18 results on '"Qin, C."'
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2. The Pro-Resolving Mediator, Annexin-A1, Regulates Blood Pressure and Signs of Stage B Heart Failure in Mice in a Sex-Specific Fashion
3. Formylpeptide Receptor Agonism as a Novel Approach for Limiting Abdominal Aortic Aneurysm: Impact on Smooth Muscle Cell Phenotypic Switching
4. Targeting Formyl Peptide Receptors as a Novel Approach to Treat Hypertension-Induced Cardiac Adverse Remodelling and Dysfunction
5. Nitroxyl Exerts Positive Inotropic and Vasodilator Effects in the Type 2 Diabetic Heart
6. The Relative Contribution of High Fat Diet and Hyperglycaemia and Their Combination to the Structural Phenotype of the Diabetic Heart
7. Defining the Characteristics of a More Clinically Relevant Mouse Model of Type-2 Diabetes (T2D)-Induced Cardiomyopathy
8. Progression of Markers Contributing to Cardiomyopathy in a Mouse Model of Type 1 Diabetes
9. Novel Adenosine A2B Receptor Signalling in Cardiac Fibroblasts: Uncovering Context-Specific Biased Agonism
10. Annexin-A1−/− Mice with Type 2 Diabetes Exhibit Cardiovascular Dysfunction and Exaggerated Inflammation Despite Less Pronounced Hyperglycaemia
11. Annexin-A1 Deficient Mice Exhibit Exaggerated Cardiovascular Dysfunction in Type 1 Diabetes
12. Cardiac-Specific Insulin-like Growth Factor-1 Receptor (IGF-1R) Expression Attenuates Upregulation of Left Ventricular Hexosamine Biosynthesis (HBP) Flux and Oxidative Stress in Diabetic Mouse Myocardium
13. Formyl Peptide Receptor (FPR) Biased Agonists as Novel Cardioprotection from Myocardial Ischaemia-reperfusion (I-R) Injury
14. Phosphoinositide 3 kinase p110a (rAAV6-caPI3K) gene therapy attenuates diabetic cardiomyopathy in mice
15. Annexin-a1 deficiency exaggerates cardiac injury following myocardial infarction (MI) in vivo
16. Targeting Ischaemia-reperfusion (I-R) Injury in the Intact Heart with Annexin-A1 (ANX-A1) Mimetics
17. Role of FPR1 Formyl Peptide Receptors in the Cardioprotective Actions of Annexin-A1 Against Ischaemia–Reperfusion (I–R) Injury
18. Nitroxyl (HNO), The Novel Redox Sibling of NO, Enhances Left Ventricular (LV) Function and Coronary Flow via Thiol-Sensitive and cGMP-Dependent Actions: Impact of Diabetes
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