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12. BPTF in bone marrow provides a potential progression biomarker regulated by TFAP4 through the PI3K/AKT pathway in neuroblastoma

15. TAF1D promotes proliferation by transcriptionally activating G2 /M phase‐related genes inMYCN‐amplified neuroblastoma

17. Data from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

18. Supplementary Figure 1 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

19. Supplementary Figure 5 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

20. Supplementary Figure 1 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

21. Supplementary Figure 2 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

22. Supplementary Figure 1 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

23. Data from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

24. Supplementary Figure 4 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

25. Supplementary Figure Legend from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

26. Data from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

27. Supplementary Figure 5 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

28. Supplementary Figure 6 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

29. Supplementary Figure 7 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

30. Supplementary Figure Legend from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

31. Supplementary Figure 7 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

32. Supplementary Figure 1 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

33. Supplementary Figure 3 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

34. Supplementary Figure 4 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

35. Supplementary Figure Legend from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

36. Supplementary Figure 3 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

37. Supplementary Figure 2 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

38. Supplementary Figure 3 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

39. Supplementary Figure 6 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

40. Supplementary Figure 2 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

41. Supplementary Figure Legend from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer

42. Supplementary Figure 3 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

43. Supplementary Figure 2 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target

48. Supplementary Figure 1 from Functional Genetic Variations in Cytotoxic T-Lymphocyte Antigen 4 and Susceptibility to Multiple Types of Cancer

49. Supplementary Figure 1 from Blockade of the Ubiquitin Protease UBP43 Destabilizes Transcription Factor PML/RARα and Inhibits the Growth of Acute Promyelocytic Leukemia

50. Supplementary Figure 1 from Functional Genetic Variations in Cytotoxic T-Lymphocyte Antigen 4 and Susceptibility to Multiple Types of Cancer

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