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1. Non-viral DNA delivery and TALEN editing correct the sickle cell mutation in hematopoietic stem cells

2. CoCas9 is a compact nuclease from the human microbiome for efficient and precise genome editing

3. Progress and harmonization of gene editing to treat human diseases: Proceeding of COST Action CA21113 GenE-HumDi

9. Novel lentiviral vectors for gene therapy of sickle cell disease combining gene addition and gene silencing strategies

12. An optimized SpCas9 high-fidelity variant for direct protein delivery

14. Novel lentiviral vectors for gene therapy of sickle cell disease combining gene addition and gene silencing strategies

16. Targeted Base Editing Strategies for Beta-Hemoglobinopathies

18. Base-editing-mediated dissection of a γ-globin cis-regulatory element for the therapeutic reactivation of fetal hemoglobin expression

21. Long-term outcomes of lentiviral gene therapy for the β-hemoglobinopathies: the HGB-205 trial

23. Combination of lentiviral and genome editing technologies for the treatment of sickle cell disease

24. Pre-Clinical Development of a Highly Efficient TALEN ®-Based Correction of the β-Globin Gene in Patient-Derived Hematopoietic Stem and Progenitor Cells (HSPCs) to Treat Sickle Cell Disease

26. Clinical Results of the Drepaglobe Trial for Sickle Cell Disease Patients

27. Base Editing-Mediated Dissection of the -200 Region of the γ-Globin Promoters to Induce Fetal Hemoglobin and Rescue Sickle Cell Disease and β-Thalassemia

28. Dysérythropoïèse dans la drépanocytose : une contribution d’origine centrale à l’anémie ?

30. Recent progress in genome editing for gene therapy applications: the French perspective

33. Correction of β-thalassemia by CRISPR/Cas9 editing of the α-globin locus in human hematopoietic stem cells

37. Editing the LRF Repressor Binding Site in the γ-Globin Promoters Induces Therapeutically Relevant Fetal Hemoglobin Levels for the Treatment of β-Hemoglobinopathies

38. Fetal Hemoglobin Rescues Ineffective Erythropoiesis in Sickle Cell Disease

41. GATA Factor-Mediated Gene Regulation in Human Erythropoiesis

42. Editing a γ-globin repressor binding site restores fetal hemoglobin synthesis and corrects the sickle cell disease phenotype

43. Results from the Completed Hgb-205 Trial of Lentiglobin for β-Thalassemia and Lentiglobin for Sickle Cell Disease Gene Therapy

44. Editing a γ-Globin Repressor Binding Site Restores Fetal Hemoglobin Synthesis and Corrects the Phenotype of Sickle Cell Disease Erythrocytes

46. Extensive multilineage analysis in patients with mixed chimerism after allogeneic transplantation for sickle cell disease: insight into hematopoiesis and engraftment thresholds for gene therapy

50. Optimization of CRISPR/Cas9 Delivery to Human Hematopoietic Stem and Progenitor Cells for Therapeutic Genomic Rearrangements

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