49 results on '"Wu, Yee Ling"'
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2. B cell intrinsic Toll-like receptor 9 signaling regulates class switching to IgE
3. An in vivo overexpression system to study the functions of complement activated products in a mouse model of lupus
4. Allergen inhalation transforms the lungs into a permissive environment for local IgE responses
5. The complement system and human autoimmune diseases
6. Complement C4 gene copy number variations and polymorphisms, autoantibodies, and clinical manifestations of juvenile dermatomyositis– a multi-center study
7. Low copy numbers of complementC4andC4Adeficiency are risk factors for myositis, its subgroups and autoantibodies
8. Lung memory B cells promote local IgE production in the respiratory tract
9. Loop extrusion promotes an alternate pathway for isotype switching
10. Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases
11. Complement in Rheumatic Diseases
12. Toll-like receptor signaling directly modulates B cell antibody responses
13. Complement Deficiencies in Human Systemic Lupus Erythematosus (SLE) and SLE Nephritis: Epidemiology and Pathogenesis
14. Complement Components C4A and C4B in Human Lupus
15. Elevated serum complement levels and higher gene copy number of complement C4B are associated with hypertension and effective response to statin therapy in childhood-onset systemic lupus erythematosus (SLE)
16. Opposite Profiles of Complement in Antiphospholipid Syndrome (APS) and Systemic Lupus Erythematosus (SLE) Among Patients With Antiphospholipid Antibodies (aPL)
17. Direct activation of Toll-like receptors induce differential B cell responses
18. 190 Opposite profiles of complement in antiphospholipid syndrome (APS) and systemic lupus erythematosus (SLE) among patients with antiphospholipid antibodies
19. Intrinsic transcriptional heterogeneity in B cells controls early class switching to IgE
20. Asynchronous digital activation of the germline Iγ1 and Iɛ promoters in single cells reveals that temporal control determines the fate of switching to IgE in B cells
21. Effects of Complement C4 Gene Copy-Number Variations, Gene Size Dichotomy and C4A-Deficiency on Genetic Risk and Clinical Presentation of East-Asian and European Systemic Lupus Erythematosus (SLE)
22. Early Components of the Complement Classical Activation Pathway in Human Systemic Autoimmune Diseases
23. Molecular fine-tuning of affinity maturation in germinal centers
24. Gene copy-number variations (CNVs) of complement C4 and C4A deficiency in genetic risk and pathogenesis of juvenile dermatomyositis
25. Three Distinct Profiles of Serum Complement C4 Proteins in Pediatric Systemic Lupus Erythematosus (SLE) Patients: Tight Associations of Complement C4 and C3 Protein Levels in SLE but not in Healthy Subjects
26. Active RNAP pre-initiation sites are highly mutated by cytidine deaminases in yeast, with AID targeting small RNA genes
27. Author response: Active RNAP pre-initiation sites are highly mutated by cytidine deaminases in yeast, with AID targeting small RNA genes
28. DNA deaminases induce break-associated mutation showers with implication of APOBEC3B and 3A in breast cancer kataegis
29. Abstract 611: AID/APOBEC cytidine deaminases can mimic the phenomenon of localized hypermutation in cancer orkataegis.
30. Author response: DNA deaminases induce break-associated mutation showers with implication of APOBEC3B and 3A in breast cancer kataegis
31. Increased frequency of complement C4B deficiency in rheumatoid arthritis
32. Gene CNVs and protein levels of complement C4A and C4B as novel biomarkers for partial disease remissions in new-onset type 1 diabetes patients
33. Sex-specific association of X-linked Toll-like receptor 7 (TLR7) with male systemic lupus erythematosus
34. Molecular basis of complement C1r deficiency in a male African American patient with systemic lupus erythematosus
35. Low gene copy-number of complement C4A, the presence of HLA-DR3, and the presence of HLA-DR2 are independent and additive risk factors for human systemic lupus erythematosus
36. Assessment of complement C4 gene copy number using the paralog ratio test
37. Chronic autoimmune activation in patients with mucopolysaccharidosis III (93.22)
38. Great genotypic and phenotypic diversities associated with copy-number variations (CNVs) of complement C4 and RP-C4-CYP21-TNX (RCCX) modules in Asian-Indian and European Americans (136.29)
39. Copy number variation (CNV) of human FCGRIIIB is concurrent with FCGRIIC and HSP70B: low copy number is a risk factor for and high copy number is a protective factor against systemic lupus erythematosus (SLE) (49.24)
40. Determining the copy number variation (CNV) of human Complement Factor H Related Genes: from Southern blots to Real-time PCRs (136.30)
41. Low gene copy of complement C4 is a common genetic risk factor for human systemic lupus erythematosus in three different racial groups
42. Genetic and clinical parameters determining human systemic lupus erythematosus (SLE) disease flares and complement C4 protein levels
43. Sensitive and Specific Real-Time Polymerase Chain Reaction Assays to Accurately Determine Copy Number Variations (CNVs) of Human Complement C4A, C4B, C4-Long, C4-Short, and RCCX Modules: Elucidation of C4 CNVs in 50 Consanguineous Subjects with Defined HLA Genotypes
44. Frequent deletion of the 76-kb genomic DNA fragment with complement factor H-like genes CFHR3 and CFHR1 is protective against systemic lupus erythematosus (SLE) in European Americans
45. Sensitive and specific real-time PCR assays to accurately determine gene copy-number variations (CNVs) of human complement C4A, C4B, C4-Long, C4-Short and RCCX modules: Elucidation of C4 GCNVs in 50 consanguineous subjects with defined HLA genotypes
46. Gene Copy-Number Variation and Associated Polymorphisms of Complement Component C4 in Human Systemic Lupus Erythematosus (SLE): Low Copy Number Is a Risk Factor for and High Copy Number Is a Protective Factor against SLE Susceptibility in European Americans
47. Frequency of Carriers of 8.1 Ancestral Haplotype and its Fragments in Two Caucasian Populations
48. The HLA 8.1 ancestral haplotype is strongly linked to the C allele of −429T>C promoter polymorphism of receptor of the advanced glycation endproduct (RAGE) gene. Haplotype-independent association of the −429C allele with high hemoglobinA1C levels in diabetic patients
49. Human Complement Components C4A and C4B Genetic Diversities: Complex Genotypes and Phenotypes
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