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The question as to whether or not uterine myoma causes heart disease has been argued since 1885 when Hof meier1 started the discussion by saying that he believed this type of tumor could injure the heart. Most of the early writers agreed with Hofmeier, although in 1890 Williams2 expressed a contrary opinion when he said that in his experience, uterine fibroid was less often associated with heart disease than was any other pelvic lesion. Strassmann and Lehmann3 in 1898 reported a series of 71 cases of uterine myoma in 34 per cent of which there was evidence of cardiac damage. These authors believed that the tumor could cause degenerative changes in the heart muscle. Other early writers who agreed with the views of Strassmann and Lehmann were Wilson,4 Fleck,5 Baldy,6 Cumston,7 Boldt8 and Kessler.9 The question as to whether secondary anemia from menorrhagia and metrorrhagia in these cases was not

PRIOR to the introduction of chemotherapy and antibiotics, bacterial endocarditis was almost always a fatal disease.1 During the past ten years cure rates of 66% to 83% for the subacute forms have been reported, indicating a far better prognosis for patients with this disease today.2,3 At the Philadelphia General Hospital a previous study showed a significant decrease in the number of patients dying with bacterial endocarditis during a recent ten-year period.4 Despite these findings and improved methods of therapy, the incidence of 9.6 patients with bacterial endocarditis per 1,000 autopsies performed at this institution is indicative of a considerable fatality rate. This observation, coupled with the deaths of several patients on the medical wards during the past year in whom bacterial endocarditis was totally unrecognized, led to the present study of 159 patients hospitalized at the Philadelphia General Hospital with this disease from 1954 to 1963. Special

Recent rather numerous reports from the various army camps throughout the country concerning the incidence and causative agent of pneumonia, during the last winter, have mentioned frequent infection with Streptococcus hemolyticus and less frequent occurrence of pneumococcus infection. Reports from the different camps would indicate that the predominating organism is not the same in all localities. In Camps Funston, Dodge, Custer and Lee hemolytic streptococcus has been reported as the prevailing organism. At Camps Wheeler, Beauregard, Hancock, Jackson, Logan, Sheridan and Travis more pneumococcus infections are reported. In the findings so far published attention has been directed chiefly to the study of bronchopneumonia and, more especially, empyema. There has been very little comparative study of lobar and bronchopneumonia. The studies of the commission headed by Cole and McCallum1 at Fort Sam Houston, have been made with this point in view and their results would indicate that the true lobar pneumonia

THE RELATIONSHIP between herpes zoster and chickenpox was first pointed out by von Bókay,1 of Budapest, in 1888, when he reported cases of the latter disease in members of two families after episodes of herpes zoster in other members. Since then, there has been a growing interest in the subject, especially in the foreign literature. For example, Le Feuvre,2 in 1917, found in the literature 37 cases of chickenpox after exposure to herpes zoster (shingles) and added 4 cases of his own, making a total of 41 cases. He also reported the converse, 5 cases of herpes zoster after exposure to chickenpox. Moreover, he summarized the reports of 4 cases, which had been noted in the British literature between 1913 and 1916, of concurrent herpes zoster and varicella. In all these cases, the chickenpox followed the herpes zoster after an interval of two to four days. Le