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Start Over Author "Andrea Gallinelli" Journal neuroendocrinology
4 results on '"Andrea Gallinelli"'

1. Subject Index Vol. 57, 1993

Author

Flemming W. Bach, Fuad Fares, Rodrigo Kuljis, Benito Regueiro, Kunïkazu Kondo, Moshe Gavish, Paul M. Dubois, Iván Nagy, Harry Brandt, Andreas Kjaer, Alessandro D. Genazzani, Zsuzsanna Ács, Kjell Fuxe, Guido Ficarra, Samuel M. McCann, Emanuel Meller, Michael D. DeBellis, B Bidzinska, Shalom Bar-Ami, Andrea Gallinelli, Valeria Sibilia, Kazuo Otake, Philip W. Gold, Jørgen Warberg, Konstantine T. Kalogeras, Stefano Angioni, Donna L. Tempel, Ulrich Knigge, Antonio Pecile, F. V. Vega, Rodrigo O. Kuljis, Antonio Cintra, Ulrich Renner, Gian Paolo Trentini, Christoph J. Auernhammer, Dipak K. Sarkar, Jamshid Rabii, Juan-Pablo Advis, Koki Fukuhara, Bruce S. McEwen, Luís Lima, Mark A. Demitrack, Nicholas C. Vamvakopoulos, Clara V. Alvarez, Francesca Guidobono, Esperanza Cancio, Karen Bohmaker, Felice Petraglia, Charles D. Conover, M. Criscuolo, Michaela Diamant, V. Solfrini, Takashi Murase, Rabia Bouali-Benazzouz, Maryse Bonnin, Bernd Bunnemann, Jason Diamond, A. G. Reznikov, Jan-Ake Gustafsson, Kent E. Vrana, Zsuzsanna Mergl, Jesús Devesa, Aziz El Amraoui, Víctor M. Arce, Abraham Weizman, Harvey J. Whitfield, Vladimir K. Patchev, Sarah F. Leibowitz, Marie-Claude Audy, Mitchel A. Kling, Günter K. Stalla, Johanna Stalla, Sam Okret, Shanaz M. Tejani-Butt, Jesús A.F. Tresguerres, Lawrence A. Frohman, Zamir Amiri, Stephen J. Walker, Francesca Bortolotti, Charles Conover, Samuel J. Listwak, Yutaka Oiso, Carmela Netti, David de Wied, Jianxin Yang, Balakrishna M. Prasad, Béla Halász, Tracy Puza, George P. Chrousos, Michael Schütte, David Okrongly, Amir Kaviani, Gábor B. Makara, Fernando Domínguez, Juan Zalvide, Menek Goldstein, Gyula Horvath, Joh Y. Lew, Andrea R. Genazzani, O. A. Müller, Makoto Sato, and Carlos Dieguez

Subjects
Cellular and Molecular Neuroscience, medicine.medical_specialty, Endocrinology, Index (economics), Endocrine and Autonomic Systems, Endocrinology, Diabetes and Metabolism, Internal medicine, medicine, Subject (documents), Medical physics, Psychology
Published
1993

2. Contents, Vol. 57, 1993

Author

Kjell Fuxe, Moshe Gavish, Harry Brandt, Andreas Kjaer, Samuel M. McCann, Jørgen Warberg, Philip W. Gold, Konstantine T. Kalogeras, Antonio Pecile, Mark A. Demitrack, F. V. Vega, Stephen J. Walker, Francesca Bortolotti, Harvey J. Whitfield, Ulrich Knigge, Michael Schütte, Antonio Cintra, Emanuel Meller, Menek Goldstein, Rodrigo Kuljis, Abraham Weizman, Andrea Gallinelli, Clara V. Alvarez, Flemming W. Bach, Charles D. Conover, Nicholas C. Vamvakopoulos, Francesca Guidobono, Andrea R. Genazzani, Takashi Murase, Maryse Bonnin, Gábor B. Makara, Jason Diamond, Sarah F. Leibowitz, Rabia Bouali-Benazzouz, Samuel J. Listwak, Joh Y. Lew, Günter K. Stalla, Marie-Claude Audy, Kunïkazu Kondo, Esperanza Cancio, David de Wied, Jianxin Yang, Jan-Ake Gustafsson, Karen Bohmaker, Kent E. Vrana, Juan-Pablo Advis, Makoto Sato, Mitchel A. Kling, Otto-Albrecht Müller, Ulrich Renner, Koki Fukuhara, Johanna Stalla, Fuad Fares, Aziz El Amraoui, George P. Chrousos, Carlos Dieguez, Stefano Angioni, Shanaz M. Tejani-Butt, Balakrishna M. Prasad, Gyula Horváth, Sam Okret, Iván Nagy, Michael D. DeBellis, B Bidzinska, Alexander G. Reznikov, Valeria Sibilia, Benito Regueiro, Vladimir K. Patchev, Dipak K. Sarkar, Felice Petraglia, Bruce S. McEwen, Luís Lima, M. Criscuolo, V. Solfrini, Jesús A.F. Tresguerres, Rodrigo O. Kuljis, Gian Paolo Trentini, David Okrongly, Amir Kaviani, Víctor M. Arce, Shalom Bar-Ami, Lawrence A. Frohman, Charles Conover, Béla Halász, Bernd Bunnemann, Alessandro D. Genazzani, Kazuo Otake, Paul M. Dubois, Zsuzsanna Ács, Guido Ficarra, Zsuzsanna Mergl, Christoph J. Auernhammer, Jamshid Rabii, Yutaka Oiso, Carmela Netti, Zamir Amiri, Tracy Puza, Fernando Domínguez, Juan Zalvide, Donna L. Tempel, Michaela Diamant, and Jesús Devesa

