51 results on '"Tomczak, Michal"'
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2. Neuro-Inclusive Job Design in a Smart Work Context: Demands-Control-Support Perspective.
3. Data from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
4. Supplementary Figure Legend from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
5. Data from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
6. Supplementary Figure S7 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
7. Supplementary Figure S4 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
8. Supplementary Figure S5 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
9. Supplementary Figure S1 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
10. Supplementary Figure S5 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
11. Supplementary Figure S3 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
12. Supplementary Table S1 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
13. Supplementary Figure Legend from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
14. Supplementary Figure S6 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
15. Supplementary Figure S7 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
16. Supplementary Figure S6 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
17. Supplementary Figure S1 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
18. Supplementary Figure S3 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
19. Supplementary Figure S4 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
20. Supplementary Table S1 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
21. Supplementary Figure S2 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
22. Supplementary Figure S2 from Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
23. A fossil diatom-based reconstruction of sea-level changes for the Late Pleistocene and Holocene period in the NW South China Sea
24. Migration of chalcophile elements in the oceanic lithosphere and implications for future SMS prospecting along the Mid-Atlantic Ridge (MAR, 26-33°N)
25. A diatom-based Holocene record of sedimentary and oceanographic environmental changes within the Beibu Gulf, NW South China Sea
26. ER Stress in Intestinal Inflammatory Disease
27. Late Quaternary Climate Variations Reflected in Baltic Sea Sediments
28. Crashing Construction Project Schedules by Relocating Resources
29. Stress Monitoring System for Individuals With Autism Spectrum Disorders
30. Selection of Construction Products Suppliers According to the Condorcet Criterion
31. Mo1872 Trough and Antibody Level Testing in Conjunction With the Use of a Standardized Clinical Assessment and Management Plan Does Not Improve Outcomes Among Patients With Inflammatory Bowel Disease
32. C25 highly branched isoprenoid alkenes in the Baltic Sea produced by the marine planktonic diatom Pseudosolenia calcar-avis
33. Obesity-Induced Colorectal Cancer Is Driven by Caloric Silencing of the Guanylin–GUCY2C Paracrine Signaling Axis
34. Last Glacial Cycle and seismic stratigraphic sequences offshore western Hainan Island, NW South China Sea
35. Efficacy of Vedolizumab as Induction Therapy in Refractory IBD Patients
36. Early-onset Crohn’s disease and autoimmunity associated with a variant in CTLA-4
37. Paneth cells as a site of origin for intestinal inflammation
38. ER stress transcription factor Xbp1 suppresses intestinal tumorigenesis and directs intestinal stem cells
39. ER stress transcription factor Xbp1 suppresses intestinal tumorigenesis and directs intestinal stem cells
40. 154 ER Stress Transcription Factor XBP1 Suppresses Intestinal Tumorigenesis
41. 631 Impaired Autophagy Promotes ER Stress Induced Intestinal Inflammation
42. The unfolded protein response and its role in intestinal homeostasis and inflammation
43. Conditional XBP1 Deletion Within the Intestinal Epithelial Cells Results in Activation of Autophagy in Paneth Cells
44. NF-κB1 Inhibits TLR-Induced IFN-β Production in Macrophages through TPL-2–Dependent ERK Activation
45. “ER Stress(ed Out)!”: Paneth Cells and Ischemia–Reperfusion Injury of the Small Intestine
46. Inhibition of Helicobacter hepaticus-Induced Colitis by IL-10 Requires the p50/p105 Subunit of NF-κB
47. Defective Activation of ERK in Macrophages Lacking the p50/p105 Subunit of NF-κB Is Responsible for Elevated Expression of IL-12 p40 Observed after Challenge with Helicobacter hepaticus
48. A Role for NF-κB Subunits p50 and p65 in the Inhibition of Lipopolysaccharide-Induced Shock
49. Regulation of PD-1, PD-L1, and PD-L2 expression during normal and autoimmune responses
50. NF-κB Is Required Within the Innate Immune System to Inhibit Microflora-Induced Colitis and Expression of IL-12 p40
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