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3. CKD PATHOPHYSIOLOGY AND CLINICAL STUDIES

Author

Paola Achilli, Francesco Marino, Ioannis Karatzas, Steven Fishbane, Alessandro Domenico Quercia, Yu Du, Richard R. Furman, Katarzyna Maresz, Jack F.M. Wetzels, Gerard A. Rongen, Hyun Yul Rhew, Ogo Egbuna, Mariano Rodriguez, Leena Patel, Kiyoto Koibuchi, Anna-Ewa O Kulik, M Weiswasser, Ken Sakai, Antoine Bouquegneau, Myles Wolf, Ilona Kurnatowska, Chantal Loirat, Alberto Ortiz, Stéphane Poitevin, Tilo Hanowski, Elvira Fernández, Abdul Rashid Qureshi, Françoise Dignat-George, Christophe Legendre, Juan Jesus Carrero, Jeffrey Kaupke, Maurizio Postorino, Pablo E. Pergola, Jaco Botha, Bernhard K Kraemer, Mariusz Kusztal, Camille L. Bedrosian, Ada Braun, Marion Sallée, Katarzyna Jankowska, Marcus E. Kleber, Magdalena Kaczmarska, Georg Schlieper, Yeon Soon Jung, Giuseppe Enia, Rosaria Lupica, Beatriz Martínez Fernández, Taro Hoshino, Peter Boor, Mai Ots-Rosenberg, Maria Pina P Madonna, Ayako Tsuchiya, Cesare Guarena, Usama Elewa, Rika Miura, Graciela E. Delgado, Winfried Maerz, Jacek C Szepietowski, Yoshifumi Ubara, Alan G. Jardine, Thiane Gama-Axelsson, Kaoru Tabei, Vincenzo Cantaluppi, Franco Brescia, Carmine Zoccali, Yao Yu, Akihiro Tsuda, Viatcheslav Rakov, Yasemin G Kurt, Sergio Dellepiane, Mahmut Ilker Yilmaz, Winnie Sohn, Bengt Lindholm, Claudia Carmone, Rabab Mohamed Elbehidy, Ye Na Kim, Bertrand Gondouin, Hark Rim, Larry Greenbaum, Dimitrios Arvanitis, Cristina Masini, Michael Chen, Laetitia Dou, Vincenzo Panichi, Leszek Bieniaszewski, Etienne Cavalier, Federica Genovese, Hikmet Tekce, Giovanni Camussi, Mahmut I Yilmaz, Tacyano Tavares Leite, Stéphane Burtey, Yoshiteru Ohno, Atsushi Aikawa, Peter Braunhofer, Johan Vande Walle, Irina Mititiuc, Olivier Devuyst, Amit Sharma, Stefan Degenhardt, Glenn M. Chertow, Henrik S. Rasmussen, Rannveig Skrunes, Dimitra Nastou, E. Marie Freel, Zbigniew Heleniak, Mahmut Gok, Heike Kielstein, Jesús Egido, Steven Zeig, Patrick B. Mark, David Arroyo, Katarzyna Kunicka, Dimosthenis Vlassopoulos, Paweł Poznański, Bruce Spinowitz, Gian Domenico D Fabbri, Jaap Deinum, Yasmine Draz, Marina Foramitti, K. Lysaja, Marian Klinger, Iris Fuhrmann, Cees Vermeer, A. Villari, Piotr Skrzypczyk, Hubert Scharnagl, Emily P. McQuarrie, Giuseppina Pettinato, Kentaro Tanaka, Bożena Werner, Yasuhisa Sakurai, MałGorzata Sojka, Bolesław Rutkowski, Cric Study Investigators, Song Rong, Adrian Covic, Lisa M. Bernard, Carlo Massimetti, Candice Bezerra Torres De Melo, Davide Medica, Jose M. Valdivielso, Martin Flamant, Markus Ketteler, Morten A. Karsdal, Shay Shemesh, Domenico Santoro, Aoi Nabata, Bhupinder Singh, Jean-Marie Krzesinski, Emmanuelle Vidal-Petiot, Tanja B. Grammer, Sandro Feriozzi, Fabio Malberti, Camilla Tøndel, Tayfun Eyileten, Nikolaos Manolios, K K