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Start Over Author "Gallagher, Peter" Journal bipolar disorders
10 results on '"Gallagher, Peter"'

1. Efficacy and safety of established and off‐label ADHD drug therapies for cognitive impairment or attention‐deficit hyperactivity disorder symptoms in bipolar disorder: A systematic review by the ISBD Targeting Cognition Task Force

Author

Miskowiak, Kamilla W., primary, Obel, Zacharias K., additional, Gugliemo, Riccardo, additional, Bonnin, Caterina del Mar, additional, Bowie, Christopher R., additional, Balanzá‐Martínez, Vicente, additional, Burdick, Katherine E., additional, Carvalho, Andre F., additional, Dols, Annemieke, additional, Douglas, Katie, additional, Gallagher, Peter, additional, Kessing, Lars V., additional, Lafer, Beny, additional, Lewandowski, Kathryn E., additional, López‐Jaramillo, Carlos, additional, Martinez‐Aran, Anabel, additional, McIntyre, Roger S., additional, Porter, Richard J., additional, Purdon, Scot E., additional, Schaffer, Ayal, additional, Stokes, Paul R. A., additional, Sumiyoshi, Tomiki, additional, Torres, Ivan J., additional, Van Rheenen, Tamsyn E., additional, Yatham, Lakshmi N., additional, Young, Allan H., additional, Vieta, Eduard, additional, and Hasler, Gregor, additional

Published
2024
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3. Processing speed and sustained attention in bipolar disorder and major depressive disorder: A systematic review and meta-analysis.

Author

Little, Bethany, Anwyll, Megan, Norsworthy, Laura, Corbett, Luke, Schultz-Froggatt, Mia, and Gallagher, Peter

Subjects
COGNITIVE processing speed, BIPOLAR disorder, NEUROPSYCHOLOGICAL tests, COGNITION disorders, COGNITIVE testing
Abstract

Objectives: Cognitive impairment is a core feature of bipolar disorder (BD) and major depressive disorder (MDD). Deficits in processing speed (PS) and sustained attention (SA) may be particularly impaired and may underpin a broader profile of deficits, however current knowledge of the nature of these impairments is limited by heterogeneous results in the literature. Few reviews to date have attempted to disentangle sources of heterogeneity to assess the presence and magnitude of impairments in PS and SA in BD and MDD. Methods: One hundred and three studies were reviewed to examine performance in tests of PS and SA in BD (n = 3452) and MDD (n = 5461) compared to healthy controls (n = 8016). Neuropsychological methodology used in the literature was summarised. Data were meta-analysed to assess impairments in PS and SA for each neuropsychological test separately. Subgroup analysis was performed across mood states to investigate sources of heterogeneity. Results: Impairments were found across most neuropsychological tests, with small to large effect sizes for BD (range: d = 0.19-0.96) and MDD (range: d = 0.29-0.86). Impairments were present in symptomatic states and euthymia in most cases. Some outcome measures were not impaired in euthymia. Heterogeneity was observed for most neuropsychological tests and remained after separating by mood state. There inadequate data to meta-analyse some outcome measures, particularly for symptomatic groups. Conclusion: Impairments in PS and SA in BD and MDD can be observed across most neuropsychological tests. Future research should further investigate the nature of these impairments across mood states, controlling for clinical confounds. [ABSTRACT FROM AUTHOR]

Published
2024
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4. Can magnetic resonance imaging enhance the assessment of potential new treatments for cognitive impairment in mood disorders? A systematic review and position paper by the International Society for Bipolar Disorders Targeting Cognition Task Force

Author

Miskowiak, Kamilla W., primary, Yalin, Nefize, additional, Seeberg, Ida, additional, Burdick, Katherine E., additional, Balanzá‐Martínez, Vicent, additional, Bonnin, Caterina del Mar, additional, Bowie, Christopher R., additional, Carvalho, Andre F., additional, Dols, Annemieke, additional, Douglas, Katie, additional, Gallagher, Peter, additional, Hasler, Gregor, additional, Kessing, Lars V., additional, Lafer, Beny, additional, Lewandowski, Kathryn E., additional, López‐Jaramillo, Carlos, additional, Martinez‐Aran, Anabel, additional, McIntyre, Roger S., additional, Porter, Richard J., additional, Purdon, Scot E., additional, Schaffer, Ayal, additional, Sumiyoshi, Tomiki, additional, Torres, Ivan J., additional, Van Rheenen, Tamsyn E., additional, Yatham, Lakshmi N., additional, Young, Allan H., additional, Vieta, Eduard, additional, and Stokes, Paul R. A., additional

Published
2022
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5. Randomised controlled cognition trials in remitted patients with mood disorders published between 2015 and 2021: A systematic review by the International Society for Bipolar Disorders Targeting Cognition Task Force

