205 results on '"Yokoi, Hideki"'
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2. A Case of C3 Nephritis With a Rare Variant of the CFHR5 Gene
3. Renal interstitial hydrostatic pressure: a culprit of pericyte detachment in renal congestion
4. Dual deletion of guanylyl cyclase-A and p38 mitogen-activated protein kinase in podocytes with aldosterone administration causes glomerular intra-capillary thrombi
5. Matrix metalloproteinase-10 deficiency has protective effects against peritoneal inflammation and fibrosis via transcription factor NFκΒ pathway inhibition
6. A report of three cases of patients with tubulointerstitial nephritis with IgM-positive plasma cells, treatment, and serum-IgM as a sensitive marker for relapse
7. Sacubitril/valsartan ameliorates renal tubulointerstitial injury through increasing renal plasma flow in a mouse model of type 2 diabetes with aldosterone excess
8. Supplementary Figure 3 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
9. Supplementary Figure 5 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
10. Supplementary Figure 6 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
11. Supplementary Figure 5 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
12. Supplementary materials from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
13. Supplementary Figure 2 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
14. Supplementary Figure 1 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
15. Supplementary Figure 4 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
16. Supplementary Figure 3 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
17. Supplementary Figure 4 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
18. Supplementary materials from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
19. Supplementary Figure 2 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
20. Supplementary Figure 1 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
21. Supplementary Figure 6 from CTGF Mediates Tumor–Stroma Interactions between Hepatoma Cells and Hepatic Stellate Cells to Accelerate HCC Progression
22. Pathological social withdrawal in autism spectrum disorder: A case control study of hikikomori in Japan
23. Nephrotoxicity associated with anticancer agents: perspective on onconephrology from nephrologists
24. A study of Ta2O5 photonic crystals waveguide for Heat-Assisted Magnetic Recording (HAMR)
25. PS-B08-3: DILATED CARDIOMYOPATHY MOUSE MODEL AGGRAVATES ACUTE KIDNEY INJURY
26. PS-R04-2: A CASE OF SUCCESSFUL PERCUTANEOUS TRANSLUMINAL RENAL ANGIOPLASTY (PTRA) FOR RENAL ARTERY STENOSIS ASSOCIATED WITH TAKAYASU'S ARTERITIS
27. STAT3 is Activated by CTGF-mediated Tumor-stroma Cross Talk to Promote HCC Progression
28. PS-BPB05-9: DUAL DELETION OF GUANYLYL CYCLASE-A AND P38 MAPK IN PODOCYTES WITH ALDOSTERONE ADMINISTRATION CAUSES GLOMERULAR INTRACAPILLARY THROMBI
29. Invasive aspergillosis in the patient with focal segmental glomerulosclerosis initiating hemodialysis: a case report and mini-review
30. Genome-wide association study of the risk of chronic kidney disease and kidney-related traits in the Japanese population: J-Kidney-Biobank
31. Inhibition of mitochondrial fission protects podocytes from albumin-induced cell damage in diabetic kidney disease
32. Analysis of Pathological Activities of CCN Proteins in Fibrotic Diseases: Kidney Fibrosis
33. Production and Analysis of Conditional KO Mice of CCN2 in Kidney
34. Creation of magnetic thin film integrated optical non-reciprocal element using supersonic free jet PVD method
35. Severe eosinophilic pneumonia during the initiation of peritoneal dialysis：a case report and literature review
36. Resuscitation from severe accidental hypothermia by active core rewarming via an indwelling peritoneal dialysis catheter
37. Calcium channel blocker in patients with chronic kidney disease
38. Osteocrin ameliorates adriamycin nephropathy via p38 mitogen-activated protein kinase inhibition
39. Osteocrin, a bone-derived humoral factor, exerts a renoprotective role in ischemia–reperfusion injury in mice
40. Evaluation of Kidney Histological Images Using Unsupervised Deep Learning
41. Peritoneal Dialysis Guidelines 2019 Part 1 (Position paper of the Japanese Society for Dialysis Therapy)
42. Dysregulation of PI3K and Hippo signaling pathways synergistically induces chronic pancreatitis via CTGF upregulation
43. Monoclonal gammopathy of renal significance (MGRS)-related AL amyloidosis complicated by amyloid myopathy: a case report
44. MAGI-2 orchestrates the localization of backbone proteins in the slit diaphragm of podocytes
45. Proteolytic cleavage of Podocin by Matriptase exacerbates podocyte injury
46. Intensified hemodialysis for complicated pregnancy in a primigravida with advanced maternal age: a case report with literature review focusing on appropriate hemodialysis management during pregnancy
47. Classification of glomerular pathological findings using deep learning and nephrologist–AI collective intelligence approach
48. Resin Made Long-Period Fiber Grating Structure for Tunable Optical Filter inside Single-Mode Fiber
49. Transcription factor MafB in podocytes protects against the development of focal segmental glomerulosclerosis
50. P1663CLINICAL AND PATHOLOGICAL FACTORS INFLUENCING RESIDUAL RENAL FUNCTION AFTER LIVING DONOR NEPHRECTOMY
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