83 results on '"Manne, Sasikanth"'
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2. Stat5 opposes the transcription factor Tox and rewires exhausted CD8+ T cells toward durable effector-like states during chronic antigen exposure
3. 1016 Enforcement of exhausted T cell epigenetic fate by HMG-box transcription factor TOX
4. 579-C Reinvigoration of progenitor-exhausted CD8 T cells by anti-CTLA-4 contributes to the sustained activity of combination checkpoint blockade
5. Trimodal single-cell profiling reveals a novel pediatric CD8αα+ T cell subset and broad age-related molecular reprogramming across the T cell compartment
6. Exploration of T-Cell Diversity Using Mass Cytometry
7. In vitro modeling of CD8 + T cell exhaustion enables CRISPR screening to reveal a role for BHLHE40
8. Tri-modal single cell (TEA-seq) profiling reveals a novel, epigenetically distinct T cell subset in children and global molecular reprogramming across age
9. Data from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis
10. Supplementary Figure, Figure Legends, Tables and Material and Methods from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis
11. Data from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis
12. Supplementary Figure, Figure Legends, Tables and Material and Methods from The Codon 72 TP53 Polymorphism Contributes to TSC Tumorigenesis through the Notch–Nodal Axis
13. Data from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
14. Supplementary Materials and Methods from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
15. Supplementary Figure Legends from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
16. Supplementary Materials and Methods from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
17. Data from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
18. Supplementary Figures 1 - 5 from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
19. Supplementary Figures 1 - 5 from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
20. Supplementary Figure Legends from Complement C5a Receptor Facilitates Cancer Metastasis by Altering T-Cell Responses in the Metastatic Niche
21. Shared and distinct biological circuits in effector, memory and exhausted CD8+ T cells revealed by temporal single-cell transcriptomics and epigenetics
22. Enhanced STAT5a activation rewires exhausted CD8 T cells during chronic stimulation to acquire a hybrid durable effector like state
23. Distinct Heterogeneity in the Naive T cell Compartments of Children and Adults
24. PD-1 directed immunotherapy alters Tfh and humoral immune responses to seasonal influenza vaccine
25. Abstract 3579: T cell intrinsic DNA damage and repair response as a novel marker associated with clinical response to PD-1 blockade
26. Autoreactive CD8+ T cells are restrained by an exhaustion-like program that is maintained by LAG3
27. PD-1 restricts the development and effector function of tissue regulatory T cells in experimental autoimmune encephalomyelitis
28. MicroRNA-29a attenuates CD8 T cell exhaustion and induces memory-like CD8 T cells during chronic infection
29. Induction of a CD8 T cell intrinsic DNA damage and repair response is associated with clinical response to PD-1 blockade in uterine cancer
30. Longitudinal single cell transcriptional and epigenetic mapping of effector, memory, and exhausted CD8 T cells reveals shared biological circuits across distinct cell fates
31. Human epigenetic and transcriptional T cell differentiation atlas for identifying functional T cell-specific enhancers
32. 253 PD1 and LAG3 converge to limit polyfunctionality and systemic immunity
33. 310 T cell intrinsic DNA damage and repair response as a novel marker associated with clinical response to PD-1 blockade
34. Author Correction: Epigenetic scarring of exhausted T cells hinders memory differentiation upon eliminating chronic antigenic stimulation
35. Epigenetic scarring of exhausted T cells hinders memory differentiation upon eliminating chronic antigenic stimulation
36. TET2 controls differentiation of terminally exhausted CD8 T cells
37. A role for the transcription factor STAT5 in antagonizing CD8+ T cell exhaustion
38. Vaccine-induced ICOS+CD38+ circulating Tfh are sensitive biosensors of age-related changes in inflammatory pathways
39. Role of nuclear localization in the regulation and function of T-bet and Eomes in exhausted CD8 T cells
40. In vivo CD8+ T cell CRISPR screening reveals control by Fli1 in infection and cancer
41. Abstract PO068: Distinct immune signatures predicting clinical response to PD-1 blockade therapy in gynecological cancers revealed by high-dimensional immune profiling
42. Inhibitory signaling sustains a distinct early memory CD8 + T cell precursor that is resistant to DNA damage
43. The Identity of Human Tissue-Emigrant CD8+ T Cells
44. 186 Distinct immune signatures predicting clinical response to PD-1 blockade therapy in gynecological cancers revealed by high-dimensional immune profiling
45. Abstract IA04: Overcoming immune resistance with rationally designed combination immunotherapy
46. The identity of human tissue-emigrant CD8+T cells
47. T cell exhaustion dynamics are linked to clinical outcomes in hepatocellular carcinoma
48. Neuropilin-1 is a T cell memory checkpoint limiting long-term antitumor immunity
49. In vivo CRISPR screening identifies Fli1 as a transcriptional safeguard that restrains effector CD8 T cell differentiation during infection and cancer
50. Developmental relationships of four exhausted CD8 T cell subsets reveals underlying transcriptional and epigenetic control mechanisms
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