ACUTE PHASE AND HEAT SHOCK STRESS ON APOPTOSIS IN INTESTINAL EPITHELIAL CELLS (CACO-2)
36. ENDOTOXIN INCREASES NITRIC OXIDE PRODUCTION WITH MESENTERIC LYMPH NODE, PLASMA AND INTESTINAL MUCUS IN RATS.
37. APOPTOSIS IN EPITHELIAL CELLS IN RESPONSE TO ACUTE PHASE AND HEAT SHOCK STIMULI.
38. THE INTESTINAL CYTOKINE RESPONSE AFTER GUT ISCHEMIA
39. ELEMENTAL DIET-INDUCED BACTERIAL TRANSLOCATION IS ASSOCIATED WITH SYSTEMIC, and INTESTINAL IMMUNE SUPPRESSION
40. Mesenteric Lymph Duct Ligation Provides Long Term Protection Against Hemorrhagic Shockinduced Lung Injury
41. Na+,K+-ATPASE ACTIVITY IS INHIBITED IN CULTURED INTESTINAL EPITHELIAL CELLS BY ENDOTOXIN AND NITRIC OXIDE.
42. A RELATIONSHIP EXISTS BETWEEN DECREASED ILEAL Na+,K+-ATPASE ACTIVITY AND BACTERIAL TRANSLOCATION AFTER ENDOTOXIN CHALLENGE.
43. NaKATPASE ACTIVITY IS INHIBITED IN CULTURED INTESTINAL EPITHELIAL CELLS BY ENDOTOXIN AND NITRIC OXIDE
44. A RELATIONSHIP EXISTS BETWEEN DECREASED ILEAL NaKATPASE ACTIVITY AND BACTERIAL TRANSLOCATION AFTER ENDOTOXIN CHALLENGE
45. ELEMENTAL DIETINDUCED BACTERIAL TRANSLOCATION IS ASSOCIATED WITH SYSTEMIC and INTESTINAL IMMUNE SUPPRESSION
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