103 results on '"Peng, Tianqing"'
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2. Nicotinamide mononucleotide as a therapeutic agent to alleviate multi-organ failure in sepsis
3. Leashing Ferroptosis in Myocardial Ischemia/Reperfusion Injury: Role of the YAP/NEDD4L/ACSL4 Pathway, Knowledge Gaps, and Potential Therapeutic Application
4. Abstract P1149: Junctophilin-2 Promotes Cardiomyocyte Survival By Blocking Murf1-mediated Junctin Ubiquitination And Proteasome-dependent Degradation
5. Lipocalin 10 is essential for protection against inflammation-triggered vascular leakage by activating LDL receptor-related protein 2-slingshot homologue 1 signalling pathway
6. Synthesis of N, N, N-trimethyl chitosan-based nanospheres for the prolonged release of curcumin
7. Junctophilin-2 promotes cardiomyocyte survival by blocking MURF1-mediated Junctin ubiquitination and proteasome-dependentdegradation
8. MLKL-mediated necroptosis is a target for cardiac protection in mouse models of type-1 diabetes
9. Renewal of embryonic and neonatal-derived cardiac-resident macrophages in response to environmental cues abrogated their potential to promote cardiomyocyte proliferation via Jagged-1–Notch1
10. Myeloid cell-specific deletion of Capns1 prevents macrophage polarization toward the M1 phenotype and reduces interstitial lung disease in the bleomycin model of systemic sclerosis
11. Loss of Lipocalin 10 Exacerbates Diabetes-Induced Cardiomyopathy via Disruption of Nr4a1-Mediated Anti-Inflammatory Response in Macrophages
12. Gamma-Aminobutyrate Transaminase Protects against Lipid Overload-Triggered Cardiac Injury in Mice
13. Pharmacological inhibition of Rac1 attenuates myocardial abnormalities in tail-suspended mice
14. Calpain-2 specifically cleaves Junctophilin-2 at the same site as Calpain-1 but with less efficacy
15. Calpain-Mediated Mitochondrial Damage: An Emerging Mechanism Contributing to Cardiac Disease
16. Sectm1a Facilitates Protection against Inflammation-Induced Organ Damage through Promoting TRM Self-Renewal
17. Administration of GDF3 Into Septic Mice Improves Survival via Enhancing LXRα-Mediated Macrophage Phagocytosis
18. Calpain activation mediates microgravity-induced myocardial abnormalities in mice via p38 and ERK1/2 MAPK pathways
19. Calpain Activation and Organ Failure in Sepsis: Molecular Insights and Therapeutic Perspectives
20. Identification of a Novel Antisepsis Pathway: Sectm1a Enhances Macrophage Phagocytosis of Bacteria through Activating GITR
21. Tsg101 Is Involved in the Sorting and Re-Distribution of Glucose Transporter-4 to the Sarcolemma Membrane of Cardiac Myocytes
22. Tsg101 positively regulates P62-Keap1-Nrf2 pathway to protect hearts against oxidative damage
23. Protective role of endothelial calpain knockout in lipopolysaccharide-induced acute kidney injury via attenuation of the p38-iNOS pathway and NO/ROS production
24. Sectm1a deficiency aggravates inflammation-triggered cardiac dysfunction through disruption of LXRα signalling in macrophages
25. GDF3 Protects Mice against Sepsis-Induced Cardiac Dysfunction and Mortality by Suppression of Macrophage Pro-Inflammatory Phenotype
26. SECTM1A Positively Regulates Tissue-Resident Macrophage Self-Renewal Capacity by Boosting T Helper Cells in Mice During Endotoxemia
27. S-Sulfhydration of SIRT3 by Hydrogen Sulfide Attenuates Mitochondrial Dysfunction in Cisplatin-Induced Acute Kidney Injury
28. Administration of losartan preserves cardiomyocyte size and prevents myocardial dysfunction in tail-suspended mice by inhibiting p47phox phosphorylation, NADPH oxidase activation and MuRF1 expression
29. Mitochondrial ROS-induced lysosomal dysfunction impairs autophagic flux and contributes to M1 macrophage polarization in a diabetic condition
30. MicroRNA-223 is essential for maintaining functional β-cell mass during diabetes through inhibiting both FOXO1 and SOX6 pathways
31. Nicotinamide riboside promotes autolysosome clearance in preventing doxorubicin-induced cardiotoxicity
32. Tsg101 positively regulates physiologic‐like cardiac hypertrophy through FIP3‐mediated endosomal recycling of IGF‐1R
33. Increased calpain-1 in mitochondria induces dilated heart failure in mice: role of mitochondrial superoxide anion
34. Calpain-2 promotes MKP-1 expression protecting cardiomyocytes in both in vitro and in vivo mouse models of doxorubicin-induced cardiotoxicity
35. Selective deletion of endothelial cell calpain in mice reduces diabetic cardiomyopathy by improving angiogenesis
36. Calpain‐2 protects against heat stress‐induced cardiomyocyte apoptosis and heart dysfunction by blocking p38 mitogen‐activated protein kinase activation
37. Administration of nicotinamide riboside prevents oxidative stress and organ injury in sepsis
38. An Hsp20-FBXO4 Axis Regulates Adipocyte Function through Modulating PPARγ Ubiquitination
39. Circulating Exosomes Isolated from Septic Mice Induce Cardiovascular Hyperpermeability Through Promoting Podosome Cluster Formation
40. Disruption of calpain reduces lipotoxicity-induced cardiac injury by preventing endoplasmic reticulum stress
41. MicroRNA-223-5p and -3p Cooperatively Suppress Necroptosis in Ischemic/Reperfused Hearts
42. Overexpression of miR-223 Tips the Balance of Pro- and Anti-hypertrophic Signaling Cascades toward Physiologic Cardiac Hypertrophy
43. Heat stress prevents lipopolysaccharide-induced apoptosis in pulmonary microvascular endothelial cells by blocking calpain/p38 MAPK signalling
44. Hsp20-Mediated Activation of Exosome Biogenesis in Cardiomyocytes Improves Cardiac Function and Angiogenesis in Diabetic Mice
45. Heat stress pretreatment decreases lipopolysaccharide-induced apoptosis via the p38 signaling pathway in human umbilical vein endothelial cells
46. Therapeutic inhibition of mitochondrial reactive oxygen species with mito-TEMPO reduces diabetic cardiomyopathy
47. Inhibition of MicroRNA 195 Prevents Apoptosis and Multiple-Organ Injury in Mouse Models of Sepsis
48. Abstract 13631: Therapeutic Inhibition of Mitochondrial Reactive Oxygen Species With Mito-tempo Reduces Diabetic Cardiomyopathy
49. Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction
50. Mitochondrial Calpain-1 Disrupts ATP Synthase and Induces Superoxide Generation in Type 1 Diabetic Hearts: A Novel Mechanism Contributing to Diabetic Cardiomyopathy
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