21 results on '"Yi Ting Wang"'
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2. Tau phosphorylation is more closely associated with amyloid‐β plaques than with tau neurofibrillary tangles
3. Apolipoprotein E ε4 associates with microglial activation in early Braak regions independently of amyloid‐β and tau
4. Impact of meningeal and age‐related off‐target binding on longitudinal [ 18 F]MK6240 quantification
5. pTau heterogeneity as a measure for disease severity in incipient Alzheimer's disease
6. Verbal recognition declines in later Braak Stages compared to verbal delayed recall
7. Neuroinflammation is associated with the rising of early Alzheimer’s disease pathology in amyloid‐negative elderly
8. Biomarker modelling of Alzheimer's disease using in vivo Braak staging
9. Brain TSPO expression is associated with plasma pTau181 & pTau231 across the AD spectrum
10. Visual memory scores are associated with lateralization of tau in the medial temporal lobe
11. Tau‐load in the lingual gyrus impacts anxiety levels during the COVID‐19 pandemic in participants of longitudinal observational studies in aging
12. Discrepancy between plasma pTau181 and tau‐PET statuses
13. Tau accumulation using [ 18 F]MK6240 PET is associated with increase in executive dysfunction in prodromal AD
14. The neuroinflammation signatures in different stages of the Alzheimer’s disease continuum
15. Visual memory test equal to commonly used verbal memory test in predicting tau in the medial temporal lobe
16. Effect of temporal meta‐ROI and Braak1&2 ROI on the dissociation between plasma pTau181 and PET statuses
17. APOE4 packs a punch in women: Sex‐specific vulnerability for tau and neuroinflammation
18. Tau deposition assessed by [ 18 F]MK6240 PET is associated with longitudinal decrease in grey matter density across the spectrum of Alzheimer’s disease
19. Microglial sex dimorphism poses greater vulnerability to Alzheimer’s disease in females: A cross‐species study
20. P3-328: DOES INSULIN RESISTANCE INFLUENCE WHITE MATTER LESIONS IN NON-DIABETIC AD SUBJECTS?
21. P2-358: INSULIN RESISTANCE INFLUENCES HIPPOCAMPAL GLUCOSE METABOLISM RATHER THAN HIPPOCAMPAL VOLUME IN NON-DIABETIC ALZHEIMER'S DISEASE PATIENTS
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