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2. Failure to apply standard limit-of-detection or limit-of-quantitation criteria to specialized pro-resolving mediator analysis incorrectly characterizes their presence in biological samples

5. Data from Biomarkers for Personalizing Omega-3 Fatty Acid Dosing

6. Data from Biomarkers for Personalizing Omega-3 Fatty Acid Dosing

15. Supplemental Figures S1-S6 from Chemotherapeutic Agents Subvert Tumor Immunity by Generating Agonists of Platelet-Activating Factor

16. Data from Chemotherapeutic Agents Subvert Tumor Immunity by Generating Agonists of Platelet-Activating Factor

17. Data from Chemotherapeutic Agents Subvert Tumor Immunity by Generating Agonists of Platelet-Activating Factor

18. Supplementary Methods from Chemotherapeutic Agents Subvert Tumor Immunity by Generating Agonists of Platelet-Activating Factor

19. Table Supplementary S-I from Chemotherapeutic Agents Subvert Tumor Immunity by Generating Agonists of Platelet-Activating Factor

20. The SARS-CoV2 envelope differs from host cells, exposes procoagulant lipids, and is disrupted in vivo by oral rinses

21. Formation, Signaling and Occurrence of Specialized Pro-Resolving Lipid Mediators—What is the Evidence so far?

22. Contributors

24. The SARS-CoV2 envelope is distinct from host membranes, exposes pro-coagulant lipids, and can be inactivated in vivo by surfactant-containing oral rinses.

25. Oxylipin metabolism is controlled by mitochondrial β-oxidation during bacterial inflammation

27. Lipidomic and transcriptional analysis of the linoleoyl-omega-hydroxyceramide biosynthetic pathway in human psoriatic lesions

29. List of contributors**Authors’ names are followed by the starting page number(s) of their contribution(s).

31. Update on LIPID MAPS classification, nomenclature, and shorthand notation for MS-derived lipid structures

32. Oxylipin metabolism is controlled by mitochondrial β-oxidation during bacterial inflammation

49. Potential Role of Oral Rinses Targeting the Viral Lipid Envelope in SARS-CoV-2 Infection

50. Revising the structure of a new eicosanoid from human platelets to 8,9–11,12-diepoxy-13-hydroxyeicosadienoic acid

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