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Start Over Author "Routier E" Journal annales de dermatologie et de venereologie
42 results on '"Routier E"'

4. Identification de TBX3 comme nouveau régulateur de l’expression de PDL1 dans le mélanome

Author

Darbord, D., primary, Cerezo, M., additional, Agoussi, S., additional, Allard, D., additional, Claps, G., additional, Girault, I., additional, Guemiri, R., additional, Libenciuc, C., additional, Moya-Plana, A., additional, Quidville, V., additional, Routier, E., additional, Roy, S., additional, Shen, S., additional, Vagner, S., additional, and Robert, C., additional

Published
2018
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11. VISMONEO : étude de phase II du vismodegib en traitement néoadjuvant du carcinome basocellulaire localement avancé : caractéristiques des patients

Author

Basset-Seguin, N., primary, Dupuy, A., additional, Saiag, P., additional, Sophie Dalac-Rat, D.-R., additional, Guillot, B., additional, Routier, E., additional, Leccia, M.-T., additional, Duhamel, A., additional, Mirabel, X., additional, Benbouta, I., additional, Mirakovska, L., additional, Meddour, D., additional, Dib, M., additional, Mahmoudi, A., additional, Guerreschi, P., additional, and Mortier, L., additional

Published
2016
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24. Tumeurs kératinocytaires induites par activation paradoxale de la voie MAPK sous inhibiteur de BRAF – Étude histologique, génétique, et virologique de 57 tumeurs secondaires

Author

Boussemart, L., primary, Girault, I., additional, Mateus, C., additional, Thomas, M., additional, Routier, E., additional, Tomasic, G., additional, Wechsler, J., additional, Kamsu-Kom, N., additional, Roy, S., additional, Favre, M., additional, Lacroix, L., additional, and Robert, C., additional

Published
2013
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28. Vemurafenib et radio-sensibilisation

Author

Boussemart, L., primary, Boivin, C., additional, Tao, Y., additional, Tomasic, G., additional, Routier, E., additional, Mateus, C., additional, Claveau, J., additional, and Robert, C., additional

Published
2012
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32. Panniculite sous inhibiteur de BRAF et MEK

Author

Mateus, C., primary, Maksimovic, L., additional, Routier, E., additional, Tomasic, G., additional, Wechsler, J., additional, Kamsu-Kom, N., additional, Boussemart, L., additional, Thomas, M., additional, and Robert, C., additional

Published
2011
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41. [Acral melanoma in a patient with hereditary keratoderma of the palms and soles (mal de Meleda): A chance association?]

Author

Korbi M, Hickman G, Routier E, Bagot M, and Bourrat E

Subjects
Humans, Male, Middle Aged, Fingers pathology, Keratoderma, Palmoplantar pathology, Melanoma pathology, Skin Neoplasms pathology
Abstract

Introduction: Development of acral malignant melanoma in Mal de Meleda is highly unusual. As far as we could ascertain, to date, only 10 previous cases have been published. Herein, we report a new case., Observation: A 64-year-old Algerian man was followed for familial Mal de Meleda. The diagnosis was based on clinical presentation as he had a non-syndromic hereditary foul-smelling and yellowish palmoplantar keratoderma transgrediens. After the failure of acitretin, which had not prevented retractile and mutilating progression of the palmoplantar keratoderma, he had undergone surgery with graft excision of both palms. At the age of 59 years, he presented a tumor on the dorsal aspect of the 1 st phalanx of the 3 rd finger of the right hand in a non-grafted area. The diagnosis of acral melanoma was confirmed histologically. The radiological findings showed a specific homolateral axillary adenopathy. He underwent digital amputation of the 3 rd finger, with lymph node dissection and chemotherapy involving dacarbazine. Follow-up at 5 years showed complete remission of the melanoma., Discussion: Mal de Meleda is a hereditary palmoplantar keratoderma due to mutation of the SLURP1 gene. Clinical diagnosis is based on the typical phenotype in adulthood. The occurrence of acral melanoma, which is a rare form of melanoma (1 to 7%), especially in the fingers, together with an unusual palmoplantar keratoderma in a subject of type IV phototype does not appear to be a chance event. This association seems to be the outcome of immune dysregulation rather than of chronic inflammation., (Copyright © 2019 Elsevier Masson SAS. All rights reserved.)

Published
2019
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42. [Two cases of psoas abscesses caused by group A beta-haemolytic streptococcal infection with a cutaneous portal of entry].

Author

Routier E, Bularca S, Sbidian E, Roujeau JC, and Bagot M

Subjects
Adult, Amoxicillin therapeutic use, Anti-Bacterial Agents therapeutic use, Combined Modality Therapy, Drainage, Fasciitis, Necrotizing microbiology, Female, Humans, Male, Middle Aged, Psoas Abscess diagnosis, Psoas Abscess diagnostic imaging, Psoas Abscess drug therapy, Psoas Abscess etiology, Psoas Abscess surgery, Skin microbiology, Streptococcal Infections diagnosis, Streptococcal Infections drug therapy, Streptococcal Infections etiology, Streptococcal Infections surgery, Substance Abuse, Intravenous complications, Systemic Inflammatory Response Syndrome etiology, Tomography, X-Ray Computed, Wound Infection complications, Wound Infection microbiology, Fasciitis, Necrotizing complications, Psoas Abscess microbiology, Skin injuries, Streptococcal Infections microbiology, Streptococcus pyogenes isolation & purification
Abstract

Background: Psoas abscess is a rare clinical entity that occurs chiefly after intra-abdominal or retroperitoneal infection. We report two cases of psoas abscesses caused by group A beta-haemolytic streptococcal infection having a cutaneous portal of entry., Case Reports: The first patient, a 50-year-old man, was feverish and had ulcerative and necrotic cutaneous lesions evocative of ecthyma that were progressing for three months and were recently associated with a painful mass in the left iliac fossa, leading to difficulties in walking. The second patient, a 35-year-old woman with a medical history of intravenous drug addiction, was admitted to intensive care for sepsis syndrome following group A beta-haemolytic streptococcal infection with a cutaneous portal of entry (swelling on left lower limb). She remained unaccountably subfebrile 10 days after the start of antibiotic therapy with amoxicillin. Abdominal CAT scans for each patient confirmed the diagnosis of left psoas abscess. For the first patient, the same group A beta-haemolytic streptococcus was isolated in drainage fluid and at the cutaneous injury site. The outcome was favourable in both cases following extensive intravenous antibiotic therapy (amoxicillin) combined with percutaneous drainage (in the first case)., Discussion: Psoas abscess can occur after locoregional infection and the portals of entry are usually gastro-intestinal, musculoskeletal or genitourinary, with many organisms capable of causing such secondary abscesses. Psoas abscess can also be a primary clinical event. Staphylococcus aureus is the most common causative organism. The presented cases comprised secondary psoas abscesses with a cutaneous portal of entry. Since the complete set of three evocative symptoms (prolonged fever, pain and psoitis) is frequently seen late, diagnosis must be made on the basis of prolonged infectious state or unaccountable feverish abdominal pain. Diagnosis is based on abdominal CAT scan and treatment involves the use of appropriate antibiotics as well as percutaneous or surgical drainage of the abscess. The mortality rate in this patient population remains high with survival being dependent on prompt initiation of therapy., (Copyright 2010 Elsevier Masson SAS. All rights reserved.)

Published
2010
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