Author

Miskowiak, Kamilla W., primary, Seeberg, Ida, additional, Jensen, Mette B., additional, Balanzá‐Martínez, Vicent, additional, del Mar Bonnin, Caterina, additional, Bowie, Christopher R., additional, Carvalho, Andre F., additional, Dols, Annemieke, additional, Douglas, Katie, additional, Gallagher, Peter, additional, Hasler, Gregor, additional, Lafer, Beny, additional, Lewandowski, Kathryn E., additional, López‐Jaramillo, Carlos, additional, Martinez‐Aran, Anabel, additional, McIntyre, Roger S., additional, Porter, Richard J., additional, Purdon, Scot E., additional, Schaffer, Ayal, additional, Stokes, Paul, additional, Sumiyoshi, Tomiki, additional, Torres, Ivan J., additional, Van Rheenen, Tamsyn E., additional, Yatham, Lakshmi N., additional, Young, Allan H., additional, Kessing, Lars V., additional, Burdick, Katherine E., additional, and Vieta, Eduard, additional

Published
2022
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7. Affective cognition in bipolar disorder: A systematic review by the ISBD targeting cognition task force

Author

Miskowiak, Kamilla W., primary, Seeberg, Ida, additional, Kjaerstad, Hanne L., additional, Burdick, Katherine E., additional, Martinez‐Aran, Anabel, additional, Bonnin, Caterina, additional, Bowie, Christopher R., additional, Carvalho, Andre F., additional, Gallagher, Peter, additional, Hasler, Gregor, additional, Lafer, Beny, additional, López‐Jaramillo, Carlos, additional, Sumiyoshi, Tomiki, additional, McIntyre, Roger S., additional, Schaffer, Ayal, additional, Porter, Richard J., additional, Purdon, Scot, additional, Torres, Ivan J., additional, Yatham, Lakshmi N., additional, Young, Allan H., additional, Kessing, Lars V., additional, Van Rheenen, Tamsyn E., additional, and Vieta, Eduard, additional

Published
2019
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8. The International Consortium Investigating Neurocognition in Bipolar Disorder (ICONIC‐BD)

Author

Burdick, Katherine E., primary, Millett, Caitlin E., additional, Bonnín, Caterina del Mar, additional, Bowie, Christopher R., additional, Carvalho, Andre F., additional, Eyler, Lisa T., additional, Gallagher, Peter, additional, Harvey, Philip D., additional, Kessing, Lars V., additional, Lafer, Beny, additional, Langenecker, Scott A., additional, Lewandowski, Kathryn E., additional, López‐Jaramillo, Carlos, additional, Marshall, David F., additional, Martinez‐Aran, Anabel, additional, McInnis, Melvin G., additional, McIntyre, Roger S., additional, Miskowiak, Kamilla W., additional, Porter, Richard J., additional, Purdon, Scot E., additional, Ryan, Kelly A., additional, Sumiyoshi, Tomiki, additional, Torres, Ivan J., additional, Van Rheenen, Tamsyn E., additional, Vieta, Eduard, additional, Woodward, Neil D., additional, Yatham, Lakshmi N., additional, and Young, Allan H., additional

Published
2019
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10. Multivariate brain-cognition associations in euthymic bipolar disorder.

Author

Little B, Flowers C, Blamire A, Thelwall P, Taylor JP, Gallagher P, Cousins DA, and Wang Y

Abstract

Background: People with bipolar disorder (BD) tend to show widespread cognitive impairment compared to healthy controls. Impairments in processing speed (PS), attention and executive function (EF) may represent 'core' impairments that have a role in wider cognitive dysfunction. Cognitive impairments appear to relate to structural brain abnormalities in BD, but whether core deficits are related to particular brain regions is unclear and much of the research on brain-cognition associations is limited by univariate analysis and small samples., Methods: Euthymic BD patients (n = 56) and matched healthy controls (n = 26) underwent T1-weighted MRI scans and completed neuropsychological tests of PS, attention and EF. We utilised public datasets to develop normative models of cortical thickness (n = 5977) to generate robust estimations of cortical abnormalities in patients. Canonical correlation analysis was used to assess multivariate brain-cognition associations in BD, controlling for age, sex and premorbid IQ., Results: BD showed impairments on tests of PS, attention and EF, and abnormal cortical thickness in several brain regions compared to healthy controls. Impairments in tests of PS and EF were most strongly associated with cortical thickness in the left inferior temporal, right entorhinal and right temporal pole areas., Conclusion: Impairments in PS, attention and EF can be observed in euthymic BD and may be related to abnormal cortical thickness in temporal regions. Future research should continue to leverage normative modelling and multivariate methods to examine complex brain-cognition associations in BD. Future research may benefit from exploring covariance between traditional brain structural morphological metrics such as cortical thickness, cortical volume and surface area., (© 2024 The Author(s). Bipolar Disorders published by John Wiley & Sons Ltd.)

Published
2024
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