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Your search keyword '"Facco, M."' showing total 7 results
Start Over Author "Facco, M." Journal leukemia research
7 results on '"Facco, M."'

1. Topic: AS04-MDS Biology and Pathogenesis/AS04h-Immune deregulation/inflammation: STAT3 MUTATIONS IMPACT ON MDS ASSOCIATION IN PATIENTS WITH LGL LEUKEMIA (LGLL)

Author

Calabretto, G., primary, Trimarco, V., additional, Teramo, A., additional, Barilà, G., additional, Gasparini, V.R., additional, Vicenzetto, C., additional, Rampazzo, E., additional, Pizzi, M., additional, Facco, M., additional, Trentin, L., additional, Semenzato, G., additional, and Zambello, R., additional

Published
2023
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4. O19 - Topic: AS04-MDS Biology and Pathogenesis/AS04h-Immune deregulation: A MULTI-CENTER EXPERIENCE OF HYPOCELLULAR MYELODYSPLASTIC SYNDROMES (H-MDS): FROM CLINICAL DESCRIPTION TO IMMUNOLOGICAL CHARACTERIZATION

Author

Calabretto, G., Attardi, E., Teramo, A., Trimarco, V., Barilà, G., Vicenzetto, C., Gasparini, V., Massarotti, L., Niscola, P., Giai, V., Poloni, A., Finelli, C., Mossuto, S., Facco, M., Vianello, F., Semenzato, G., Santini, V., and Zambello, R.

Published
2021
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5. Topic: AS01-Diagnosis/AS01b-Flow cytometry

Author

Vicenzetto, C., primary, Calabretto, G., additional, Barilà, G., additional, Gasparini, V., additional, Teramo, A., additional, Trimarco, V., additional, Carraro, S., additional, Tonini, A., additional, Semenzato, G., additional, Zambello, R., additional, and Facco, M., additional

Published
2021
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6. Topic: AS04-MDS Biology and Pathogenesis/AS04h-Immune deregulation

Author

Calabretto, G., primary, Attardi, E., additional, Teramo, A., additional, Trimarco, V., additional, Barilà, G., additional, Vicenzetto, C., additional, Gasparini, V., additional, Massarotti, L., additional, Niscola, P., additional, Giai, V., additional, Poloni, A., additional, Finelli, C., additional, Mossuto, S., additional, Facco, M., additional, Vianello, F., additional, Semenzato, G., additional, Santini, V., additional, and Zambello, R., additional

Published
2021
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7. T-cell type lymphoproliferative disease of granular lymphocytes (LDGL) is equipped with a phenotypic pattern typical of effector cytotoxic cells.

Author

Baesso I, Pavan L, Boscaro E, Miorin M, Facco M, Trentin L, Agostini C, Zambello R, and Semenzato G

Subjects
Aged, CD3 Complex immunology, Cell Differentiation immunology, Female, Humans, Immunophenotyping, Male, NK Cell Lectin-Like Receptor Subfamily K, Receptors, IgG immunology, Receptors, Immunologic biosynthesis, Receptors, Immunologic immunology, Receptors, KIR, Receptors, Natural Killer Cell, Lymphocytes immunology, Lymphoproliferative Disorders immunology, T-Lymphocytes, Cytotoxic immunology
Abstract

By analyzing the expression of several cytotoxic markers, killer-immunoglobulin-like receptors (KIRs), CD94/CD159, CD314 and natural cytotoxicity receptors (NCRs), in 22 CD3+ lymphoproliferative disease of granular lymphocyte (LDGL) patients we investigated whether granular lymphocytes (GLs) displayed the phenotype of fully differentiated cytotoxic cells. Our results demonstrate that GLs express a pattern consistent with fully differentiated CTLs. KIRs are expressed only in a fraction of patients (7/22), as is CD94/CD159 (5/22). In conclusion, GLs in CD3+ LDGL patients typically show the phenotype of fully differentiated CTL, whereas the expression of NK receptors does not represent a common feature of the proliferating clone.

Published
2007
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