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1. Networking to Optimize Dmd exon 53 Skipping in the Brain of mdx52 Mouse Model

3. Partial restoration of brain dystrophin by tricyclo-DNA antisense oligonucleotides alleviates emotional deficits in mdx52 mice

5. Oligonucleotide Enhancing Compound Increases Tricyclo-DNA Mediated Exon-Skipping Efficacy in the Mdx Mouse Model

6. Dystrophin myonuclear domain restoration governs treatment efficacy in dystrophic muscle

9. Palmitic acid conjugation enhances potency of tricyclo-DNA splice switching oligonucleotides

10. Identifying and Avoiding tcDNA-ASO Sequence-Specific Toxicity for the Development of DMD Exon 51 Skipping Therapy

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