21 results on '"Sanjo A"'
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2. MOLECULAR IMAGING AND TREATMENT OF ALZHEIMER’S DISEASE BY DEVELOPING AMYLOID-β OLIGOMER ANTIBODIES THAT CROSS THE BLOOD-BRAIN BARRIER
3. DOT BLOT ASSAY FOR QUANTITATIVE MEASUREMENT OF AMYLOID BETA OLIGOMER
4. POSSIBLE HUMAN-TO-HUMAN TRANSMISSION OF CEREBRAL β-AMYLOIDOSIS VIA CADAVERIC DURA MATER GRAFTING
5. IMMUNOHISTOLOGICAL ANALYSIS OF AMYLOID-β OLIGOMER DEPOSITS IN APP KNOCK-IN MICE
6. COLOCALIZATION OF Aβ42 WITH PRPSC-PLAQUES IN THE BRAIN OF GERSTMANN-STRÄUSSLER-SCHEINKER DISEASE WITH THE P105L MUTATION
7. Relationship between attention and gait cycle in patients with Alzheimer’s disease
8. RELATIONSHIP BETWEEN GAIT AND COGNITIVE FUNCTION IN PATIENTS WITH ALZHEIMER'S DISEASE
9. Visual reproduction on the Wechsler Memory Scale-Revised as a predictor of Alzheimer's disease in Japanese people with mild cognitive impairment
10. Quantitative analysis of gait disorders in people with Alzheimer's disease by using a portable gait rhythmograph
11. Pattern of beta-amyloid production by mouse embryonic fibroblasts overexpressing psen1 with I143T mutation
12. An Effect of I143T PSEN1 Mutation on The Generation of Amyloid B in Culture Cells
13. Human prion diseases in Japan: analysis of 1,552 patients in a prospective 11-year surveillance
14. Usefulness of logical memory II of WMS-R in memory clinic outpatients
15. Presenilin-1 holoprotein is an interacting partner of sarco endoplasmic reticulum calcium-ATPase and confers resistance to endoplasmic reticulum stress
16. Protective effect of endogenous PS1 holoprotein under ER stress condition
17. P3‐072: QUANTITATIVE MEASUREMENT OF AMYLOID BETA OLIGOMER IN MOUSE BRAIN USING DOT BLOT ASSAY.
18. P2-157: Serine422 phosphorylation of tau protein by oxidative stress via c-Jun N-terminal kinase (JNK) activation
19. P1‐103: DOT BLOT ASSAY FOR QUANTITATIVE MEASUREMENT OF AMYLOID BETA OLIGOMER.
20. O1-02-01: A novel presenilin complex component which modulates gamma- but not epsilon-secretase activity
21. P2‐054: MOLECULAR IMAGING AND TREATMENT OF ALZHEIMER'S DISEASE BY DEVELOPING AMYLOID‐β OLIGOMER ANTIBODIES THAT CROSS THE BLOOD‐BRAIN BARRIER.
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