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Your search keyword '"Veres, Gábor"' showing total 13 results
Start Over Author "Veres, Gábor" Journal journal of pediatric gastroenterology & nutrition
13 results on '"Veres, Gábor"'

1. Use of Biosimilars in Pediatric Inflammatory Bowel Disease: An Updated Position Statement of the Pediatric IBD Porto Group of ESPGHAN

Author

de Ridder, Lissy, primary, Assa, Amit, additional, Bronsky, Jiri, additional, Romano, Claudio, additional, Russell, Richard K., additional, Afzal, Nadeem A., additional, Hauer, Almuthe C., additional, Knafelz, Daniela, additional, Lionetti, Paolo, additional, Strisciuglio, Caterina, additional, Veres, Gábor, additional, Winter, Harland, additional, Wolters, Victorien M., additional, Sladek, Malgorzata, additional, Vulto, Arnold G., additional, and Dias, Jorge A., additional

Published
2019
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3. Use of Biosimilars in Pediatric Inflammatory Bowel Disease

Author

Ridder, Lissy, Assa, Amit, Bronsky, Jiri, Romano, Claudio, Russell, Richard K., Afzal, Nadeem A., Hauer, Almuthe C., Knafelz, Daniela, Lionetti, Paolo, Strisciuglio, Caterina, Veres, Gábor, Winter, Harland, Wolters, Victorien M., Sladek, Malgorzata, Vulto, Arnold G., and Dias, Jorge A.

Abstract

Biologic therapies have changed the outcome of both adult and pediatric patients with Inflammatory Bowel Disease (IBD). In September 2013, the first biosimilar of infliximab was introduced into the pharmaceutical market. In 2015, a first position paper on the use of biosimilars in pediatric IBD was published by the ESPGHAN IBD Porto group. Since then, more data have accumulated for both adults and children demonstrating biosimilars are an effective and safe alternative to the originator. In this updated position statement, we summarize current evidence and provide joint consensus statements regarding the recommended practice of biosimilar use in children with IBD.

Published
2019
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4. Risk of Infection and Prevention in Pediatric Patients With IBD

Author

Veereman-Wauters, Gigi, primary, de Ridder, Lissy, additional, Veres, Gábor, additional, Kolacek, Sanja, additional, Fell, John, additional, Malmborg, Petter, additional, Koletzko, Sibylle, additional, Dias, Jorge Amil, additional, Misak, Zrinjka, additional, Rahier, Jean-François, additional, and Escher, Johanna C., additional

Published
2012
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5. Increased Heat Shock Protein 72 Expression in Celiac Disease

Author

Sziksz, Erna, primary, Veres, Gábor, additional, Vannay, Ádám, additional, Prókai, Ágnes, additional, Gál, Krisztina, additional, Ónody, Anna, additional, Korponay-Szabó, Ilma Rita, additional, Reusz, György, additional, Szabó, András, additional, Tulassay, Tivadar, additional, Arató, András, additional, and Szebeni, Beáta, additional

Published
2010
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8. Genetic polymorphisms of CD14, toll-like receptor 4, and caspase-recruitment domain 15 are not associated with necrotizing enterocolitis in very low birth weight infants.

Author

Szebeni, Beáta, Szekeres, Roxána, Rusai, Krisztina, Vannay, Adám, Veres, Gábor, Treszl, András, Arató, András, Tulassay, Tivadar, and Vásárhelyi, Barna

Abstract

Objectives: Inadequate response of the innate immune system to bacterial antigens present in the intestinal flora may play a role in the development of necrotizing enterocolitis (NEC). Pattern recognition receptors such as CD14, toll-like receptor (TLR) 4, and caspase-recruitment domain (CARD) 15 bind bacterial lipopolysaccharide and peptidoglycan, and their activation leads to production of inflammatory cytokines. Our aim was to evaluate whether single nucleotide polymorphisms (SNPs) of CD14, TLR4, and CARD15 are associated with the risk of NEC in very low birth weight (VLBW) infants.Patients and Methods: We determined the CD14 C-260T, TLR4 A +896G, C +1196T, and CARD15 G +2722C, C +2104T, 3020insC functional SNPs in dried blood samples from 118 VLBW infants (of those, 41 developed NEC) and from 146 healthy term newborns using polymerase chain reaction and restriction fragment length polymorphism methods. We tested the association between genotype and risk of NEC.Results: No significant differences were found in the prevalence of CD14 -260T, TLR4 +896G, +1196T, and CARD15 +2722C, +2104T, 3020insC alleles between VLBW infants and healthy term newborns (P = NS). The frequencies of investigated genotypes were similar in infants with and without NEC (P = NS). Furthermore, we did not find any association between genotype and prematurity or sepsis, which are important risk factors of NEC.Conclusions: Carrier state of the tested CD14, TLR4, and CARD15 SNPs is not associated with NEC risk in VLBW infants. [ABSTRACT FROM AUTHOR]

Published
2006
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