17 results on '"Hirose, Hiroshi"'
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2. High-molecular-weight adiponectin is a predictor of progression to metabolic syndrome: a population-based 6-year follow-up study in Japanese men
3. Significance of leptin and high–molecular weight adiponectin in the general population of Japanese male adolescents
4. High molecular weight multimer form of adiponectin as a useful marker to evaluate insulin resistance and metabolic syndrome in Japanese men
5. Cellular insulin resistance in Epstein-Barr virus-transformed lymphoblasts from young insulin-resistant Japanese men
6. GPR40 gene Arg211His polymorphism may contribute to the variation of insulin secretory capacity in Japanese men
7. Relationships between serum soluble leptin receptor level and serum leptin and adiponectin levels, insulin resistance index, lipid profile, and leptin receptor gene polymorphisms in the Japanese population
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9. PPAR[gamma ]2 gene Pro12Ala polymorphism may influence serum level of an adipocyte-derived protein, adiponectin, in the Japanese population
10. Effects of pioglitazone on metabolic parameters, body fat distribution, and serum adiponectin levels in Japanese male patients with type 2 diabetes
11. Effects of intestinal fatty acid-binding protein gene Ala54Thr polymorphism and [beta ]3-adrenergic receptor gene Trp64Arg polymorphism on insulin resistance and fasting plasma glucose in young to older Japanese men
12. Tumor necrosis factor alpha gene G-308A polymorphism, insulin resistance, and fasting plasma glucose in young, older, and diabetic Japanese men
13. Effects of troglitazone on fat distribution in the treatment of male type 2 diabetes
14. Effects of α2-adrenergic agonism, imidazolines, and G-protein on insulin secretion in β cells
15. Exogenous insulin dose-dependently suppresses glucopenia-induced glucagon secretion from perfused rat pancreas
16. Effects of α2- and β-adrenergic agonism on glucagon secretion from perfused pancreata of normal and streptozocin-induced diabetic rats
17. Effects of α 2- and β-adrenergic agonism on glucagon secretion from perfused pancreata of normal and streptozocin-induced diabetic rats
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