24 results on '"MYERS, A. G."'
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2. Nucleus of the Solitary Tract Serotonin 5-HT2C Receptors Modulate Food Intake
3. A Parabrachial-Hypothalamic Cholecystokinin Neurocircuit Controls Counterregulatory Responses to Hypoglycemia
4. The Thermogenic Effect of Leptin Is Dependent on a Distinct Population of Prolactin-Releasing Peptide Neurons in the Dorsomedial Hypothalamus
5. IRS2 Signaling in LepR-b Neurons Suppresses FoxO1 to Control Energy Balance Independently of Leptin Action
6. Challenges and Opportunities of Defining Clinical Leptin Resistance
7. Leptin Action via Neurotensin Neurons Controls Orexin, the Mesolimbic Dopamine System and Energy Balance
8. Endoplasmic Reticulum Stress Plays a Central Role in Development of Leptin Resistance
9. Leptin Acts via Leptin Receptor-Expressing Lateral Hypothalamic Neurons to Modulate the Mesolimbic Dopamine System and Suppress Feeding
10. Critical Role for Hypothalamic mTOR Activity in Energy Balance
11. The Geometry of Leptin Action in the Brain: More Complicated Than a Simple ARC
12. A New Role for Glia in Leptin Action and Energy Balance
13. Leptin Keeps Working, Even in Obesity
14. Critical role of STAT3 in leptin's metabolic actions
15. SnapShot: Neural Pathways that Control Feeding
16. Roles for leptin receptor/STAT3-dependent and -independent signals in the regulation of glucose homeostasis
17. How Is the Hungry Brain like a Sieve?
18. Setting the Tone: Reactive Oxygen Species and the Control of Appetitive Melanocortin Neurons
19. Role reversal: Brain insulin and liver STAT3
20. Retraction Notice to: Impairment of Central Leptin-Mediated PI3K Signaling Manifested as Hepatic Steatosis Independent of Hyperphagia and Obesity
21. RETRACTED: Impairment of Central Leptin-Mediated PI3K Signaling Manifested as Hepatic Steatosis Independent of Hyperphagia and Obesity
22. Leptin Does Not Directly Affect CNS Serotonin Neurons to Influence Appetite
23. Insulin action in the brain contributes to glucose lowering during insulin treatment of diabetes
24. Impairment of Central Leptin-Mediated PI3K Signaling Manifested as Hepatic Steatosis Independent of Hyperphagia and Obesity.
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