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3. DNA polymerase θ protects leukemia cells from metabolically induced DNA damage.

4. Tyrosine kinase inhibitor-induced defects in DNA repair sensitize FLT3(ITD)-positive leukemia cells to PARP1 inhibitors.

5. Ruxolitinib-induced defects in DNA repair cause sensitivity to PARP inhibitors in myeloproliferative neoplasms.

6. Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases.

7. Personalized synthetic lethality induced by targeting RAD52 in leukemias identified by gene mutation and expression profile.

8. Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells.

9. Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors.

10. Targeting RAD51 phosphotyrosine-315 to prevent unfaithful recombination repair in BCR-ABL1 leukemia.

11. Enhanced phosphorylation of Nbs1, a member of DNA repair/checkpoint complex Mre11-RAD50-Nbs1, can be targeted to increase the efficacy of imatinib mesylate against BCR/ABL-positive leukemia cells.

12. BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance.

13. BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks.

14. p210 BCR/ABL kinase regulates nucleotide excision repair (NER) and resistance to UV radiation.

15. Complementary functions of the antiapoptotic protein A1 and serine/threonine kinase pim-1 in the BCR/ABL-mediated leukemogenesis.

16. Role of p53 in hematopoietic recovery after cytotoxic treatment.

17. The SH3 domain contributes to BCR/ABL-dependent leukemogenesis in vivo: role in adhesion, invasion, and homing.