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1. Next generation microfluidics: fulfilling the promise of lab-on-a-chip technologies

2. CDK4/6 inhibition enhances SHP2 inhibitor efficacy and is dependent upon RB function in malignant peripheral nerve sheath tumors

3. Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling

4. iCLOTS: open-source, artificial intelligence-enabled software for analyses of blood cells in microfluidic and microscopy-based assays

5. Ex vivo to in vivo model of malignant peripheral nerve sheath tumors for precision oncology

8. CDK4/6 inhibition enhances SHP2 inhibitor efficacy and is dependent upon restoration of RB function in malignant peripheral nerve sheath tumors

9. Fluorescence Lifetime Measurement of Prefibrillar Sickle Hemoglobin Oligomers as a Platform for Drug Discovery in Sickle Cell Disease

10. Ex vivo to in vivo model of malignant peripheral nerve sheath tumors for precision oncology

13. Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma

20. Effects of BCL11A Shmir-Induced Post-Transcriptional Silencing on Hemoglobin Polymer Inhibition in Single Red Blood Cells at Physiologic Oxygen Tension

23. An irradiated marrow niche reveals a small non-collagenous protein mediator of homing, dermatopontin

25. MetAP2 inhibition modifies hemoglobin S to delay polymerization and improves blood flow in sickle cell disease

27. Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma

28. CometChip

30. Robust Pre-Clinical Results and Large-Scale Manufacturing Process for Edit-301: An Autologous Cell Therapy for the Potential Treatment of SCD

38. Validation of BCL11A As Therapeutic Target in Sickle Cell Disease: Results from the Adult Cohort of a Pilot/Feasibility Gene Therapy Trial Inducing Sustained Expression of Fetal Hemoglobin Using Post-Transcriptional Gene Silencing

39. MetAP2 Inhibition Modifies Hemoglobin S (HbS) to Delay Polymerization and Improve Blood Flow in Sickle Cell Disease

40. SEMA4C is a novel target to limit osteosarcoma growth, progression, and metastasis

42. SEMA4C is a novel target to limit osteosarcoma growth, progression, and metastasis

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