28 results on '"Jonas, Jean-Christophe"'
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2. Mechanisms of Control of the Free Ca2+ Concentration in the Endoplasmic Reticulum of Mouse Pancreatic β-Cells: Interplay With Cell Metabolism and [Ca2+]c and Role of SERCA2b and SERCA3
3. Probe-Independent and Direct Quantification of Insulin mRNA and Growth Hormone mRNA in Enriched Cell Preparations
4. Mechanisms of Pancreatic β-Cell Death in Type 1 and Type 2 Diabetes: Many Differences, Few Similarities
5. SERCA3 Ablation Does Not Impair Insulin Secretion but Suggests Distinct Roles of Different Sarcoendoplasmic Reticulum Ca2+ Pumps for Ca2+ Homeostasis in Pancreatic β-cells
6. Increased Glucose Sensitivity of Stimulus-Secretion Coupling in Islets From Psammomys obesus After Diet Induction of Diabetes
7. Do Oscillations of Insulin Secretion Occur in the Absence of Cytoplasmic Ca2+ Oscillations in β-Cells?
8. Control Mechanisms of the Oscillations of Insulin Secretion In Vitro and In Vivo
9. Signals and Pools Underlying Biphasic Insulin Secretion
10. Increased Expression of Antioxidant and Antiapoptotic Genes in Islets That May Contribute to β-Cell Survival During Chronic Hyperglycemia
11. β-Cell Adaptation to Hyperglycemia
12. Potential Role of the Early Response Gene c-myc in β-Cell Adaptation to Changes in Glucose Concentration
13. Temporal and Quantitative Correlations Between Insulin Secretion and Stably Elevated or Oscillatory Cytoplasmic Ca2+ in Mouse Pancreatic beta-Cells
14. Somatostatin Is Only Partly Required for the Glucagonostatic Effect of Glucose but Is Necessary for the Glucagonostatic Effect of KATP Channel Blockers
15. Somatostatin Is Only Partly Required for the Glucagonostatic Effect of Glucose but Is Necessary for the Glucagonostatic Effect of KATP Channel Blockers.
16. Activation of Protective and Destructive Stress Genes Accompanies Beta Cell Hypertrophy in Hyperglycemic Rats
17. Islet Cell Specific Gene Expression Is Normal Following Adaptation in [Beta]-Cell Mass To Substrate Oversupply
18. Inositol Trisphosphate Receptor Expression in Rat Pancreatic Islets Is Modulated by Cyclic AMP and Glucose
19. Increased Glucose Sensitivity of Stimulus-Secretion Coupling in Islets FromPsammomys obesusAfter Diet Induction of Diabetes
20. Temporal and Quantitative Correlations Between Insulin Secretion and Stably Elevated or Oscillatory Cytoplasmic Ca2+ in Mouse Pancreatic β-Cells
21. Rapid opposite effects of glucose on XBP1 mRNA unconventional splicing and GADD153 expression in cultured rat islets
22. Mechanisms of Control of the Free Ca2+ Concentration in the Endoplasmic Reticulum of Mouse Pancreatic β-Cells.
23. SERCA3 ablation does not impair insulin secretion but suggests distinct roles of different sarcoendoplasmic reticulum Ca(2+) pumps for Ca(2+) homeostasis in pancreatic beta-cells.
24. Do oscillations of insulin secretion occur in the absence of cytoplasmic Ca2+ oscillations in beta-cells?
25. Increased expression of antioxidant and antiapoptotic genes in islets that may contribute to beta-cell survival during chronic hyperglycemia.
26. Potential role of the early response gene c-myc in beta-cell adaptation to changes in glucose concentration.
27. Somatostatin Is Only Partly Required for the Glucagonostatic Effect of Glucose but Is Necessary for the Glucagonostatic Effect of K ATP Channel Blockers.
28. Mechanisms of pancreatic beta-cell death in type 1 and type 2 diabetes: many differences, few similarities.
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