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1. INVOLVEMENT OF PAR-4 AND OXIDATIVE STRESS IN TROPHIC FACTOR WITHDRAWAL-INDUCED APOPTOSIS OF HIPPOCAMPAL NEURONS: PROTECTION BY ESTROGEN AND ANTI-OXIDANTS.

Author

Chan, Sic L. and Mattson, Mark P.

Subjects
*NEUROCHEMISTRY, *OXIDATIVE stress, *NEURONS, *APOPTOSIS, *CONFERENCES & conventions, *ASSOCIATIONS, institutions, etc.
Abstract

The article presents an abstract of the paper "Involvement of Par-4 and Oxidative Stress in Trophic Factor Withdrawal-induced Apoptosis of Hippocampal Neurons: Protection by Estrogen and Anti-oxidants," which will be presented at the 30th annual meeting of the American Society for Neurochemistry to be held from March 14-17, 1999 in New Orleans, Louisiana. The paper discusses the role of Par-4 and oxidative stress in neuronal apoptosis resulting from trophic factor withdrawal.

Published
1999

2. SECRETORY PHOSPHOLIPASE A2 (SPLA2) MODULATES NEURONAL EXCITATION AND IS RELEASED FROM NEURONAL CULTURES.

Author

DeCoster, Mark A. and Bazan, Nicolas G.

Subjects
*PHOSPHOLIPASE A2, *METHYL aspartate, *PHOSPHOLIPASES, *NEURONS
Abstract

The article presents an abstract of the paper "Secretory Phospholipase A2 (SPLA2) Modulates Neuronal Excitation and Is Released From Neuronal Cultures," which will be presented at the 30th Annual Meeting of the American Society for Neurochemistry to be held in New Orleans, Louisiana from March 14-17, 1999. It indicates that the enhancement of glutamate-induced intracellular calcium concentration may be due to modulation at the NMDA receptor.

Published
1999

3. POTENTIATION OF GLUTAMATE-INDUCED INCREASES IN [Ca2+]i AND NEURON CELL DEATH BY THE HIV-1 PROTEIN TAT.

Author

Haughey, N. J., Nath, A., and Geiger, J. D.

Subjects
*MEDICAL research, *MEETINGS, *HIV infections, *NEURONS, *AIDS dementia complex
Abstract

The article presents an abstract of the medical research paper "Potentiation of Glutamate-Induced Increases in [Ca 2+], And Neuron Cell Death by the HIV-1 Protein Tat," which will be presented in the 30th meeting of the American Society for Neurochemistry, to be held in New Orleans, Louisiana. The research examines HIV-1 infection which leads to neuronal cell death and a debilitating syndrome known as AIDS-related dementia complex.

Published
1999

4. Early embryonic chick cortical neurons are susceptible to apoptosis induced by tyrosine phosphorylation and free radicals.

Author

Dawson, S. A., Kilkus, J. P., and Dawson, G.

Subjects
*NEURONS, *NEUROCHEMISTRY, *MEETINGS, *EMBRYOLOGY, *ASTROCYTES, *CELL death
Abstract

The article presents an abstract of the research paper "Early embryonic chick cortical neurons are susceptible to apoptosis induced by tyrosine phosphorylation and free radicals," which will be presented at the 30th American Society for Neurochemistry meeting to be held from March 14 to 17, 1999 in New Orleans, Louisiana. The study indicated the possibility of developing drugs to protect embryonic and mature neurons and glia from apoptotic cell death.

Published
1999

5. RAT NEURAL CREST CELLS EXPRESS FUNCTIONAL GAP JUNCTIONS IN VIVO-EVIDENCE THAT NEURAL CREST CELL SURVIVAL REQUIRES GAP JUNCTION FUNCTION.

Author

Bannerman, P., Nichols, W., Oliver, T., Berman, M., and Pleasure, D.

Subjects
*NEURONS, *NEUROCHEMISTRY, *MEETINGS, *GAP junctions (Cell biology), *SPINAL cord
Abstract

The article presents an abstract of the research paper "Rat Neural Crest Cells Express Functional Gap Junctions in Vivo-Evidence That Neural Crest Cell Survival Requires Gap Junction Function," which will be presented at the 30th American Society for Neurochemistry meeting to be held from March 14 to 17, 1999 in New Orleans, Louisiana. The results of the study suggest that gap junctions play an important role in the survival of spinal neural crest cells in vivo.

Published
1999