Subjects
Cellular and Molecular Neuroscience, medicine.medical_specialty, Endocrinology, Traditional medicine, Endocrine and Autonomic Systems, business.industry, Endocrinology, Diabetes and Metabolism, Internal medicine, medicine, business
Published
1993

3. Deficit of Galanin-Like Immunostaining in the Median Eminence of Adult Hypothyroid Rats

Author

Andrea Gallinelli, L. Giardino, Laura Calzà, and A. Velardo

Subjects
Male, endocrine system, medicine.medical_specialty, endocrine system diseases, Vasopressins, Endocrinology, Diabetes and Metabolism, Hypothalamus, Fluorescent Antibody Technique, Neuropeptide, Galanin, Biology, Oxytocin, Cellular and Molecular Neuroscience, Endocrinology, Hypothyroidism, Internal medicine, medicine, Animals, Endocrine and Autonomic Systems, digestive, oral, and skin physiology, Median Eminence, Rats, Inbred Strains, Growth hormone secretion, Rats, nervous system, Median eminence, Medulla oblongata, Magnocellular cell, Peptides, hormones, hormone substitutes, and hormone antagonists, Paraventricular Hypothalamic Nucleus, medicine.drug
Abstract

In this paper we describe the modification of the galanin (GAL)-like immunostaining in the hypothalamus of rats, which were made hypothyroid at 52 days after birth. On 21st day after the surgical ablation of the thyroid gland, the staining of the GAL-immunoreactive fibers in the median eminence decreased and on the 84th day disappeared almost totally. The GAL-immunoreactive distribution in other areas of the hypothalamus, e.g. the anterior hypothalamus and the dorsomedial nucleus, is only slightly affected by the absence of thyroid hormones, whereas the GAL-staining of medulla oblongata (vagal complex) is equal in both control and hypothyroid rats. In hypothyroid colchicine-treated rats, we were unable to stain GAL-immunoreactive neurons in the paraventricular nucleus (PVN). Oxytocin- and vasopressin-like material was present in the magnocellular neurons and the staining pattern in hypothyroid rats was the same as that of control animals. Our data show a marked reduction in the expression of the GAL-like immunoreactivity of the PVN and median eminence of adult hypothyroid rats. The possible role of this deficit in the pathogenesis of the GH secretion impairment that is observed in hypothyroid rats is discussed.

Published
1992

4. Effect of different chronic intermittent stressors and acetyl-l-carnitine on hypothalamic beta-endorphin and GnRH and on plasma testosterone levels in male rats

Author

M. Criscuolo, Felice Petraglia, Gian Paolo Trentini, Andrea Gallinelli, Stefano Angioni, Andrea R. Genazzani, Alessandro D. Genazzani, Guido Ficarra, and B Bidzinska

Subjects
Acetyl-l-carnitine, Acute stress, Chronic stress, Ether stress, GnRH, Swimming stress, Testosterone, β-Endorphin, Male, medicine.medical_specialty, medicine.drug_class, Endocrinology, Diabetes and Metabolism, Hypothalamus, Radioimmunoassay, Gonadotropin-releasing hormone, Biology, Ether, Gonadotropin-Releasing Hormone, stress, Cellular and Molecular Neuroscience, chemistry.chemical_compound, Endocrinology, Stress, Physiological, Internal medicine, medicine, Animals, Rats, Wistar, Acetylcarnitine, Swimming, Endocrine and Autonomic Systems, beta-Endorphin, Androgen, Rats, chemistry, Chronic Disease, Hormone, medicine.drug
Abstract

Chronic stress affects the reproductive function by modifying the neuroendocrine homeostasis. The aim of the present study was to clarify the neuroendocrine and the gonadal changes following chronic intermittent stress in male rats and the action of a neuroactive drug, acetyl-l-carnitine (ALC). The effect of two different stressors, cold water swimming or ether, on central beta-endorphin (beta-EP) and GnRH contents, and on plasma testosterone levels was investigated. In addition, the response to an acute stress in chronically stressed rats, treated or untreated with ALC (10 mg/day/rat p.o.), was evaluated. The stressors were applied twice a day for 10 days, and rats were killed before, during and after the last stress session. Mediobasal hypothalamus (MBH) beta-EP and GnRH contents, and plasma testosterone levels were evaluated by radioimmunoassay. The following results were obtained: (1) both chronic swimming and ether stress caused a decrease in hypothalamic beta-EP contents; (2) MBH GnRH contents increased after chronic swimming stress but not after ether stress; (3) chronic swimming stress induced a twofold decrease in plasma testosterone levels, while no changes were observed after ether stress; (4) the treatment with ALC prevented the decrease in plasma testosterone levels after chronic swimming stress, and (5) acute stress in chronically stressed animals caused an increase in MBH-beta-EP. The present data showed that chronic swimming stress reduces the reproductive capacity and impairs the capacity to respond to the acute stress and that ALC modulates the hormonal changes to physical stress and prevents the antireproductive effect of chronic cold swimming.

Published
1993

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