39 results on '"Tommaso Mello"'
Search Results
2. A mitochondrial role of RuvBL1 ATPase in the regulation of HCC cell metabolism
3. T02.02.6 NON-SMALL-BOWEL LESIONS IDENTIFICATION BY CAPSULE ENDOSCOPY: A SINGLE CENTRE RETROSPECTIVE STUDY
4. P.04.4 NUCLEAR ORPHAN RECEPTOR COUP-TF2 INCREASED THE RESISTANCE TO ANIKOISIS AND THE METASTATIC POTENTIAL IN HEPATOCELLULAR CARCINOMA
5. Nuclear orphan receptor COUP-TF2 induces anoikis resistance, amoeboid migration and metastatic potential in hepatocellular carcinoma (HCC)
6. A new regulator of mitochondrial OXPHOS in hepatocytes and HCC cell lines: the AAA+ ATPase RuvBL1
7. Ruvbl1 regulates liver glucose metabolism via the Akt/mTOR pathway: Implications for hepatocellular carcinoma progression
8. Chronic particulate matter exposition and western diet induce the progression from steatosis to steatohepatitis
9. P.01.4 ADAM-9 EXPRESSION MEDIATES COUP-TFII-DEPENDENT INVASIVENESS OF PANCREATIC CANCER CELLS
10. SIRT1 activity in hepatic stellate cells during liver injury is a target for the modulation of the fibrogenic process
11. The AAA+ ATPase Ruvbl1 coordinates liver metabolism and hepatocellular carcinoma progression
12. Pharmacological inhibition of Sirt1 promotes apoptosis and senescence of hepatic stellate cells during liver injury
13. The two Thieno-triazolodiazepines WEB 2086 and WEB 2170 block Hepatocarcinoma progression by Hsp-dependent client kinoma protein ubiquitination
14. PC.02.2 TARGET THERAPY: A TELOMERASE ACTIVATED THYMIDINE ANALOGUE PRO-DRUG FOR THE TREATMENT OF HEPATOCELLULAR CARCINOMA
15. P.10.21 COUP-TFII DOWNREGULATION INHIBITS PANCREATIC CANCER GROWTH
16. OC.05.3 ROSIGLITAZONE REDUCES THE IN VITRO AND IN VIVO ONCOGENIC POTENTIAL OF HCC CELLS THROUGH A PPARGAMMA-INDEPENDENT NON-GENOMIC INHIBITION OF RUVBL-1
17. P.03.2 KEY REGULATION OF SIRT1 IN THE PRO-SURVIVAL AND ACTIVATION MECHANISMS OF HEPATIC STELLATE CELLS IN RESPONSE TO OXIDATIVE STRESS RELATED DAMAGE
18. P.01.2 PAF-RECEPTOR ANTAGONISTS INHIBIT HEPATOCELLULAR CARCINOMA GROWTH
19. PAF-receptor antagonists reduce HCC progression blocking cancer cell proliferation and migration
20. Antagonistic signalling by PPARgamma and rosiglitazone converge on Ruvbl1 regulation in HCC cells
21. ADAM-9 PROMOTES CELL PROLIFERATION AND EXTRACELLULAR-MATRIX INVASION IN HUMAN PANCREATIC CANCER CELL LINES
22. T-32 In vivo and in vitro models support the therapeutic effects of acycloguanosyl 5’-thymidyltriphosphate for the hepatocellular carcinoma via telomerase activation
23. T-33 Knockdown of Ruvbl-1 reduces HCC cells growth in vitro and in a new orthotopic mice model
24. P.1.133: PONTIN52 IS A PPARGAMMA TARGET GENE AND ITS KNOCKDOWN REDUCES HEPATIC CANCER IN AN ORTHOTOPIC-SINGENIC MOUSE MODEL
25. P.1.142: TELOMERASE ACTIVATED THYMIDINE ANALOGUE PRO-DRUG IS A NEW MOLECULE TARGETING HEPATOCELLULAR CARCINOMA
26. P.153 PES-003, A NEW PRO-DRUG FOR PANCREATIC CANCER THERAPY
27. T.N.23 ROSIGLITAZONE INHIBITS HEPATOCELLULAR CANCER CELL GROWTH AND DOWN-REGULATES RUVBL-1: EVIDENCES OF A PPARgamma-INDEPENDENT MECHANISM OF CELL GROWTH INHIBITION
28. IDENTIFICATION OF MODULATED PROTEINS BY THIAZOLIDINEDIONES TREATMENT IN HBV TRANSGENIC MOUSE MODEL
29. THE NUCLEAR RECEPTOR ARP-1: A NOVEL REGULATOR OF PROFIBROGENIC RESPONSE DURING LIVER FIBROSIS
30. THIAZOLIDINEDIONES INHIBIT HEPATIC TUMOUR FORMATION IN HBV TRANSGENIC MICE BY A PPARγ-INDEPENDENT REGULATION OF NUCLEOPHOSMIN
31. SILENCING OF ARP-1 NUCLEAR RECEPTOR REDUCES PANCREATIC CANCER GROWTH IN VITRO AND IN VIVO
32. Acetaldehyde induced collagen synthesis by inhibition of ppargamma transcriptional activity via hydrogen peroxide-mediated activaction of C-ABL non-receptor tyrosine kinase in human hepatic stellate cells
33. 11 Genisten significantly reduces HCC development in HBV transgenic mice
34. 4 PS Antidiabetic thiazolidinediones inhibit growth and invasiveness of pancreatic cancer cells via a ppargamma dependent and independent manner
35. Reactive oxygen species (ROS) modulate different profibrogenic responses in human hepatic stellate cells
36. Antidiabetic thiazolidinediones inhibit collagen synthesis and hepatic stellate cells activation in models of liver fibrosis
37. Peroxisome proliferator-activated receptor γ (PPARγ) transcriptional control is involved in hepatic stellate cells activation and modulates collagen synthesis
38. Acetaldehyde inhibits peroxisome proliferator-activated receptor γ (PPARγ) transcriptional activity in hepatic stellate cells
39. Human pancreatic adenocarcinoma cells modulate pancreatic stellate cells (PSC) activation and collagen synthesis
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