21 results on '"Cone, Roger"'
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2. Characterization of the Hyperphagic Response to Dietary Fat in the MC4R Knockout Mouse
3. Neuropeptide W: An Anorectic Peptide Regulated by Leptin and Metabolic State
4. Obesity-Induced Inflammation in White Adipose Tissue Is Attenuated by Loss of Melanocortin-3 Receptor Signaling
5. Role of the Central Melanocortin Circuitry in Adaptive Thermogenesis of Brown Adipose Tissue
6. Characterization of Leptin-Responsive Neurons in the Caudal Brainstem
7. Fasting Induces a Large, Leptin-Dependent Increase in the Intrinsic Action Potential Frequency of Orexigenic Arcuate Nucleus Neuropeptide Y/Agouti-Related Protein Neurons
8. Effect of Agouti-Related Protein in Regulation of the Hypothalamic-Pituitary-Thyroid Axis in the Melanocortin 4 Receptor Knockout Mouse
9. Peptide YY3-36 Inhibits Food Intake in Mice through a Melanocortin-4 Receptor-Independent Mechanism
10. The Anorexigenic Fatty Acid Synthase Inhibitor, C75, Is a Nonspecific Neuronal Activator
11. A Role for the Endogenous Opioid [beta]-Endorphin in Energy Homeostasis
12. Differential Role of Melanocortin Receptor Subtypes in Cachexia
13. The Central Melanocortin System Can Directly Regulate Serum Insulin Levels*
14. Editorial: The Corticotropin-Releasing Hormone System and Feeding Behavior-A Complex Web Begins to Unravel
15. Editorial: The Ups and Downs of Leptin Action
16. Altered Expression of Agouti-Related Protein and Its Colocalization with Neuropeptide Y in the Arcuate Nucleus of the Hypothalamus during Lactation*
17. Characterization of the Neuroanatomical Distribution of Agouti-Related Protein Immunoreactivity in the Rhesus Monkey and the Rat*
18. Peptide YY3–36Inhibits Food Intake in Mice through a Melanocortin-4 Receptor-Independent Mechanism
19. A Role for the Endogenous Opioid β-Endorphin in Energy Homeostasis
20. A Unique Metalolic Sysdrone Causes Obesity in the Melanocortin-3 Receptor-Deficient Mouse
21. The Interaction of Signal Transduction Pathways in FRTL5 Thyroid Follicular Cells: Studies with Stable Expression of β2-Adrenergic Receptors*
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