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In the two cases reported, pulmonary oedema developed immediately following revascularisation of the lower limbs after prolonged ischaemia. Without it being possible to formally exclude the possible role of a haemodynamic factor, the authors suggest the hypothesis of a predominantly lesional mechanism. The "reconnection to the circuit" is the provoking factor in this oedema, the alveolo-capillary membrane being previously altered by the prolonged ischaemia, infection and massive transfusion. The pulmonary manifestations would seem to enter within the classification of somatolysis.