72 results on '"Yoon-Sang Oh"'
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2. Ran-Binding Protein 3 Phosphorylation Links the Ras and PI3-Kinase Pathways to Nucleocytoplasmic Transport
3. mTOR inhibition reprograms cellular proteostasis by regulating eIF3D-mediated selective mRNA translation and promotes cell phenotype switching
4. Improved recurrence-free survival in hepatocellular carcinoma patients with post-transplant plasma exchange
5. Figure S2 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
6. Figure S1 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
7. Figure S1 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
8. Figure S2 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
9. Data from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
10. Figure S1 Identification of rapamycin collaborators through small molecule high-throughout screening from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
11. Figure S2 Inhibition of mTORC1 and glutamate cysteine ligase induces cytotoxicity in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
12. SI materials and methods from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
13. Supplementary Figures 1 - 5 from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
14. Figure S3. Elevated levels of reactive oxygen species are responsible for cell death in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
15. SI materials and methods from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
16. Supporting information_Figure legends from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
17. Supplementary Figures 1 - 5 from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
18. Figure S4. The combination of BSO and rapamycin induces mitochondrial ROS and alters mitochondrial morphology from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
19. Supplementary Figure Legend from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
20. Supporting information_Figure legends from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
21. Figure S1 Identification of rapamycin collaborators through small molecule high-throughout screening from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
22. Figure S4. The combination of BSO and rapamycin induces mitochondrial ROS and alters mitochondrial morphology from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
23. Figure S3. Elevated levels of reactive oxygen species are responsible for cell death in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
24. Data from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
25. Data from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
26. Figure S2 Inhibition of mTORC1 and glutamate cysteine ligase induces cytotoxicity in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
27. Supplementary Figure Legend from Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
28. Data from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
29. The PIKK-AKT connection in the DNA damage response
30. Editorial Note to: Glucose Addiction of TSC Null Cells Is Caused by Failed mTORC1-Dependent Balancing of Metabolic Demand with Supply
31. The primitive growth factor NME7AB induces mitochondrially active naïve-like pluripotent stem cells
32. Antioxidant Character of Selenium and Its Survival Signals by Activation of PI3-K/Akt Pathways
33. Dynamic Incorporation of Histone H3 Variants into Chromatin Is Essential for Acquisition of Aggressive Traits and Metastatic Colonization
34. Corrigendum to “Metabolic switching in pluripotent stem cells reorganizes energy metabolism and subcellular organelles” [379 (2019) 55–64]
35. Metabolic switching in pluripotent stem cells reorganizes energy metabolism and subcellular organelles
36. ERK2 regulates epithelial-to-mesenchymal plasticity through DOCK10-dependent Rac1/FoxO1 activation
37. mTORC1 Promotes Metabolic Reprogramming by the Suppression of GSK3-Dependent Foxk1 Phosphorylation
38. RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
39. Focal Adhesion- and IGF1R-Dependent Survival and Migratory Pathways Mediate Tumor Resistance to mTORC1/2 Inhibition
40. Pharmacologic Targeting of S6K1 in PTEN-Deficient Neoplasia
41. mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
42. ERK2 Mediates Metabolic Stress Response to Regulate Cell Fate
43. The mTORC1/S6K1 Pathway Regulates Glutamine Metabolism through the eIF4B-Dependent Control of c-Myc Translation
44. Casein Kinase 1ϵ Promotes Cell Proliferation by Regulating mRNA Translation
45. Rapamycin Resistance: mTORC1 Substrates Hold Some of the Answers
46. Transcriptional Repression of Bim by a Novel YY1-RelA Complex Is Essential for the Survival and Growth of Multiple Myeloma
47. Regulation of endothelial cell morphogenesis by the protein kinase D (PKD)/glycogen synthase kinase 3 (GSK3)β pathway
48. Glycogen synthase kinase (GSK)-3 and mammalian target of rapamycin complex 1 (mTORC1) cooperate to regulate protein S6 kinase 1 (S6K1)
49. Integrin Trafficking and Tumor Progression
50. Glycogen synthase kinase (GSK)-3 promotes p70 ribosomal protein S6 kinase (p70S6K) activity and cell proliferation
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