Larsen, Camillo Porta, Junichi Hoshino, Filippo Benedetto, Jesper N. Bech, Larry A. Greenbaum, Rolfdieter Krause, Dimitrie Siriopol, Catherine Delmas-Frenette, Hideaki Shima, Reginaldo Filho, Katarzyna Kilis-Pstrusinska, Stuart M. Sprague, Akifumi Kushiyama, Kiyonori Ito, Katsunori Saito, Ludomir Stefańczyk, Mari Aoe, Juliette Hadchouel, Juergen Floege, Jürgen Floege, Lara Cavalcante Vaz Cunha, Fernanda Macedo de Oliveira Neves, Honami Mori, Mutlu Saglam, Giovanni Tripepi, Yasemin Gulcan Kurt, Mohamed A El-Shahawy, Ewa Aleksandrowicz, Kristin Jäger, Graziella Caridi, Pierre Delanaye, Moriatsu Miyagi, Desmond Padhi, Mai Sugahara, Kiryong Park, Mait Raag, Renata de Almeida Leitão, Thomas D. Wooldridge, Keiji Hirai, Gianluca Trifirò, Elzbieta Prus-Wojtowicz, Naoki Sawa, Murat Karaman, Alexandre Braga Libório, François Vrtovsnik, Ewa Świerblewska, Yuichirou Ueda, Eiji Ishimura, Yusuf Oguz, Masayo Ogawa, Geoffrey A. Block, Frank H Mose, Ho Sik Shin, Yahsou Delmas, Magdalena Okarska-Napierała, Toshiyuki Aoki, Richard Amdur, Hilmi Umut Unal, Stéphan Troyanov, Tammo Lesch, Amal A Alshal, Edward Chong, Ülle Pechter, Alison Taylor, Katsuhito Mori, Mehmet Kanbay, Akinobu Ochi, Claire Cerini, Naobumi Mise, Susumu Ookawara, Joris H. Robben, Hakki Cetinkaya, Anna Masajtis-Zagajewska, Davide Bolignano, Hilmi Umut Ünal, Mitsuru Ichii, Kensuke Hamada, Naoya Sugiyama, Sebahattin Sari, Gianluca Leonardi, Abdulgaffar Vural, Noémie Jourde-Chiche, Sankar D. Navaneethan, N. Dimkovic, Franziska Knöfel, Marios Papasotiriou, Peter Mt Deen, Fadi Fakhouri, Dimitrios Hadjiyannakos, Erling B. Pedersen, Luigi Biancone, Vassilis Filiopoulos, N. Marx, Masayoshi Mori, Zbigniew Zdrojewski, Giovanni F.M. Strippoli, Yoshio Kaku, Masatomo Chikamori, Angels Betriu, Michele Buemi, Kenmei Takaichi, William T. Smith, Izumi Sugimoto, A. Savvaidis, Daijo Inaguma, Giuseppe Costantino, John F Kincaid, Laura Cosmai, Radosław Pietrzak, Roberto Sabbatini, Michał Nowicki, Stephen R. Ash, Kenichi Ishizawa, Masaaki Inaba, Daniela Leonardis, Piotr Grzelak, Jonas Axelsson, Robert A. Fenton, Massimiliano Migliori, Junichiro Yamamoto, Diana J Leeming, Giovanna Parlongo, Philip T. Lavin, Buket Kin Tekce, Dominic S. Raj, James Cotton, David J. Cohen, Shinya Nakatani, Sangeon Gwoo, Shigeko Hara, Frank Schiepe, Philip Awadalla, Gulali Aktas, Massimo Gai, Maria Roszkowska-Blaim, Neil S. Sheerin, Lei Nan, K: Hess, Haruhisa Miyazawa, Silvia Lucisano, Grahame J Elder, François Madore, Helena Ziółkowska, Cai-Li Wang, Einar Svarstad, Giuseppina Lorenzano, Maria Teresa T Muratore, Alex Yang, Graziella D'Arrigo, Doaa Mohammed Youssef, Janni M Jensen, Marta Gracia, Suetonia C. Palmer, Valeria Cernaro, Borja Quiroga, Anton E. Daul, Francesca Mallamaci, Philippe Brunet, Tomoko Honda, Gerjan Navis, Izumi Yoshida, Domenico Trimboli, and Anjay Rastogi

