33 results on '"Fournié A"'
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2. Antinociceptive and anti-allodynic effects of oral PL37, a complete inhibitor of enkephalin-catabolizing enzymes, in a rat model of peripheral neuropathic pain induced by vincristine
3. Inhibition of osteosarcoma-induced thermal hyperalgesia in mice by the orally active dual enkephalinase inhibitor PL37. Potentiation by gabapentin
4. Depressant effect on a C-fibre reflex in the rat, of RB101, a dual inhibitor of enkephalin-degrading enzymes
5. RB101(S), a dual inhibitor of enkephalinases does not induce antinociceptive tolerance, or cross-tolerance with morphine: a c-Fos study at the spinal level
6. Analgesic doses of the enkephalin degrading enzyme inhibitor RB 120 do not have discriminative stimulus properties
7. Analgesic responses elecited by endogenous enkephalins (protected by mixed peptidase inhibitors) in a variety of morphine-sensitive noxious tests
8. Antinociception produced by the peptidase inhibitor, RB 101, in rats with adrenal medullary transplant into the spinal cord
9. Characterization of neutral endopeptidase in vascular cells, modulation of vasoactive peptide levels
10. Antidepressant-like effects of CCKB receptor antagonists: involvement of the opioid system
11. Similar involvement of several brain areas in the antinociception of endogenous and exogenous opioids
12. Effects of angiotensin-converting enzyme and neutral endopeptidase inhibitors: influence of bradykinin
13. Weak tolerance to the antinociceptive effect induced by the association of a peptidase inhibitor and a CCKB receptor antagonist
14. A selective CCKB receptor antagonist potentiates μ-, but not δ-opioid receptor-mediated antinociception in the formalin test
15. Effects of opioids and non-opioids on c-Fos-like immunoreactivity induced in rat lumbar spinal cord neurons by noxious heat stimulation
16. Assessment of physical dependence after continuous perfusion into the rat jugular vein of the mixed inhibitor of enkephalin-degrading enzymes, RB 101
17. Antinociceptive effect of systemic PC 12, a prodrug mixed inhibitor of enkephalin-degrading enzymes, in normal and arthritic rats
18. Repeated systemic administration of the mixed inhibitor of enkephalin-degrading enzymes, RB101, does not induce either antinociceptive tolerance or cross-tolerance with morphine
19. Lack of physical dependence in mice after repeated systemic administration of the mixed inhibitor prodrug of enkephalin-degrading enzymes, RB101
20. Antidepressant-type effects of endogenous enkephalins protected by systemic RB 101 are mediated by opioid δ and dopamine D1 receptor stimulation
21. Acebutolol, a beta-blocking agent, down modulates the spontaneous polyclonal activation of lymphocytes of NZB x NZW mice
22. Comparison of selective and complete inhibitors of enkephalin-degrading enzymes on morphine withdrawal syndrome
23. Unlike morphine the endogenous enkephalins protected by RB101 are unable to establish a conditioned place preference in mice
24. Further evidence for a role of δ-opiate receptors in the presynaptic regulation of newly synthesized dopamine release
25. Antidepressant-type effects of endogenous enkephalins protected by systemic RB 101 are mediated by opioid δ and dopamine D 1 receptor stimulation
26. In vitro and in vivo effects of kelatorphan on enkephalin metabolism in rodent brain
27. Antidepressant-like effects of CCK B receptor antagonists: involvement of the opioid system
28. A selective CCK B receptor antagonist potentiates μ-, but not δ-opioid receptor-mediated antinociception in the formalin test
29. The μ rather than the δ subtype of opioid receptors appears to be involved in enkephalin-induced analgesia
30. Weak tolerance to the antinociceptive effect induced by the association of a peptidase inhibitor and a CCK B receptor antagonist
31. Analgesic effects of kelatorphan, a new highly potent inhibitor of multiple enkephalin degrading enzymes
32. Demonstration of the crucial role of the phenylalanine moiety in enkephalin analogues for differential recognition of the μ- and δ-receptors
33. Extensive biochemical and pharmacological studies of the first systemically-active, mixed inhibitors of enkephalin-degrading enzymes
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