Subjects
Transplantation, medicine.medical_specialty, Nephrology, business.industry, Medicine, business, Intensive care medicine, Pathophysiology
Published
2014

5. Uraemic plasma decreases the expression of ABCA1, ABCG1 and cell-cycle genes in human coronary arterial endothelial cells

Author

Marie-Josée Hébert, François Madore, Marc-André Raymond, and Héloïse Cardinal

Subjects
Male, medicine.medical_specialty, Endothelium, Gene Expression, Apoptosis, Polymerase Chain Reaction, Plasma, chemistry.chemical_compound, Internal medicine, medicine, Humans, Endothelial dysfunction, Cells, Cultured, ATP Binding Cassette Transporter, Subfamily G, Member 1, Aged, Cell Proliferation, Uremia, Transplantation, biology, Cholesterol, business.industry, Cell Cycle, Middle Aged, Cell cycle, Microarray Analysis, medicine.disease, Coronary Vessels, Endothelial stem cell, medicine.anatomical_structure, Endocrinology, chemistry, Nephrology, ABCA1, biology.protein, RNA, ATP-Binding Cassette Transporters, Female, Endothelium, Vascular, business, ATP Binding Cassette Transporter 1
Abstract

BACKGROUND Uraemia is associated with endothelial dysfunction, but the effect of uraemic plasma on the gene expression pattern of human coronary arterial endothelial cells (HCAEC) has never been defined. METHODS HCAECs were exposed for 48 h to a culture medium supplemented with 20% uraemic vs normal plasma. We extracted mRNA and hybridized it onto Affymetrix HG-U133 Plus2 microarrays. We validated our findings for five genes of interest by real-time PCR and performed evaluations of cell proliferation and apoptosis in HCAECs exposed to uraemic vs normal plasma. RESULTS Six genes involved in the regulation of cell-cycle progression (CDK-1, topoisomerase II, PDZ-binding kinase, CDCA1, protein SDP35, E2F transcription factor 8) and two genes of the cholesterol efflux system (ABCA1 and ABCG1) were down-regulated in HCAECs exposed to uraemic plasma (>1.75-fold change vs normal). Real-time PCR confirmed the down-regulation observed in the microarray experiment. Cell proliferation was significantly decreased in HCAECs exposed to uraemic vs normal plasma for 48 h (86 vs 95% of serum-starved control, P = 0.006). Exposure to uraemic plasma for 48 h was associated with increased apoptosis of HCAEC as compared with normal plasma (7.7 vs 2.8%, P < 0.001), a phenomenon that was further enhanced when oxidized LDLs (150 microg protein/ml) were added to the medium containing uraemic plasma (16.9 vs 7.7%, P < 0.001). CONCLUSIONS The down-regulation of genes involved in cell-cycle progression and cholesterol efflux from HCAECs exposed to uraemic conditions could contribute to enhancing endothelial dysfunction and atherosclerosis in patients with chronic renal failure.

Published
2006

6. SP481DALTEPARIN SODIUM FOR THE PREVENTION OF CLOTTING IN THE EXTRACORPOREAL CIRCUIT DURING HEMODIALYSIS: A PHASE IIIB OPEN-LABEL STUDY TO OPTIMIZE A SINGLE BOLUS DOSE—THE PARROT STUDY

Author

François Madore, Fiona Hilton, Brendan J. Barrett, Norman Muirhead, Jo-Anne Wilson, Martin MacKinnon, Nancy Sherman, Sandra Donnelly, Serge H. Cournoyer, Louise Roy, Steven D. Soroka, Guillaume Feugère, Kevin Wolter, Mohsen Agharazii, John Orazem, and Neesh Pannu

Subjects
Transplantation, medicine.medical_specialty, business.industry, medicine.medical_treatment, Sodium, chemistry.chemical_element, Extracorporeal, Surgery, Single bolus, Open label study, chemistry, Nephrology, Anesthesia, medicine, Hemodialysis, business
Published
2017

7. COPEPTIN IN CKD

Author

Fred Paccaud, Pierre-Yves Martin, Murielle Bochud, Idris Guessous, Mila Tang, Daniel Ackermann, Andrzej Wiecek, Claudio Rigatto, Agata Kujawa-Szewieczek, Michel Marre, Aureliusz Kolonko, Olivier Devuyst, Francesca Martino, Jerzy Chudek, Beata Czerwieńska, Ronan Roussel, Ognjenka Djurdjev, Lise Bankir, Valentina Corradi, Philippe Vuistiner, Elisa Scalzotto, François Alhenc-Gelas, Nadine Bouby, Fiorella Gastaldon, Adeera Levin, Brendan J. Barrett, Belen Ponte, Daniel T. Holmes, Claudio Ronco, Markus G. Mohaupt, Menno Pruijm, Beverley Balkau, Federico Nalesso, Georg Ehret, Antoinette Pechère-Bertschi, Antonio Fortunato, Michel Burnier, Gilberto Velho, Norman Muirhead, Catherine M. Clase, François Madore, and Davide Giavarina

Subjects
Transplantation, medicine.medical_specialty, Copeptin, Nephrology, business.industry, Internal medicine, medicine, Cardiology, business
Published
2014

8. The effect of sodium citrate 4% locking solution for central venous dialysis catheter on the international normalized ratio (INR) value

Author

Stéphan Troyanov, François Madore, Bruno De Bortoli, and Jean-Philippe Rioux

Subjects
Catheterization, Central Venous, medicine.medical_specialty, Pilot Projects, Sodium Citrate, chemistry.chemical_compound, Text mining, Renal Dialysis, Sodium citrate, Humans, Medicine, False Positive Reactions, Citrates, International Normalized Ratio, Transplantation, Heparin, business.industry, Anticoagulants, Reproducibility of Results, Dialysis catheter, Surgery, chemistry, Nephrology, Anesthesia, Warfarin, business, Value (mathematics)
Published
2008

9. Uraemic plasma decreases the expression of ABCA1, ABCG1 and cell-cycle genes in human coronary arterial endothelial cells.

Author

Héloise Cardinal, Marc-André Raymond, Marie-Josée Hébert, and François Madore

Subjects
UREMIA, KIDNEY diseases, ENDOTHELIUM diseases, BLOOD plasma
Abstract

Background. Uraemia is associated with endothelial dysfunction, but the effect of uraemic plasma on the gene expression pattern of human coronary arterial endothelial cells (HCAEC) has never been defined.Methods. HCAECs were exposed for 48 h to a culture medium supplemented with 20% uraemic vs normal plasma. We extracted mRNA and hybridized it onto Affymetrix HG-U133 Plus2 microarrays. We validated our findings for five genes of interest by real-time PCR and performed evaluations of cell proliferation and apoptosis in HCAECs exposed to uraemic vs normal plasma.Results. Six genes involved in the regulation of cell-cycle progression (CDK-1, topoisomerase II, PDZ-binding kinase, CDCA1, protein SDP35, E2F transcription factor 8) and two genes of the cholesterol efflux system (ABCA1 and ABCG1) were down-regulated in HCAECs exposed to uraemic plasma (>1.75-fold change vs normal). Real-time PCR confirmed the down-regulation observed in the microarray experiment. Cell proliferation was significantly decreased in HCAECs exposed to uraemic vs normal plasma for 48 h (86 vs 95% of serum-starved control, P = 0.006). Exposure to uraemic plasma for 48 h was associated with increased apoptosis of HCAEC as compared with normal plasma (7.7 vs 2.8%, P < 0.001), a phenomenon that was further enhanced when oxidized LDLs (150 µg protein/ml) were added to the medium containing uraemic plasma (16.9 vs 7.7%, P < 0.001).Conclusions. The down-regulation of genes involved in cell-cycle progression and cholesterol efflux from HCAECs exposed to uraemic conditions could contribute to enhancing endothelial dysfunction and atherosclerosis in patients with chronic renal failure. [ABSTRACT FROM AUTHOR]

Published
2007

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